Effects of Preloads of Differing Energy and Macronutrient Content on Eating Behavior in Bulimia Nervosa

Appetite ◽  
1997 ◽  
Vol 29 (3) ◽  
pp. 353-367 ◽  
Author(s):  
BARBARA J ROLLS ◽  
MARION M HETHERINGTON ◽  
SUSAN A STONER ◽  
ARNOLD E ANDERSEN
Keyword(s):  
Bulimia Nervosa ◽  
Eating Behavior ◽  
Macronutrient Content

The 196G/A (val66met) polymorphism of the BDNF gene is significantly associated with binge eating behavior in women with bulimia nervosa or binge eating disorder

2006 ◽  
Vol 406 (1-2) ◽  
pp. 133-137 ◽  
Author(s):  
Palmiero Monteleone ◽  
Roberta Zanardini ◽  
Alfonso Tortorella ◽  
Massimo Gennarelli ◽  
Eloisa Castaldo ◽  
...  
Keyword(s):  
Eating Disorder ◽  
Bulimia Nervosa ◽  
Binge Eating ◽  
Eating Behavior ◽  
Binge Eating Disorder ◽  
Bdnf Gene ◽  
Val66met Polymorphism

scholarly journals Temporal associations between affective instability and dysregulated eating behavior in bulimia nervosa

2017 ◽  
Vol 92 ◽  
pp. 183-190 ◽  
Author(s):  
Laura A. Berner ◽  
Ross D. Crosby ◽  
Li Cao ◽  
Scott G. Engel ◽  
Jason M. Lavender ◽  
...  
Keyword(s):  
Bulimia Nervosa ◽  
Eating Behavior ◽  
Affective Instability

Eating behavior in bulimia nervosa: multiple meal analyses

1994 ◽  
Vol 60 (6) ◽  
pp. 864-873 ◽  
Author(s):  
M M Hetherington ◽  
M Altemus ◽  
M L Nelson ◽  
A S Bernat ◽  
P W Gold
Keyword(s):  
Bulimia Nervosa ◽  
Eating Behavior

Eating behavior of patients with bulimia nervosa

1989 ◽  
Vol 8 (5) ◽  
pp. 557-568 ◽  
Author(s):  
Christine Woell ◽  
Manfred M. Fichter ◽  
Karl-Martin Pirke ◽  
Guenter Wolfram
Keyword(s):  
Bulimia Nervosa ◽  
Eating Behavior

Insulinogenic index at 15min as a marker of stable eating behavior in bulimia nervosa

2004 ◽  
Vol 23 (4) ◽  
pp. 711-720 ◽  
Author(s):  
Daisuke Yasuhara ◽  
Yoshiki Tatebe ◽  
Takashi Nakayama ◽  
Tetsuro Muranaga ◽  
Shin-ichi Nozoe ◽  
...  
Keyword(s):  
Bulimia Nervosa ◽  
Eating Behavior ◽  
Insulinogenic Index

The relevance of interoception for eating behavior and eating disorders

2018 ◽  
Author(s):  
Beate M. Herbert ◽  
Olga Pollatos
Keyword(s):  
Eating Disorders ◽  
Body Weight ◽  
Bulimia Nervosa ◽  
Psychiatric Disorders ◽  
Eating Behavior ◽  
Predictive Coding ◽  
Well Being ◽  
The Self ◽  
Maladaptive Behavior ◽  
Per Se

The importance of interoception for adaptive and maladaptive behavior, as well as for psychopathology, has gained growing interest, and dysfunctional interoception has been recognized as representing a core impairment across psychosomatic and psychiatric disorders. Eating is intrinsically guided by interoceptive signals and is directly associated with homeostatic psychophysiological needs, well-being, and survival. This chapter provides conceptually and empirically drawn conclusions focusing on the relevance of distinguishable dimensions of interoception for shaping eating behavior and body weight, and for eating disorders. Going beyond eating behavior per se, anorexia and bulimia nervosa are conceptualized as characterized by profound impairment of the self, with dysfunctional interoception at its core. Predictive coding models are addressed to integrate conclusions and empirical findings tentatively.

scholarly journals The Role of “Mixed” Orexigenic and Anorexigenic Signals and Autoantibodies Reacting with Appetite-Regulating Neuropeptides and Peptides of the Adipose Tissue-Gut-Brain Axis: Relevance to Food Intake and Nutritional Status in Patients with Anorexia Nervosa and Bulimia Nervosa

2013 ◽  
Vol 2013 ◽  
pp. 1-21 ◽  
Author(s):  
Kvido Smitka ◽  
Hana Papezova ◽  
Karel Vondra ◽  
Martin Hill ◽  
Vojtech Hainer ◽  
...  
Keyword(s):  
Eating Disorders ◽  
Adipose Tissue ◽  
Food Intake ◽  
Bulimia Nervosa ◽  
Eating Behavior ◽  
Peptide Yy ◽  
Abnormal Eating ◽  
Normal Body Weight ◽  
Circulating Autoantibodies ◽  
The Individual

Eating disorders such as anorexia (AN) and bulimia nervosa (BN) are characterized by abnormal eating behavior. The essential aspect of AN is that the individual refuses to maintain a minimal normal body weight. The main features of BN are binge eating and inappropriate compensatory methods to prevent weight gain. The gut-brain-adipose tissue (AT) peptides and neutralizing autoantibodies play an important role in the regulation of eating behavior and growth hormone release. The mechanisms for controlling food intake involve an interplay between gut, brain, and AT. Parasympathetic, sympathetic, and serotoninergic systems are required for communication between brain satiety centre, gut, and AT. These neuronal circuits include neuropeptides ghrelin, neuropeptide Y (NPY), peptide YY (PYY), cholecystokinin (CCK), leptin, putative anorexigen obestatin, monoamines dopamine, norepinephrine (NE), serotonin, and neutralizing autoantibodies. This extensive and detailed report reviews data that demonstrate that hunger-satiety signals play an important role in the pathogenesis of eating disorders. Neuroendocrine dysregulations of the AT-gut-brain axis peptides and neutralizing autoantibodies may result in AN and BN. The circulating autoantibodies can be purified and used as pharmacological tools in AN and BN. Further research is required to investigate the orexigenic/anorexigenic synthetic analogs and monoclonal antibodies for potential treatment of eating disorders in clinical practice.

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