scholarly journals Fast inversion recovery magnetic resonance angiography of the intracranial arteries

2010 ◽  
Vol 63 (6) ◽  
pp. 1648-1658 ◽  
Author(s):  
Ek T. Tan ◽  
John Huston ◽  
Norbert G. Campeau ◽  
Stephen J. Riederer
Keyword(s):  
Magnetic Resonance ◽  
Magnetic Resonance Angiography ◽  
Inversion Recovery ◽  
Intracranial Arteries ◽  
Fast Inversion

scholarly journals On the dual contrast enhancement mechanism in frequency-selective inversion-recovery magnetic resonance angiography (IRON-MRA)

2009 ◽  
Vol 62 (2) ◽  
pp. 314-324 ◽  
Author(s):  
Evert-jan Vonken ◽  
Grigorios Korosoglou ◽  
Jing Yu ◽  
Michael Schär ◽  
Ralph Weissleder ◽  
...  
Keyword(s):  
Magnetic Resonance ◽  
Magnetic Resonance Angiography ◽  
Contrast Enhancement ◽  
Inversion Recovery ◽  
Enhancement Mechanism ◽  
Selective Inversion ◽  
Frequency Selective

Cystic Encephalomalacia following Vasculopathy and Vasospasm of Proximal Intracranial Arteries Due to Pneumococcal Meningitis in a Infant

2018 ◽  
Vol 49 (03) ◽  
pp. 213-216 ◽  
Author(s):  
Wibke Janzarik ◽  
Peter Franck ◽  
Markus Hufnagel ◽  
Christopher Beck ◽  
Rudolf Korinthenberg ◽  
...  
Keyword(s):  
Blood Flow ◽  
Cerebral Blood Flow ◽  
Magnetic Resonance ◽  
Magnetic Resonance Angiography ◽  
Bacterial Meningitis ◽  
Effective Treatment ◽  
Pneumococcal Meningitis ◽  
Severe Disease ◽  
Treatment Resistant ◽  
Intracranial Arteries

AbstractDespite the availability of modern antibiotics, pneumococcal meningitis in both children and adults remains a severe disease—one known to frequently cause grave complications and residual disability. Although the appearance of arterial vasospasms in bacterial meningitis systematically has been investigated and reported on for adult patients, such research is lacking when it comes to infants. We report on a 4-week-old infant who, 6 days after onset of pneumococcal meningitis, suffered severe neurological deterioration with treatment-resistant seizures and coma. Generalized cortical and subcortical edema developed in conjunction with diminished cerebral blood flow, as depicted in magnetic resonance angiography and serial Doppler-sonographic examinations. The ischemia resulted in extensive cystic encephalomalacia. We propose that the degree of variation in cerebral blood flow in the acute phase was the result of an extensive arterial vasculopathy involving vasospasms. Awareness of this complication and prospective serial Doppler-sonographic examinations may improve our understanding of the connection between brain edema and vasculopathy. At present, however, no effective treatment appears available.

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