The crystal structure of type III effector protein XopQ fromXanthomonas oryzaecomplexed with adenosine diphosphate ribose

2014 ◽  
Vol 82 (11) ◽  
pp. 2910-2914 ◽  
Author(s):  
Sangheon Yu ◽  
Ingyu Hwang ◽  
Sangkee Rhee
Keyword(s):  
Crystal Structure ◽  
Adenosine Diphosphate ◽  
Effector Protein ◽  
Type Iii Effector ◽  
Type Iii

scholarly journals Genetic and molecular evidence that the Pseudomonas syringae type III effector protein AvrRpt2 is a cysteine protease

2003 ◽  
Vol 49 (6) ◽  
pp. 1537-1546 ◽  
Author(s):  
Michael J. Axtell ◽  
Stephen T. Chisholm ◽  
Douglas Dahlbeck ◽  
Brian J. Staskawicz
Keyword(s):  
Pseudomonas Syringae ◽  
Cysteine Protease ◽  
Molecular Evidence ◽  
Effector Protein ◽  
Type Iii Effector ◽  
Type Iii

scholarly journals Functional Dissection of SseF, a Type III Effector Protein Involved in Positioning the Salmonella-Containing Vacuole

Traffic ◽  
2006 ◽  
Vol 7 (8) ◽  
pp. 950-965 ◽  
Author(s):  
Garth L. Abrahams ◽  
Petra Müller ◽  
Michael Hensel
Keyword(s):  
Effector Protein ◽  
Type Iii Effector ◽  
Type Iii

scholarly journals Characterization, Phylogeny, and Genome Analyses of Nonpathogenic Xanthomonas campestris Strains Isolated from Brassica Seeds

2020 ◽  
Vol 110 (5) ◽  
pp. 981-988 ◽  
Author(s):  
Yung-An Lee ◽  
Pei-Yu Yang ◽  
Shau-Chang Huang
Keyword(s):  
Type Iii Secretion ◽  
Xanthomonas Campestris ◽  
Pathogenicity Island ◽  
Whole Genome Sequence ◽  
Effector Protein ◽  
Type Iii Effector ◽  
Type Iii ◽  
Effector Genes ◽  
Genome Analyses ◽  
Xanthomonas Campestris Pv Campestris

Xanthomonads were detected by using the Xan-D(CCF) medium from the brassica seeds, and their pathogenicity was determined by plant inoculation tests. It was found that some seed lots were infested with Xanthomonas campestris pv. campestris, some with X. campestris pv. raphani, and some with nonpathogenic xanthomonads. The nonpathogenic xanthomonad strains were identified as X. campestris, and the multilocus sequence analysis showed that the nonpathogenic X. campestris strains were grouped together with pathogenic X. campestris, but not with nonpathogenic strains of X. arboricola. In addition, all isolated X. campestris pv. campestris and X. campestris pv. raphani strains were positive in the hrpF-PCR, but the nonpathogenic strains were negative. It was further found that nonpathogenic X. campestris strain nE1 does not contain the entire pathogenicity island (hrp gene cluster; type III secretion system) and all type III effector protein genes based on the whole genome sequence analyses. The nonpathogenic X. campestris strain nE1 could acquire the entire pathogenicity island from the endemic X. campestris pv. campestris and X. campestris pv. raphani strains by conjugation, but type III effector genes were not cotransferred. The studies showed that the nonpathogenic X. campestris strains indeed exist on the brassica seeds, but it could be differentiated by the PCR assays on the hrp and type III effector genes. Nevertheless, the nonpathogenic X. campestris strains cannot be ignored because they may be potential gene resources to increase genetic diversity in the endemic pathogenic X. campestris pv. campestris and X. campestris pv. raphani strains.

Distribution of espI among clinical enterohaemorrhagic and enteropathogenic Escherichia coli isolates

2004 ◽  
Vol 53 (11) ◽  
pp. 1145-1149 ◽  
Author(s):  
Rosanna Mundy ◽  
Claire Jenkins ◽  
Jun Yu ◽  
Henry Smith ◽  
Gad Frankel
Keyword(s):  
Escherichia Coli ◽  
Type Iii Secretion ◽  
Severe Disease ◽  
Pathogenicity Island ◽  
Effector Proteins ◽  
Effector Protein ◽  
Enteropathogenic Escherichia Coli ◽  
Type Iii Effector ◽  
Type Iii

Enterohaemorrhagic (EHEC) and enteropathogenic (EPEC) Escherichia coli are important diarrhoeagenic pathogens; infection is dependent on translocation of a number of type III effector proteins. Until recently all the known effectors were encoded on the LEE pathogenicity island, which also encodes the adhesin intimin and the type III secretion apparatus. Recently, a novel non-LEE effector protein, EspI/NleA, which is required for full virulence in vivo and is encoded on a prophage, was identified. The aim of this study was to determine the distribution of espI among clinical EHEC and EPEC isolates. espI was detected in 86 % and 53 % of LEE+ EHEC and EPEC strains, respectively. Moreover, the espI gene was more commonly found in patients suffering from a more severe disease.

scholarly journals TccP is an enterohaemorrhagic Escherichia coli O157:H7 type III effector protein that couples Tir to the actin-cytoskeleton+

2004 ◽  
Vol 6 (12) ◽  
pp. 1167-1183 ◽  
Author(s):  
Junkal Garmendia ◽  
Alan D. Phillips ◽  
Marie-France Carlier ◽  
Yuwen Chong ◽  
Stephanie Schuller ◽  
...  
Keyword(s):  
Escherichia Coli ◽  
Actin Cytoskeleton ◽  
Effector Protein ◽  
Escherichia Coli O157 ◽  
Type Iii Effector ◽  
Type Iii ◽  
Enterohaemorrhagic Escherichia Coli

scholarly journals The Solution Structure of Type III Effector Protein AvrPto Reveals Conformational and Dynamic Features Important for Plant Pathogenesis

Structure ◽  
2004 ◽  
Vol 12 (7) ◽  
pp. 1257-1268 ◽  
Author(s):  
Jennifer Wulf ◽  
Pete E Pascuzzi ◽  
Amr Fahmy ◽  
Gregory B Martin ◽  
Linda K Nicholson
Keyword(s):  
Solution Structure ◽  
Effector Protein ◽  
Dynamic Features ◽  
Type Iii Effector ◽  
Type Iii ◽  
Plant Pathogenesis
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