Reproductive Function in Female Mice Lacking the Gene for Endothelial Nitric Oxide Synthase

Nitric Oxide ◽  
1999 ◽  
Vol 3 (5) ◽  
pp. 366-374 ◽  
Author(s):  
Deborah L. Drazen ◽  
Sabra L. Klein ◽  
Arthur L. Burnett ◽  
Edward E. Wallach ◽  
Julie K. Crone ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Nitric Oxide Synthase ◽  
Endothelial Nitric Oxide Synthase ◽  
Reproductive Function ◽  
Female Mice ◽  
Oxide Synthase ◽  
Endothelial Nitric Oxide

scholarly journals ET-1 overexpression and endothelial nitric oxide synthase knock-out induce different pathological responses in the heart of male and female mice

Life Sciences ◽  
2013 ◽  
Vol 93 (25-26) ◽  
pp. e13
Author(s):  
Nicolas Vignon-Zellweger ◽  
Katharina Relle ◽  
Jan Rahnenfuhrer ◽  
Karima Schwab ◽  
Berthold Hocher ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Nitric Oxide Synthase ◽  
Endothelial Nitric Oxide Synthase ◽  
Male And Female ◽  
Knock Out ◽  
Female Mice ◽  
Oxide Synthase ◽  
Endothelial Nitric Oxide

scholarly journals Endothelin-1 overexpression and endothelial nitric oxide synthase knock-out induce different pathological responses in the heart of male and female mice

Life Sciences ◽  
2014 ◽  
Vol 118 (2) ◽  
pp. 219-225 ◽  
Author(s):  
Nicolas Vignon-Zellweger ◽  
Katharina Relle ◽  
Jan Rahnenführer ◽  
Karima Schwab ◽  
Berthold Hocher ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Nitric Oxide Synthase ◽  
Endothelial Nitric Oxide Synthase ◽  
Male And Female ◽  
Knock Out ◽  
Female Mice ◽  
Endothelin 1 ◽  
Oxide Synthase ◽  
Endothelial Nitric Oxide

Localization of endothelial nitric oxide synthase in the normal and failing human atrial myocardium

1996 ◽  
Vol 54 ◽  
pp. 786-787
Author(s):  
Chi-Ming Wei ◽  
Margarita Bracamonte ◽  
Shi-Wen Jiang ◽  
Richard C. Daly ◽  
Christopher G.A. McGregor ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Heart Failure ◽  
Nitric Oxide Synthase ◽  
Endothelial Nitric Oxide Synthase ◽  
Cardiac Tissues ◽  
Mammalian Tissues ◽  
End Stage Heart Failure ◽  
End Stage ◽  
Oxide Synthase ◽  
Endothelial Nitric Oxide

Nitric oxide (NO) is a potent endothelium-derived relaxing factor which also may modulate cardiomyocyte inotropism and growth via increasing cGMP. While endothelial nitric oxide synthase (eNOS) isoforms have been detected in non-human mammalian tissues, expression and localization of eNOS in the normal and failing human myocardium are poorly defined. Therefore, the present study was designed to investigate eNOS in human cardiac tissues in the presence and absence of congestive heart failure (CHF).Normal and failing atrial tissue were obtained from six cardiac donors and six end-stage heart failure patients undergoing primary cardiac transplantation. ENOS protein expression and localization was investigated utilizing Western blot analysis and immunohistochemical staining with the polyclonal rabbit antibody to eNOS (Transduction Laboratories, Lexington, Kentucky).

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