OP19.01: Congenital Chylothorax. Prenatal diagnosis and long-term follow-up

2008 ◽  
Vol 32 (3) ◽  
pp. 375-375
Author(s):  
A. Galindo ◽  
P. Martin ◽  
J. M. Puente ◽  
A. Grañeras ◽  
J. M. Hernandez-García
1998 ◽  
Vol 18 (1) ◽  
pp. 51-59 ◽  
Author(s):  
Berit Kerner ◽  
Eugene Flaum ◽  
Holli Mathews ◽  
Dru E. Carlson ◽  
Samuel H. Pepkowitz ◽  
...  

Author(s):  
Ana Paula Pinho Matos ◽  
Heron Werner ◽  
Pedro Teixeira Castro ◽  
Tatiana Mendonça Fazecas e Costa ◽  
Renata do Amaral Nogueira ◽  
...  

2021 ◽  
Vol 58 (S1) ◽  
pp. 186-186
Author(s):  
D. Hernández Pérez ◽  
R. Garcia Delgado ◽  
R. Garcia Rodriguez ◽  
A. Amaro Acosta ◽  
I. Ortega Cárdenes ◽  
...  

2020 ◽  
Vol 8 (8) ◽  
pp. 1369-1375
Author(s):  
Sheng Mou Lin ◽  
Ho Ming Luk ◽  
Ivan Fai Man Lo ◽  
Wai‐Keung Tam ◽  
Kelvin Yuen Kwong Chan ◽  
...  

2018 ◽  
Vol 33 (13) ◽  
pp. 2181-2189
Author(s):  
Ana Paula Pinho Matos ◽  
Pedro Teixeira Castro ◽  
Luciana de Barros Duarte ◽  
Adauto Dutra Moraes Barbosa ◽  
Pedro Daltro ◽  
...  

2012 ◽  
Vol 40 (4) ◽  
pp. 1060-1062 ◽  
Author(s):  
Bernhard Resch ◽  
Melanie Halmer ◽  
Wilhelm D. Müller ◽  
Ernst Eber

2008 ◽  
Vol 32 (3) ◽  
pp. 300-300
Author(s):  
A. Galindo ◽  
R. Carpintero ◽  
D. Escribano ◽  
O. Nieto ◽  
J. Arbues ◽  
...  

Author(s):  
Michiel F. Schreuder

In renal agenesis, the kidney never forms due to an absence of interaction in the developing embryo between the ureteric bud and the metanephric mesenchyme. Most cases of renal agenesis are unilateral, for which the reported incidence is around 1 in 3000. The prenatal diagnosis of unilateral renal agenesis is based on the absence of a recognizable kidney, either at the normal or an ectopic site. However, such cases of an empty renal fossa can also be explained by an involuted multicystic dysplastic kidney, or by renal aplasia. Based on the hyperfiltration hypothesis and clinical studies, glomerular hyperfiltration can be expected, resulting in hypertension, albuminuria, and renal injury, for which long-term follow-up of all patients with renal agenesis is desirable.


Neurosurgery ◽  
2015 ◽  
Vol 78 (3) ◽  
pp. 370-380 ◽  
Author(s):  
Arthur André ◽  
Michel Zérah ◽  
Thomas Roujeau ◽  
Francis Brunelle ◽  
Thomas Blauwblomme ◽  
...  

Abstract BACKGROUND: Suprasellar arachnoid cysts (SAC) represent between 9% and 21% of pediatric arachnoid cysts. Recent improvements in magnetic resonance imaging, as well as increasing prenatal diagnosis, have allowed more precise knowledge and follow-up. OBJECTIVE: To describe a novel classification of SAC. METHODS: We present 35 cases of SAC treated between 1996 and 2014. Patient records and imaging studies were reviewed retrospectively to assess symptomatology, radiological findings, treatment, and long-term follow-up. RESULTS: Fourteen SAC were diagnosed prenatally (39%). We observed 15 (43%) cases presenting hydrocephalus (SAC-1) removing Liliequist membrane downward. Lower forms (SAC-2) with free third ventricle were observed in 11 (31%) cases. Asymmetrical forms (SAC-3) with Sylvian or temporal extension were seen in the 9 (26%) remaining patients. Twenty-three (66%) patients were treated by ventriculocisternostomy, 3 (8.5%) by shunt surgery, and 3 (8.5%) by craniotomy. Six (17%) patients had no surgery, including 5 cases (14%) that had prenatal diagnosis. Outcomes were initially favorable in 26 cases (87%). Eight (22%) patients had endocrine abnormalities at the end of the follow-up, 3 (8.5%) had developmental delay, and 6 (17%) had minor neuropsychological disturbances. CONCLUSION: SAC are heterogeneous entities. SAC-1 may come from an expansion of the diencephalic leaf of the Liliequist membrane. SAC-2 show a dilatation of the interpeduncular cistern and correspond to a defect of the mesencephalic leaf of the Liliequist membrane. SAC-3 correspond to the asymmetrical forms expanding to other subarachnoid spaces. Surgical treatment is not always necessary. The recognition of the different subtypes will allow choosing the best treatment option.


2019 ◽  
Vol 42 ◽  
Author(s):  
John P. A. Ioannidis

AbstractNeurobiology-based interventions for mental diseases and searches for useful biomarkers of treatment response have largely failed. Clinical trials should assess interventions related to environmental and social stressors, with long-term follow-up; social rather than biological endpoints; personalized outcomes; and suitable cluster, adaptive, and n-of-1 designs. Labor, education, financial, and other social/political decisions should be evaluated for their impacts on mental disease.


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