scholarly journals P26.23: The termination of pregnancy under ultrasound control in cases of bilateral renal agenesis, multicystic dysplasia and infantile polycystic disease

2007 ◽  
Vol 30 (4) ◽  
pp. 552-552
Author(s):  
L. Teregulova ◽  
A. Zalaletdinov ◽  
N. Golovanova ◽  
O. Evgrafov
Keyword(s):  
Renal Agenesis ◽  
Termination Of Pregnancy ◽  
Polycystic Disease ◽  
Multicystic Dysplasia

Transient Nephromegaly Simulating Infantile Polycystic Disease of the Kidneys

PEDIATRICS ◽  
1981 ◽  
Vol 67 (4) ◽  
pp. 554-559
Author(s):  
F. Bruder Stapleton ◽  
Saskia Hilton ◽  
John Wilcox ◽  
George R. Leopold
Keyword(s):  
Kidney Disease ◽  
Polycystic Kidney Disease ◽  
Poor Prognosis ◽  
Obstructive Uropathy ◽  
Good Health ◽  
Polycystic Kidney ◽  
Kidney Size ◽  
Excretory Urogram ◽  
Polycystic Disease ◽  
Multicystic Dysplasia

The discovery of bilaterally enlarged kidneys near the time of birth often portends a poor prognosis for renal function. Important diagnostic considerations include obstructive uropathy, renal venous thromboses, Wilms'tumor, infantile polycystic kidney disease, and renal multicystic dysplasia. In this report, we describe a child in whom enlarged bilateral kidneys were noted during the first day of life and to whom a presumptive clinical diagnosis of infantile polycystic kidney disease was incorrectly assigned, primarily on the basis of the excretory urogram. By 6 weeks of age, however, the kidney size was normal, and at 2 years of age, the boy remains in good health with radiographically normal kidneys. CASE REPORT

Renal agenesis, multicystic dysplasia, and uretero-pelvic junction obstruction— a common pathogenesis?

1994 ◽  
Vol 53 (3) ◽  
pp. 302-302 ◽  
Author(s):  
Wm. Lane M. Robson ◽  
R. Curtis Rogers ◽  
Alexander K. C. Leung
Keyword(s):  
Renal Agenesis ◽  
Common Pathogenesis ◽  
Multicystic Dysplasia

Polycystic Disease of the Liver: A Study of 24 Cases

1952 ◽  
Vol 20 (1) ◽  
pp. 60-78 ◽  
Author(s):  
Mandred W. Comfort ◽  
Howard K. Gray ◽  
David C. Dahlin ◽  
Frank B. Whitesell
Keyword(s):  
Polycystic Disease

ZUR BEDEUTUNG DES HYPOPHYSÄREN LUTEINISIERUNGSHORMONS FÜR SCHWANGERSCHAFT, GEBURT UND LACTATION BEI DER RATTE

1970 ◽  
Vol 65 (3_Suppl) ◽  
pp. S5-S32 ◽  
Author(s):  
K. Loewit
Keyword(s):  
Luteinizing Hormone ◽  
Early Pregnancy ◽  
Hydroxysteroid Dehydrogenase ◽  
The State ◽  
Termination Of Pregnancy ◽  
Progesterone Level ◽  
Corpora Lutea ◽  
Regulatory Properties ◽  
Duration Of Pregnancy

ABSTRACT The role of luteinizing hormone (LH) for the maintenance of pregnancy, parturition and lactation was investigated by immunological and histochemical methods in the rat. Neutralisation of endogenous rat-LH with Rabbit-Anti-Bovine-LH-Serum (selective hypophysectomy) from days 7-12 of pregnancy resulted in reabsorption of the foetuses and the reappearance of strong 20α-hydroxysteroid-dehydrogenase (20α-OHSD) activity in the corpora lutea (CL) of pregnancy, which normally show no such activity at that time. This effect could be prevented in part by concurrent pregnenolone administration and fully by progesterone, but was not influenced by oestrogen or prolactin. It is concluded that in early pregnancy LH is the main luteotrophic hormone in the rat even though prolactin might act synergistically with it. Antiserum treatment after the 12th day of gestation had no influence on the state or duration of pregnancy or on parturition. LH-injections during the first half of pregnancy had no luteolytic effects i. e. they did not activate 20α-OHSD activity. After day 16 they advanced the reappearance of the enzyme, but delayed parturition or resulted in stillbirths. Neither LH nor antiserum seemed to alter lactation. Since progesterone prevented both the termination of pregnancy and the recurrence of 20α-OHSD activity, it should have some regulatory properties on the enzyme. It is discussed whether the gonadotrophin-dependent progesterone level could regulate the 20α-OHSD activity rather than result from it.

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