The figwort mosaic virus gene VI promoter region contains a sequence highly homologous to the octopine synthase (ocs) enhancer element

1990 ◽  
Vol 15 (1) ◽  
pp. 181-182 ◽  
Author(s):  
Richard Cooke
Keyword(s):  
Mosaic Virus ◽  
Promoter Region ◽  
Enhancer Element ◽  
Virus Gene ◽  
Figwort Mosaic Virus

scholarly journals A second cauliflower mosaic virus gene product influences the structure of the viral inclusion body

1984 ◽  
Vol 3 (6) ◽  
pp. 1423-1427 ◽  
Author(s):  
L. Givord ◽  
C. Xiong ◽  
M. Giband ◽  
I. Koenig ◽  
T. Hohn ◽  
...  
Keyword(s):  
Mosaic Virus ◽  
Gene Product ◽  
Inclusion Body ◽  
Cauliflower Mosaic Virus ◽  
Virus Gene ◽  
Viral Inclusion

Immunological detection of cauliflower mosaic virus gene V protein produced in engineered bacteria or infected plants

Gene ◽  
1985 ◽  
Vol 36 (3) ◽  
pp. 271-279 ◽  
Author(s):  
Véronique Ziegler ◽  
Patricia Laquel ◽  
Hubert Guilley ◽  
Ken Richards ◽  
Gérard Jonard
Keyword(s):  
Mosaic Virus ◽  
Cauliflower Mosaic Virus ◽  
V Protein ◽  
Virus Gene ◽  
Immunological Detection ◽  
Engineered Bacteria

scholarly journals The 5′ Third of Cauliflower mosaic virus Gene VI Conditions Resistance Breakage in Arabidopsis Ecotype Tsu-0

2002 ◽  
Vol 92 (2) ◽  
pp. 190-196 ◽  
Author(s):  
K. Agama ◽  
S. Beach ◽  
J. Schoelz ◽  
S. M. Leisner
Keyword(s):  
Arabidopsis Thaliana ◽  
Mosaic Virus ◽  
Viral Genome ◽  
Plant Defenses ◽  
Cauliflower Mosaic Virus ◽  
Virus Gene ◽  
Arabidopsis Ecotype ◽  
Passive Mechanism ◽  
Different Types ◽  
Solanaceous Plants

Arabidopsis thaliana ecotypes vary in their responses to viruses. In this study, we analyzed the variation in response of A. thaliana ecotype Tsu-0 to Cauliflower mosaic virus (CaMV). This ecotype was previously reported to be resistant to two CaMV isolates (CM1841 and CM4-184), but susceptible to W260. In this study, we show that Tsu-0 is resistant to four additional CaMV isolates. CaMV propagated within the rosette leaves of Tsu-0 plants, but did not appear to spread systemically into the inflorescence. However, virus viability in rosette leaves of Tsu-0 plants apparently was not compromised because infectious CaMV could be recovered from these organs. W260 overcomes Tsu-0 resistance by a passive mechanism (i.e., this virus avoids activating plant defenses). The portion of the viral genome responsible for W260 resistance breakage was mapped to the 5′ third of gene VI, which we have termed RBR-1. This region is also responsible for controlling the ability of CaMV to infect different types of solanaceous plants. Hence, the pathways by which plants of different families interact with CaMV may be conserved through evolution.

A naturally occurring deletion mutant of figwort mosaic virus (caulimovirus) is generated by RNA splicing

Virology ◽  
1991 ◽  
Vol 184 (1) ◽  
pp. 290-298 ◽  
Author(s):  
Herman B. Scholthof ◽  
Fang C. Wu ◽  
Richard D. Richins ◽  
Robert J. Shepherd
Keyword(s):  
Mosaic Virus ◽  
Rna Splicing ◽  
Deletion Mutant ◽  
Figwort Mosaic Virus ◽  
Naturally Occurring

Location and sequence of a region of cauliflower mosaic virus gene 2 responsible for aphid transmissibility

Virology ◽  
1987 ◽  
Vol 160 (1) ◽  
pp. 246-251 ◽  
Author(s):  
Crispin J. Woolston ◽  
Lloyd G. Czaplewski ◽  
Peter G. Markham ◽  
Alison S. Goad ◽  
Roger Hull ◽  
...  
Keyword(s):  
Mosaic Virus ◽  
Cauliflower Mosaic Virus ◽  
Virus Gene
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