Therapeutic efficacy of milrinone in the management of enterovirus 71-induced pulmonary edema

2005 ◽  
Vol 39 (3) ◽  
pp. 219-223 ◽  
Author(s):  
Shih-Min Wang ◽  
Huan-Yao Lei ◽  
Mei-Chih Huang ◽  
Jing-Min Wu ◽  
Chun-Ta Chen ◽  
...  
Keyword(s):  
Pulmonary Edema ◽  
Therapeutic Efficacy ◽  
Enterovirus 71

scholarly journals Association Between Angiopoietin-2 and Enterovirus 71 Induced Pulmonary Edema

2015 ◽  
Vol 83 (5) ◽  
pp. 391-396
Author(s):  
Zhijiang Qi ◽  
Zhi Li ◽  
Dong Hao ◽  
Tao Wang ◽  
Yongfu Xia ◽  
...  
Keyword(s):  
Pulmonary Edema ◽  
Enterovirus 71 ◽  
Angiopoietin 2

scholarly journals Mast cells contribute to Enterovirus 71 infection-induced pulmonary edema in neonatal mice

2018 ◽  
Vol 98 (8) ◽  
pp. 1039-1051 ◽  
Author(s):  
Yuefei Jin ◽  
Chao Zhang ◽  
Hui Wang ◽  
Guangyuan Zhou ◽  
Xiangpeng Wang ◽  
...  
Keyword(s):  
Mast Cells ◽  
Pulmonary Edema ◽  
Enterovirus 71 ◽  
Neonatal Mice ◽  
Enterovirus 71 Infection

Survival after pulmonary edema due to enterovirus 71 encephalitis

Neurology ◽  
2003 ◽  
Vol 60 (10) ◽  
pp. 1651-1656 ◽  
Author(s):  
M. A. Nolan ◽  
M. E. Craig ◽  
M. M. Lahra ◽  
W. D. Rawlinson ◽  
P. C. Prager ◽  
...  
Keyword(s):  
Pulmonary Edema ◽  
Enterovirus 71

scholarly journals Neurological Perspectives of Neurogenic Pulmonary Edema

2019 ◽  
Vol 81 (1-2) ◽  
pp. 94-102 ◽  
Author(s):  
Josef Finsterer
Keyword(s):  
Respiratory Distress ◽  
Pulmonary Edema ◽  
Enterovirus 71 ◽  
Neurogenic Pulmonary Edema ◽  
Brainstem Encephalitis ◽  
Simultaneous Treatment ◽  
X Ray ◽  
Subarachnoid Bleeding ◽  
The Central Nervous System ◽  
Intra Cranial Pressure

Background: Neurogenic pulmonary edema (NPE) is characterized by acute respiratory distress triggered by acute, severe compromise of the central nervous system (CNS). This review aims at summarizing and discussing recent and previous findings about the type and frequency of CNS triggers of NPE, pathogenesis, diagnosis, treatment, and outcome of patients experiencing NPE. Key Messages: NPE is diagnosed in the presence of pink, frothy sputum, pulmonary edema, bilateral opacities on X-ray, PaO2:PiO2 <200 mm Hg, acute CNS compromise with increased intra-cranial pressure, rapid resolution within 48–72 h, and the absence of alternative causes of respiratory distress. The most common cerebral triggers of NPE include enterovirus-71-associated brainstem encephalitis, subarachnoid bleeding, intracerebral bleeding, traumatic brain injury, epilepsy, ischemic stroke, intracranial/spinal surgery, multiple sclerosis, electroconvulsive therapy, subdural/epidural hematoma, intoxication, hypoxia, and hydrocephalus. Simultaneous treatment of CNS and pulmonary compromise is required. Cerebral treatment involves infectiologists, neurologists, and neurosurgeons. Pulmonary treatment is mainly supportive, but if ineffective, extracorporeal membrane oxygenation or thermodilution are alternative options. Applying intensive care measures, the outcome of NPE has improved. Summary: CNS-disease triggering NPE is more variegated than anticipated. Delineation of NPE from other pulmonary or cardiac conditions mimicking NPE is crucial to take appropriate measures and improve the outcome of these patients.

scholarly journals Mechanism of Fulminant Pulmonary Edema Caused by Enterovirus 71

2004 ◽  
Vol 38 (12) ◽  
pp. 1784-1788 ◽  
Author(s):  
S. J. Kao ◽  
F. L. Yang ◽  
Y. H. Hsu ◽  
H. I. Chen
Keyword(s):  
Pulmonary Edema ◽  
Enterovirus 71
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