OC008: Posterior fossa pathology: high-resolution US versus Fetal MRI

2003 ◽  
Vol 22 (S1) ◽  
pp. 2-3
Author(s):  
V. Tina ◽  
S. Kumar ◽  
F. Cowan ◽  
N. M. Fisk ◽  
M. Rutherford
2003 ◽  
Vol 23 (sup1) ◽  
pp. S41-S41
Author(s):  
T. Vanderheyden ◽  
S. Kumar ◽  
F. Cowan ◽  
N. M. Fisk ◽  
M. Rutherford

Author(s):  
M.I. Botez ◽  
Ezzedine Attig ◽  
Jean Lorrain Vézina

ABSTRACT:High-resolution CT scans of the brain and posterior fossa were performed on 106 phenytoin (PHT)- treated epileptics, 28 de novo epileptics and 43 control subjects. A higher incidence of cerebellar and brainstem (CBS) atrophy was observed in chronic PHT- or PHT+ phenobarbital-treated epileptics compared to the two other groups. Some control subjects and de novo epileptics presented mild CBS atrophy, whereas moderate to severe atrophy was noted exclusively in chronically-treated patients. In attempting to delineate the etiology of CBS atrophy, epileptic patients were divided in three groups: 55 subjects with normal CT scans, 30 with both cerebral and CBS atrophy, and 49 with pure CBS atrophy. Their ages, length of illness, number of generalized seizures, number of other seizures, and amount of PHT received during their lifetime were assessed. Statistical analysis revealed that posterior fossa atrophy in epileptics was significantly correlated with both the length of the illness and the amount of PHT ingested during the patient's lifetime. The number of seizures appears to not be related to CBS atrophy.


2014 ◽  
Vol 37 (3) ◽  
pp. E2 ◽  
Author(s):  
Saul F. Morales-Valero ◽  
Carlo Bortolotti ◽  
Carmelo Sturiale ◽  
Giuseppe Lanzino

A long-held dogma in neurosurgery is that parenchymal arteriovenous malformations (AVMs) are congenital. However, there is no strong evidence supporting this theory. An increasing number of documented cases of de novo formation of parenchymal AVMs cast doubt on their congenital nature and suggest that indeed the majority of these lesions may form after birth. Further evidence suggesting the postnatal development of parenchymal AVMs comes from the exceedingly rare diagnosis of these lesions in utero despite the widespread availability of high-resolution imaging modalities such as ultrasound and fetal MRI. The exact mechanism of AVM formation has yet to be elucidated, but most likely involves genetic susceptibility and environmental triggering factors. In this review, the authors report 2 cases of de novo AVM formation and analyze the evidence suggesting that they represent an acquired condition.


2020 ◽  
Author(s):  
Katie Mckinnon ◽  
Giles S. Kendall ◽  
Cally J. Tann ◽  
Leigh Dyet ◽  
Magdalena Sokolska ◽  
...  

1994 ◽  
Vol 81 (3) ◽  
pp. 443-448 ◽  
Author(s):  
Leslie N. Sutton ◽  
Suzanne L. Wehrli ◽  
Laura Gennarelli ◽  
Zhiyue Wang ◽  
Robert Zimmerman ◽  
...  

✓ High-resolution proton magnetic resonance (MR) spectroscopy was performed on perchlorate extracts of tumors (24 cases) or peritumoral vermis (five cases) obtained at surgery. Fifteen tumors were typical cerebellar astrocytomas and nine were posterior fossa primitive neuroectodermal tumors/medulloblastomas. Spectra obtained from the five samples of peritumoral vermis revealed a pattern of metabolites similar to that reported for cerebellar tissue, but concentrations of most metabolites were low, perhaps due to dilution from peritumoral edema. The astrocytomas were characterized by high levels of valine, alanine, and choline, an increase in the choline:N-acetylaspartate (NAA) ratio, and a shift from glutamate to glutamine. Elevations in lactate, pyruvate, and glucose were the result of ischemia during sampling. The primitive neuroectodermal tumors/medulloblastomas were distinguished from astrocytomas by a greater increase in the choline:NAA ratio, a smaller decrease in the glutamate:glutamine ratio, and a relative increase in glycine, taurine, and inositol levels. These metabolic patterns may be of value diagnostically as in vivo MR spectroscopy achieves higher resolution.


2005 ◽  
Vol 20 (6) ◽  
pp. 524-527 ◽  
Author(s):  
Mehmet Teksam ◽  
Umut Ozyer ◽  
Alexander McKinney ◽  
Ismail Kirbas ◽  
Banu Cakir

2016 ◽  
Vol 9 (5) ◽  
pp. e17-e17 ◽  
Author(s):  
Stefan Kliesch ◽  
Christian Bauknecht ◽  
Georg Bohner ◽  
Thomas Liebig ◽  
Eberhard Siebert

We report a rare case of entrapment of the basilar artery into the sphenoid bone caused by a longitudinal fracture of the clivus. Using high resolution three-dimensional flat panel angiography, we show preservation of the basilar artery perforators in spite of severe stenosis of the entrapped segment of the basilar artery. There were no obvious signs and symptoms of posterior fossa stroke clinically or radiographically as far as could be assessed under given clinical circumstances.


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