scholarly journals Erratum: Inheritance mode and metabolic mechanism of the sulfoximine insecticide sulfoxaflor resistance in Oxycarenus hyalinipennis (Costa)

2021 ◽  
Vol 77 (9) ◽  
pp. 4201-4201
Keyword(s):  
Inheritance Mode ◽  
Metabolic Mechanism ◽  
Oxycarenus Hyalinipennis

scholarly journals The Synergistic Role of Diet and Exercise in the Prevention, Pathogenesis, and Management of Ulcerative Colitis: An Underlying Metabolic Mechanism

2019 ◽  
Vol 12 ◽  
pp. 117863881983452 ◽  
Author(s):  
Jonah Stavsky ◽  
Radhashree Maitra
Keyword(s):  
Ulcerative Colitis ◽  
Aerobic Exercise ◽  
Exercise Frequency ◽  
Simultaneous Reduction ◽  
Diet And Exercise ◽  
Literature Reviews ◽  
Amino Acid Intake ◽  
Metabolic Mechanism ◽  
Immunological Research

Ulcerative colitis (UC) is a biologically complex condition characterized by chronic, relapsing inflammation of the gastrointestinal tract. The relative incidence of this debilitating condition is increasing and sociologically damaging outcomes are a continued reality. Several etiological theories for UC are currently under investigation, spanning between genetic and environmental determinants. From an environmental perspective, previous literature reviews have demonstrated the independent effectiveness of specific diet and exercise patterns in modifying UC immuno-pathophysiology. This article explores the synergistic role of diet and aerobic exercise in the prevention, pathogenesis, and management of UC in the context of recent immunological research. Through a unifying mechanism—that is, microbial influence of colonic inflammation and immuno-pathophysiology—the simultaneous reduction of pro-inflammatory dietary sulfurous amino acid intake (ie methionine, cysteine, homocysteine, and taurine) and the upregulation of aerobic exercise frequency (which spurs the colonization of anti-inflammatory butyrate, acetate, and propionate producing microbial taxa) demonstrate the clinical efficacy of incorporating both diet and exercise modifications for UC prevention and management through pathogenic alterations.

Genetic and Nongenetic Bases for the L-Shaped Distribution of Quantitative Trait Loci Effects

Genetics ◽  
2001 ◽  
Vol 157 (4) ◽  
pp. 1773-1787 ◽  
Author(s):  
Bruno Bost ◽  
Dominique de Vienne ◽  
Frédéric Hospital ◽  
Laurence Moreau ◽  
Christine Dillmann
Keyword(s):  
Linkage Disequilibrium ◽  
Quantitative Trait Loci ◽  
Population Size ◽  
Quantitative Trait ◽  
Metabolic Flux ◽  
Genetic Model ◽  
Small Population ◽  
Additive Genetic Model ◽  
Metabolic Mechanism ◽  
Qtl Effects

Abstract The L-Shaped distribution of estimated QTL effects (R2) has long been reported. We recently showed that a metabolic mechanism could account for this phenomenon. But other nonexclusive genetic or nongenetic causes may contribute to generate such a distribution. Using analysis and simulations of an additive genetic model, we show that linkage disequilibrium between QTL, low heritability, and small population size may also be involved, regardless of the gene effect distribution. In addition, a comparison of the additive and metabolic genetic models revealed that estimates of the QTL effects for traits proportional to metabolic flux are far less robust than for additive traits. However, in both models the highest R2's repeatedly correspond to the same set of QTL.

Characterization of a protein-bound polysaccharide from Herba Epimedii and its metabolic mechanism in chronic fatigue syndrome

2017 ◽  
Vol 203 ◽  
pp. 241-251 ◽  
Author(s):  
Aiping Chi ◽  
Zhimei Shen ◽  
Wenfei Zhu ◽  
Yuliang Sun ◽  
Yijiang Kang ◽  
...  
Keyword(s):  
Chronic Fatigue Syndrome ◽  
Chronic Fatigue ◽  
Fatigue Syndrome ◽  
Herba Epimedii ◽  
Metabolic Mechanism
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