Wolfberry‐Derived Zeaxanthin Dipalmitate Attenuates Ethanol‐Induced Hepatic Damage

2019 ◽  
Vol 63 (11) ◽  
pp. 1801339 ◽  
Author(s):  
Hao Gao ◽  
Yi Lv ◽  
Yingxia Liu ◽  
Jingjing Li ◽  
Xiaogang Wang ◽  
...  
Keyword(s):  
Hepatic Damage ◽  
Zeaxanthin Dipalmitate

Atypical Alcohol Dehydrogenase in Chilean Alcohol Addicts With and Without Hepatic Damage

1970 ◽  
Vol 31 (3) ◽  
pp. 571-577 ◽  
Author(s):  
Guillermo Ugarte ◽  
María Eugenia Pino ◽  
Herbert Altschiller ◽  
Tamara Pereda
Keyword(s):  
Alcohol Dehydrogenase ◽  
Hepatic Damage

scholarly journals Potential antioxidant properties and hepatoprotective effects of Juniperus phoenicea berries against CCl 4 induced hepatic damage in rats

2017 ◽  
Vol 10 (3) ◽  
pp. 263-269 ◽  
Author(s):  
Amel Laouar ◽  
Fahima Klibet ◽  
Ezzeddine Bourogaa ◽  
Amel Benamara ◽  
Amel Boumendjel ◽  
...  
Keyword(s):  
Antioxidant Properties ◽  
Hepatic Damage ◽  
Juniperus Phoenicea ◽  
Hepatoprotective Effects

scholarly journals Role of Dietary Nutritional Treatment on Hepatic and Intestinal Damage in Transplantation with Steatotic and Non-Steatotic Liver Grafts from Brain Dead Donors

Nutrients ◽  
2021 ◽  
Vol 13 (8) ◽  
pp. 2554
Author(s):  
Marc Micó-Carnero ◽  
Araní Casillas-Ramírez ◽  
Albert Caballeria-Casals ◽  
Carlos Rojano-Alfonso ◽  
Alfredo Sánchez-González ◽  
...  
Keyword(s):  
Growth Factor ◽  
Gut Microbiota ◽  
Intestinal Inflammation ◽  
Mucosal Damage ◽  
Hepatic Damage ◽  
Intestinal Damage ◽  
Edema Formation ◽  
Steatotic Liver ◽  
Damage And Failure

Herein, we investigate whether: (1) the administration of glucose or a lipid emulsion is useful in liver transplantation (LT) using steatotic (induced genetically or nutritionally) or non-steatotic livers from donors after brain death (DBDs); and (2) any such benefits are due to reductions in intestinal damage and consequently to gut microbiota preservation. In recipients from DBDs, we show increased hepatic damage and failure in the maintenance of ATP, glycogen, phospholipid and growth factor (HGF, IGF1 and VEGFA) levels, compared to recipients from non-DBDs. In recipients of non-steatotic grafts from DBDs, the administration of glucose or lipids did not protect against hepatic damage. This was associated with unchanged ATP, glycogen, phospholipid and growth factor levels. However, the administration of lipids in steatotic grafts from DBDs protected against damage and ATP and glycogen drop and increased phospholipid levels. This was associated with increases in growth factors. In all recipients from DBDs, intestinal inflammation and damage (evaluated by LPS, vascular permeability, mucosal damage, TLR4, TNF, IL1, IL-10, MPO, MDA and edema formation) was not shown. In such cases, potential changes in gut microbiota would not be relevant since neither inflammation nor damage was evidenced in the intestine following LT in any of the groups evaluated. In conclusion, lipid treatment is the preferable nutritional support to protect against hepatic damage in steatotic LT from DBDs; the benefits were independent of alterations in the recipient intestine.

Protective Activity of Cubebin from Syringa oblata Against CCl4-Induced Hepatic Damage in Mice

2021 ◽  
Author(s):  
Zhiqiang Li ◽  
Junmao Li ◽  
Zhifeng Li ◽  
Haonan Zhang ◽  
Shilin Yang ◽  
...  
Keyword(s):  
Hepatic Damage ◽  
Protective Activity

scholarly journals Acute Lethal Toxicity, Hyperkalemia Associated with Renal Injury and Hepatic Damage after Intravenous Administration of Cadmium Nitrate in Rats

2007 ◽  
Vol 49 (1) ◽  
pp. 17-24 ◽  
Author(s):  
Emi Dote ◽  
Tomotaro Dote ◽  
Hiroyasu Shimizu ◽  
Yukari Shimbo ◽  
Michiko Fujihara ◽  
...  
Keyword(s):  
Intravenous Administration ◽  
Renal Injury ◽  
Hepatic Damage ◽  
Cadmium Nitrate ◽  
Lethal Toxicity
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