Wall shear stress determination from near-wall mean velocity data in turbulent pipe and channel flows

1996 ◽  
Vol 20 (6) ◽  
pp. 417-428 ◽  
Author(s):  
F. Durst ◽  
H. Kikura ◽  
I. Lekakis ◽  
J. Jovanović ◽  
Q. Ye
Keyword(s):  
Shear Stress ◽  
Wall Shear Stress ◽  
Channel Flows ◽  
Stress Determination ◽  
Velocity Data ◽  
Mean Velocity ◽  
Wall Shear ◽  
Near Wall

Near-Wall Measurements in Turbulent Boundary Layers Using Laser Doppler Anemometry

2002 ◽  
Author(s):  
T. Gunnar Johansson ◽  
Luciano Castillo
Keyword(s):  
Boundary Layer ◽  
Shear Stress ◽  
Wall Shear Stress ◽  
Pressure Gradient ◽  
Laser Doppler Anemometry ◽  
Reynolds Numbers ◽  
Mean Velocity ◽  
The Mean ◽  
Wall Shear ◽  
Near Wall

Near wall measurements have been performed in a zero pressure gradient turbulent boundary layer at low to moderate local Reynolds numbers using Laser-Doppler Anemometry in order to investigate how accurately the wall shear stress can be determined. Also, scaling problems are particularly difficult at low Reynolds numbers since they involve simultaneous influences of both inner and outer scales and this is most clearly observed in the near-wall region. In order to fully describe the zero pressure gradient turbulent boundary layer at low to moderate local Reynolds numbers it is necessary to accurately measure a number of quantities. These include the mean velocity and Reynolds stresses, and their spatial derivatives all the way down to the wall (y+∼1). Integral parameters that need to be measured are the wall shear stress and boundary layer thickness, particularly the momentum thickness. Problems with the measurement of field properties get worse close to a wall, and they get worse for increasing local Reynolds number. Three different approaches to measure the wall shear stress were examined. It was found that small measurement errors in the mean velocity close to the wall significantly reduced the accuracy in determining the wall shear stress by measuring the velocity gradient at the wall. The constant stress layer was found to be affected by the advection terms. However, it was found that taking the small pressure gradient into account and improving on the spatial resolution in the outer part of the boundary layer made the momentum integral method reliable.

A Dynamic Procedure for Wall-Modeled Large-Eddy Simulation of High Reynolds Number Flows

2011 ◽  
Author(s):  
Soshi Kawai
Keyword(s):  
Reynolds Number ◽  
Shear Stress ◽  
Wall Shear Stress ◽  
Large Eddy Simulation ◽  
High Reynolds Number ◽  
Eddy Simulation ◽  
Large Eddy ◽  
Wall Shear ◽  
High Reynolds ◽  
Near Wall

This paper addresses the error in large-eddy simulation with wall-modeling (i.e., when the wall shear stress is modeled and the viscous near-wall layer is not resolved): the error in estimating the wall shear stress from a given outer-layer velocity field using auxiliary near-wall RANS equations where convection is not neglected. By considering the behavior of turbulence length scales near a wall, the cause of the errors is diagnosed and solutions that remove the errors are proposed based solidly on physical reasoning. The resulting method is shown to accurately predict equilibrium boundary layers at very high Reynolds number, with both realistic instantaneous fields (without overly elongated unphysical near-wall structures) and accurate statistics (both skin friction and turbulence quantities).

scholarly journals On the structure of streamwise wall-shear stress fluctuations in turbulent channel flows

2020 ◽  
Vol 1522 ◽  
pp. 012010
Author(s):  
Cheng Cheng ◽  
Weipeng Li ◽  
Adrián Lozano-Durán ◽  
Yitong Fan ◽  
Hong Liu
Keyword(s):  
Shear Stress ◽  
Wall Shear Stress ◽  
Channel Flows ◽  
Turbulent Channel Flows ◽  
Wall Shear

Numerical Simulation of Wall Shear Stress Conditions and Platelet Localization in Realistic End-to-Side Arterial Anastomoses

2003 ◽  
Vol 125 (5) ◽  
pp. 671-681 ◽  
Author(s):  
P. Worth Longest ◽  
Clement Kleinstreuer
Keyword(s):  
Shear Stress ◽  
Wall Shear Stress ◽  
Bypass Graft ◽  
Self Regulation ◽  
Suture Line ◽  
Cellular Level ◽  
Vascular Surface ◽  
Wall Shear ◽  
Wall Interactions ◽  
Near Wall

Research studies over the last three decades have established that hemodynamic interactions with the vascular surface as well as surgical injury are inciting mechanisms capable of eliciting distal anastomotic intimal hyperplasia (IH) and ultimate bypass graft failure. While abnormal wall shear stress (WSS) conditions have been widely shown to affect vascular biology and arterial wall self-regulation, the near-wall localization of critical blood particles by convection and diffusion may also play a significant role in IH development. It is hypothesized that locations of elevated platelet interactions with reactive or activated vascular surfaces, due to injury or endothelial dysfunction, are highly susceptible to IH initialization and progression. In an effort to assess the potential role of platelet-wall interactions, experimentally validated particle-hemodynamic simulations have been conducted for two commonly implemented end-to-side anastomotic configurations, with and without proximal outflow. Specifically, sites of significant particle interactions with the vascular surface have been identified by a novel near-wall residence time (NWRT) model for platelets, which includes shear stress-based factors for platelet activation as well as endothelial cell expression of thrombogenic and anti-thrombogenic compounds. Results indicate that the composite NWRT model for platelet-wall interactions effectively captures a reported shift in significant IH formation from the arterial floor of a relatively high-angle (30 deg) graft with no proximal outflow to the graft hood of a low-angle graft (10 deg) with 20% proximal outflow. In contrast, other WSS-based hemodynamic parameters did not identify the observed system-dependent shift in IH formation. However, large variations in WSS-vector magnitude and direction, as encapsulated by the WSS-gradient and WSS-angle-gradient parameters, were consistently observed along the IH-prone suture-line region. Of the multiple hemodynamic factors capable of eliciting a hyperplastic response at the cellular level, results of this study indicate the potential significance of platelet-wall interactions coinciding with regions of low WSS in the development of IH.

The Law of the Wall for Swirling Flow in Annular Ducts

1989 ◽  
Vol 111 (2) ◽  
pp. 160-164 ◽  
Author(s):  
R. J. Kind ◽  
F. M. Yowakim ◽  
S. A. Sjolander
Keyword(s):  
Shear Stress ◽  
Wall Shear Stress ◽  
Swirling Flow ◽  
Tangential Velocity ◽  
Wall Region ◽  
Law Of The Wall ◽  
Resultant Velocity ◽  
The Law ◽  
Wall Shear ◽  
Near Wall

Expressions for the logarithmic portion of the law of the wall are derived for the axial and tangential velocity components of swirling flow in annular ducts. These expressions involve new shear-velocity scales and curvature terms. They are shown to agree well with experiment over a substantial portion of the flow near both walls of an annulus. The resultant velocity data also agree with the law of the wall. The success of the proposed logarithmic expressions implies that the mixing-length model used in deriving them correctly describes flow-velocity behavior. This model indicates that the velocity gradient at any height y in the near-wall region is determined by the wall shear stress, not by the local shear stress. This suggests that the influence of wall shear stress is dominant and that it determines the near-wall wall flow even in flows with curvature and pressure gradient. A physical explanation is suggested for this.

scholarly journals Effect of reverse flow on the pattern of wall shear stress near arterial branches

2011 ◽  
Vol 8 (64) ◽  
pp. 1594-1603 ◽  
Author(s):  
A. Kazakidi ◽  
A. M. Plata ◽  
S. J. Sherwin ◽  
P. D. Weinberg
Keyword(s):  
Shear Stress ◽  
Wall Shear Stress ◽  
Steady Flow ◽  
Stokes Equations ◽  
Reverse Flow ◽  
Side Branch ◽  
Navier Stokes ◽  
Wall Region ◽  
Wall Shear ◽  
Near Wall

Atherosclerotic lesions have a patchy distribution within arteries that suggests a controlling influence of haemodynamic stresses on their development. The distribution near aortic branches varies with age and species, perhaps reflecting differences in these stresses. Our previous work, which assumed steady flow, revealed a dependence of wall shear stress (WSS) patterns on Reynolds number and side-branch flow rate. Here, we examine effects of pulsatile flow. Flow and WSS patterns were computed by applying high-order unstructured spectral/hp element methods to the Newtonian incompressible Navier–Stokes equations in a geometrically simplified model of an aorto-intercostal junction. The effect of pulsatile but non-reversing side-branch flow was small; the aortic WSS pattern resembled that obtained under steady flow conditions, with high WSS upstream and downstream of the branch. When flow in the side branch or in the aortic near-wall region reversed during part of the cycle, significantly different instantaneous patterns were generated, with low WSS appearing upstream and downstream. Time-averaged WSS was similar to the steady flow case, reflecting the short duration of these events, but patterns of the oscillatory shear index for reversing aortic near-wall flow were profoundly altered. Effects of reverse flow may help explain the different distributions of lesions.

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