Position modifies carotid artery blood flow velocity during straining

1990 ◽  
Vol 13 (2) ◽  
pp. 69-76
Author(s):  
Barbara Therrien
Vascular ◽  
2020 ◽  
pp. 170853812096392
Author(s):  
Arda Aybars Pala ◽  
Yusuf Salim Urcun

Objectives The increase in carotid artery blood flow velocity is a measure of the severity of the carotid artery stenosis caused by atherosclerosis. Carotid artery stenosis is progressive and is of great importance due to the risk of stroke it creates. As an alternative to radiological examinations in these patients, patient follow-up can be facilitated by associating novel laboratory parameters with the severity of stenosis. In this study, we aimed to investigate the effect of the calculated plasma osmolality and atherogenic index of plasma on carotid artery blood flow velocities in patients with carotid artery stenosis. Methods A total of 161 patients diagnosed with carotid artery stenosis who admitted to our clinic between May 2018 and May 2020 were retrospectively analyzed. According to the carotid artery blood flow velocities measured with the Doppler ultrasonography, the patients were divided into two groups as “Normal flow velocity group” ( n = 62) and “Increased flow velocity group” ( n = 99). Results The calculated plasma osmolality, atherogenic index of plasma, and mean platelet volume were significantly associated with increased carotid artery blood flow velocity ( p < 0.001, p < 0.001, p = 0.006; respectively). Calculated plasma osmolality and atherogenic index of plasma were identified as independent predictors of increase in carotid artery blood flow velocity ( p < 0.001, p < 0.001; respectively). In the Receiver Operating Characteristic curve analysis, the plasma osmolality cut-off value, which predicts the increase in carotid artery blood flow velocity was found to be 291.45 mOsm/kg (Area Under the Curve: 0.746, p < 0.001, 65.7% sensitivity, and 67.7% specificity), and atherogenic index of plasma cut-off value was 0.20 (Area Under the Curve: 0.735, p < 0.001, 65.7% sensitivity, and 66.1% specificity). Conclusion There was a significant relationship between the increase in carotid artery blood flow velocity and the increase in plasma osmolality and atherogenic index of plasma values. As a result, we can predict the increase in carotid artery blood flow velocity, that is, the increase in the severity of the carotid artery stenosis, with plasma osmolality and atherogenic index of plasma values that can be calculated simply from routine biochemical tests.


Stroke ◽  
1989 ◽  
Vol 20 (10) ◽  
pp. 1327-1330 ◽  
Author(s):  
G Jungquist ◽  
B Nilsson ◽  
H Ostberg ◽  
S O Isacsson ◽  
L Janzon ◽  
...  

CHEST Journal ◽  
1973 ◽  
Vol 63 (4) ◽  
pp. 607-609 ◽  
Author(s):  
Hilton Buggs ◽  
Fred B. Balguma ◽  
Paul E. Johnson ◽  
Carter A. Printup ◽  
John R.F. Penido

1996 ◽  
Vol 81 (1) ◽  
pp. 413-418 ◽  
Author(s):  
G. Hellstrom ◽  
W. Fischer-Colbrie ◽  
N. G. Wahlgren ◽  
T. Jogestrand

Factors controlling cerebral blood flow (CBF) during exercise are complex and incompletely known. Different techniques have shown partly contradictory results of changes in regional and global cerebral perfusion during dynamic exercise in healthy subjects. To elucidate the global CBF response to supine stepwise increasing physical exercise, we measured blood flow in the left common carotid artery (QCCA) and the left internal carotid artery (QICA) simultaneously with the blood flow velocity in the ipsilateral middle cerebral artery (VMCA) using duplex ultrasonography and transcranial Doppler ultrasonography. During moderate exercise intensity (60-67% of maximal capacity), the VMCA increased 14% (P < 0.001), the QICA 17% (P < 0.01), and the QCCA 33% (P < 0.001) compared with baseline values. High physical exercise intensity (80-90% of maximal capacity) tended to reduce VMCA and QICA compared with moderate exercise, in contrast to a continued increase in QCCA. The results indicate an increased global CBF during exercise. This increase was reduced during hard exercise due to a decrease of the arterial PCO2 secondary to hyperventilation.


Stroke ◽  
1974 ◽  
Vol 5 (1) ◽  
pp. 60-67 ◽  
Author(s):  
ALBERTO BENCHIMOL ◽  
JOSE BALDI ◽  
KENNETH B. DESSER

Maturitas ◽  
2021 ◽  
Vol 148 ◽  
pp. 24-32
Author(s):  
Stefanie L Ruediger ◽  
Jodie L Koep ◽  
Shelley E Keating ◽  
Faith K Pizzey ◽  
Jeff S Coombes ◽  
...  

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