Bone Morphogenetic Proteins but Not Growth Differentiation Factors Induce Dopaminergic Differentiation in Mesencephalic Precursors

2002 ◽  
Vol 21 (3) ◽  
pp. 367-378 ◽  
Author(s):  
A Brederlau
Keyword(s):  
Bone Morphogenetic Proteins ◽  
Differentiation Factors ◽  
Dopaminergic Differentiation ◽  
Growth Differentiation Factors

Dual release of a hydrophilic and a hydrophobic osteogenic factor from a single liposome

RSC Advances ◽  
2016 ◽  
Vol 6 (115) ◽  
pp. 114599-114612 ◽  
Author(s):  
Nelson Monteiro ◽  
Albino Martins ◽  
Ricardo A. Pires ◽  
Susana Faria ◽  
Nuno A. Fonseca ◽  
...  
Keyword(s):  
Stem Cells ◽  
Mesenchymal Stem Cells ◽  
Osteogenic Differentiation ◽  
Differentiation Factors ◽  
Dual Release ◽  
Osteogenic Factor ◽  
Growth Differentiation Factors

Dual release of growth/differentiation factors from liposomes induced osteogenic differentiation of mesenchymal stem cells.

scholarly journals The TGFβ superfamily in cardiac dysfunction

2018 ◽  
Vol 50 (4) ◽  
pp. 323-335 ◽  
Author(s):  
Jian Wu ◽  
Olan Jackson-Weaver ◽  
Jian Xu
Keyword(s):  
Bone Morphogenetic Proteins ◽  
Immune Cells ◽  
Heart Development ◽  
Transforming Growth Factor ◽  
Cardiac Fibroblasts ◽  
Injury Repair ◽  
Differentiation Factors ◽  
Coronary Endothelial Cells ◽  
Cellular Components ◽  
Tgfβ Superfamily

Abstract TGFβ superfamily includes the transforming growth factor βs (TGFβs), bone morphogenetic proteins (BMPs), growth and differentiation factors (GDFs) and Activin/Inhibin families of ligands. Among the 33 members of TGFβ superfamily ligands, many act on multiple types of cells within the heart, including cardiomyocytes, cardiac fibroblasts/myofibroblasts, coronary endothelial cells, smooth muscle cells, and immune cells (e.g. monocytes/macrophages and neutrophils). In this review, we highlight recent discoveries on TGFβs, BMPs, and GDFs in different cardiac residential cellular components, in association with functional impacts in heart development, injury repair, and dysfunction. Specifically, we will review the roles of TGFβs, BMPs, and GDFs in cardiac hypertrophy, fibrosis, contractility, metabolism, angiogenesis, and regeneration.

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