Mean activity coefficients of NaCl in (sodium chloride + sodium bicarbonate+ water) fromT= (293.15 to 308.15) K

2001 ◽  
Vol 33 (9) ◽  
pp. 1107-1119 ◽  
Author(s):  
Xiaoyan Ji ◽  
Xiaohua Lu ◽  
Weilu Lin ◽  
Luzheng Zhang ◽  
Yanru Wang ◽  
...  
Keyword(s):  
Sodium Chloride ◽  
Sodium Bicarbonate ◽  
Activity Coefficients ◽  
Mean Activity Coefficients

scholarly journals Mean Activity Coefficients of Sodium Chloride in Water-Ethanol Solvents at 25 °C

1999 ◽  
Vol 17 (2) ◽  
pp. 201-208 ◽  
Author(s):  
A Lopes ◽  
J Santos ◽  
F Farelo ◽  
M I A Ferra
Keyword(s):  
Sodium Chloride ◽  
Activity Coefficients ◽  
Mean Activity Coefficients

scholarly journals Effect of sodium bicarbonate supplementation on the renin-angiotensin system in patients with chronic kidney disease and acidosis: a randomized clinical trial

2020 ◽  
Author(s):  
Dominique M. Bovée ◽  
Lodi C. W. Roksnoer ◽  
Cornelis van Kooten ◽  
Joris I. Rotmans ◽  
Liffert Vogt ◽  
...  
Keyword(s):  
Chronic Kidney Disease ◽  
Sodium Chloride ◽  
Kidney Disease ◽  
Sodium Bicarbonate ◽  
Renin Angiotensin System ◽  
Ammonium Excretion ◽  
Angiotensin System ◽  
Renin Angiotensin ◽  
Endothelin 1 ◽  
Plasma Bicarbonate

Abstract Background Acidosis-induced kidney injury is mediated by the intrarenal renin-angiotensin system, for which urinary renin is a potential marker. Therefore, we hypothesized that sodium bicarbonate supplementation reduces urinary renin excretion in patients with chronic kidney disease (CKD) and metabolic acidosis. Methods Patients with CKD stage G4 and plasma bicarbonate 15–24 mmol/l were randomized to receive sodium bicarbonate (3 × 1000 mg/day, ~ 0.5 mEq/kg), sodium chloride (2 × 1,00 mg/day), or no treatment for 4 weeks (n = 15/arm). The effects on urinary renin excretion (primary outcome), other plasma and urine parameters of the renin-angiotensin system, endothelin-1, and proteinuria were analyzed. Results Forty-five patients were included (62 ± 15 years, eGFR 21 ± 5 ml/min/1.73m2, plasma bicarbonate 21.7 ± 3.3 mmol/l). Sodium bicarbonate supplementation increased plasma bicarbonate (20.8 to 23.8 mmol/l) and reduced urinary ammonium excretion (15 to 8 mmol/day, both P < 0.05). Furthermore, a trend towards lower plasma aldosterone (291 to 204 ng/L, P = 0.07) and potassium (5.1 to 4.8 mmol/l, P = 0.06) was observed in patients receiving sodium bicarbonate. Sodium bicarbonate did not significantly change the urinary excretion of renin, angiotensinogen, aldosterone, endothelin-1, albumin, or α1-microglobulin. Sodium chloride supplementation reduced plasma renin (166 to 122 ng/L), and increased the urinary excretions of angiotensinogen, albumin, and α1-microglobulin (all P < 0.05). Conclusions Despite correction of acidosis and reduction in urinary ammonium excretion, sodium bicarbonate supplementation did not improve urinary markers of the renin-angiotensin system, endothelin-1, or proteinuria. Possible explanations include bicarbonate dose, short treatment time, or the inability of urinary renin to reflect intrarenal renin-angiotensin system activity. Graphic abstract

Effect of sodium bicarbonate and sodium chloride on aggregation and conformation of pork myofibrillar protein

2021 ◽  
Vol 350 ◽  
pp. 129233
Author(s):  
Yan-ping Li ◽  
Xue-hua Zhang ◽  
Fei Lu ◽  
Zhuang-Li Kang
Keyword(s):  
Sodium Chloride ◽  
Sodium Bicarbonate ◽  
Myofibrillar Protein
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