Ontogenetic changes and intraspecific resource partitioning in the tahoe sucker, Catostomus tahoensis

1983 ◽  
Vol 8 (1) ◽  
pp. 39-47 ◽  
Author(s):  
Donn L. Marrin
Keyword(s):  
Resource Partitioning ◽  
Ontogenetic Changes ◽  
Catostomus Tahoensis

scholarly journals Ontogenetic niche shifts and resource partitioning of lake trout morphotypes

2009 ◽  
Vol 66 (6) ◽  
pp. 1007-1018 ◽  
Author(s):  
Mara S. Zimmerman ◽  
Stephanie N. Schmidt ◽  
Charles C. Krueger ◽  
M. Jake Vander Zanden ◽  
Randy L. Eshenroder
Keyword(s):  
Stable Isotope ◽  
Resource Partitioning ◽  
Lake Trout ◽  
Niche Partitioning ◽  
Ontogenetic Changes ◽  
Large Lakes ◽  
Isotope Data ◽  
Ontogenetic Niche Shifts ◽  
Niche Shifts ◽  
Stable Isotope Data

Resource polymorphisms are widely observed in fishes; however, ontogenetic contributions to morphological and ecological differences are poorly understood. This study examined whether ontogenetic changes in niche partitioning could explain morphological and buoyancy differences between lake trout ( Salvelinus namaycush ) morphotypes in Great Slave Lake (Northwest Territories, Canada). Morphometric analysis, buoyancy, capture depth, diet, and stable isotope data were used in concert to determine whether (i) differences occur in small, as well as large, lake trout, (ii) ontogenetic changes in morphology and buoyancy correlate with shifts in depth or diet, and (iii) a subset of small trout, putatively identified as “humpers”, are distinct from other morphotypes. Ontogenetic changes in lake trout morphology were associated with an ecological shift between benthic and pelagic feeding. Resource partitioning between lean and siscowet-like trout occurred within benthic (small trout) and pelagic (large trout) habitats. The humper subset did not differ from small siscowet-like trout. By combining multiple methods and an ontogenetic perspective, our study provides novel perspectives on resource polymorphisms in large, deep lakes and on existing interpretations of stable isotope data from large lakes in general.

Brief description of prey selectivity and ontogenetic changes in the diet of the invasive lionfish Pterois volitans (Actinopterygii, Scorpaenidae) in the Mexican Caribbean

2018 ◽  
Author(s):  
Juan Carlos Villaseñor-Derbez
Keyword(s):  
Stomach Contents ◽  
Ontogenetic Change ◽  
Prey Selectivity ◽  
Ontogenetic Changes ◽  
Pterois Volitans ◽  
Mexican Caribbean ◽  
Invasive Lionfish

Stomach contents were analyzed from 109 individuals. A total of 4 Genera and 14 Species were identified. Crustaceans accounted for %N=67.39% , %IRI= 86.37% of the total identified taxa and Teleosts %N=32.61% (%IRI = 13.63%). An ontogenetic change was observed in P. volitans diet.

Ontogenetic Changes in the Respiration Coefficients of Grain Sorghum

Crop Science ◽  
1988 ◽  
Vol 28 (1) ◽  
pp. 111-113 ◽  
Author(s):  
Randal S. Stahl ◽  
J. McCree
Keyword(s):  
Grain Sorghum ◽  
Ontogenetic Changes

Dietary overlap and food resource partitioning among fish species of a tropical estuary in Northeastern Brazil

Gaia Scientia ◽  
2016 ◽  
Vol 10 (4) ◽  
pp. 86-95 ◽  
Author(s):  
Adna Ferreira da Silva Garcia ◽  
Ana Lúcia Vendel
Keyword(s):  
Resource Partitioning ◽  
Species Coexistence ◽  
Food Resource ◽  
River Estuary ◽  
Niche Overlap ◽  
Northeastern Brazil ◽  
Dietary Overlap ◽  
Feeding Niche ◽  
Tropical Environments ◽  
Food Resource Partitioning

The current work investigates dietary overlap and food partitioning among nine abundant carnivorous fishes caught in the shallow waters of the Paraíba do Norte river estuary, Paraíba State, Brazil. Fishes were sampled with a beach seine net between January and December 2008 and a total of 958 specimens had their stomach content analyzed. Crustacea was the dominant food resource for Lutjanus alexandrei, L. jocu and Bathygobius soporator, whereas Telostei were consumed mainly by Centropomus undecimalis and C. parallelus. In contrast, Polychaeta were preyed upon mainly by Diapterus rhombeus, Eucinostomus argenteus, Sciades herzbergii and S. parkeri. Although most species consumed similar food items, they did that in varying proportions and amounts. Overall, the niche overlap among species was low (< 0.60), but there were several cases where pair of species had their feeding niche highly overlapped (between 0.72 and 0.97). These findings corroborate the hypothesis that food resource partitioning determines species coexistence in estuarine tropical environments.

scholarly journals Chromosome Instability, Aging and Brain Diseases

Cells ◽  
2021 ◽  
Vol 10 (5) ◽  
pp. 1256
Author(s):  
Ivan Y. Iourov ◽  
Yuri B. Yurov ◽  
Svetlana G. Vorsanova ◽  
Sergei I. Kutsev
Keyword(s):  
Brain Aging ◽  
Chromosome Instability ◽  
Neuronal Cell Death ◽  
Neuronal Cell ◽  
Accelerated Aging ◽  
Brain Diseases ◽  
Aging Brain ◽  
Ontogenetic Changes ◽  
Health And Disease

Chromosome instability (CIN) has been repeatedly associated with aging and progeroid phenotypes. Moreover, brain-specific CIN seems to be an important element of pathogenic cascades leading to neurodegeneration in late adulthood. Alternatively, CIN and aneuploidy (chromosomal loss/gain) syndromes exhibit accelerated aging phenotypes. Molecularly, cellular senescence, which seems to be mediated by CIN and aneuploidy, is likely to contribute to brain aging in health and disease. However, there is no consensus about the occurrence of CIN in the aging brain. As a result, the role of CIN/somatic aneuploidy in normal and pathological brain aging is a matter of debate. Still, taking into account the effects of CIN on cellular homeostasis, the possibility of involvement in brain aging is highly likely. More importantly, the CIN contribution to neuronal cell death may be responsible for neurodegeneration and the aging-related deterioration of the brain. The loss of CIN-affected neurons probably underlies the contradiction between reports addressing ontogenetic changes of karyotypes within the aged brain. In future studies, the combination of single-cell visualization and whole-genome techniques with systems biology methods would certainly define the intrinsic role of CIN in the aging of the normal and diseased brain.

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