Pediatric dysphagia: Is interarytenoid mucosal height significant?

2019 ◽  
Vol 129 (11) ◽  
pp. 2588-2593 ◽  
Author(s):  
Reema Padia ◽  
Steven Coppess ◽  
David L. Horn ◽  
Sanjay R. Parikh ◽  
Jennifer Hoang ◽  
...  
Keyword(s):  
Pediatric Dysphagia

scholarly journals An Unusual Cause of Pediatric Dysphagia

2017 ◽  
Vol 4 ◽  
pp. 2333794X1668649
Author(s):  
Abha Kaistha ◽  
Jeremiah Levine
Keyword(s):  
Pediatric Dysphagia

scholarly journals Selective disruption of trigeminal sensory neurogenesis and differentiation in a mouse model of 22q11.2 deletion syndrome

2021 ◽  
pp. dmm.047357
Author(s):  
Beverly A. Karpinski ◽  
Thomas M. Maynard ◽  
Corey A. Bryan ◽  
Gelila Yitsege ◽  
Anelia Horvath ◽  
...  
Keyword(s):  
Mouse Model ◽  
Neural Crest ◽  
Sensory Neuron ◽  
Cranial Nerve ◽  
22Q11.2 Deletion Syndrome ◽  
Deletion Syndrome ◽  
22Q11.2 Deletion ◽  
Neuron Differentiation ◽  
Pediatric Dysphagia ◽  
Cell Cell

22q11.2 Deletion Syndrome (22q11DS) is a neurodevelopmental disorder associated with cranial nerve anomalies and disordered oropharyngeal function including pediatric dysphagia. Using the LgDel 22q11DS mouse model, we asked whether sensory neuron differentiation in the trigeminal ganglion (CNgV) , which is essential for normal orofacial function, is disrupted. We did not detect changes in cranial placode cell translocation or neural crest migration at early stages of LgDel CNgV development. As the ganglion coalesces, however, proportions of placode-derived LgDel CNgV cells increase relative to neural crest cells. In addition, local aggregation of placode-derived cells increases and aggregation of neural crest-derived cells decreases in LgDel CNgV. This change in cell-cell relationships was accompanied by altered proliferation of placode-derived cells at E9.5, and premature neurogenesis from neural crest-derived precursors, reflected by increased frequency of asymmetric neurogenic divisions for neural crest-derived precursors by E10.5. These early differences in LgDel CNgV genesis prefigure changes in sensory neuron differentiation and gene expression by P8, when early signs of cranial nerve dysfunction associated with pediatric dysphagia are observed in LgDel mice. Apparently, 22q11 deletion destabilizes CNgV sensory neuron genesis and differentiation by increasing variability in cell-cell interaction, proliferation, and sensory neuron differentiation. This early developmental divergence and its consequences may contribute to oropharyngeal dysfunction including suckling, feeding and swallowing disruptions at birth and additional orofacial sensory/motor deficits throughout life.

Acute Characteristics of Pediatric Dysphagia Subsequent to Traumatic Brain Injury

2002 ◽  
Vol 17 (3) ◽  
pp. 220-241 ◽  
Author(s):  
Angela Morgan ◽  
Elizabeth Ward ◽  
Bruce Murdoch ◽  
Kate Bilbie
Keyword(s):  
Traumatic Brain Injury ◽  
Brain Injury ◽  
Pediatric Dysphagia

Temporal Trends of Pediatric Dysphagia in Hospitalized Patients

Dysphagia ◽  
2018 ◽  
Vol 33 (5) ◽  
pp. 655-661 ◽  
Author(s):  
Joshua Horton ◽  
Carlyn Atwood ◽  
Sharon Gnagi ◽  
Ronald Teufel ◽  
Clarice Clemmens
Keyword(s):  
Temporal Trends ◽  
Hospitalized Patients ◽  
Pediatric Dysphagia

Instrumental Assessment of Pediatric Dysphagia

2017 ◽  
Vol 38 (02) ◽  
pp. 135-146 ◽  
Author(s):  
Joan Arvedson ◽  
Maureen Lefton-Greif
Keyword(s):  
Instrumental Assessment ◽  
Pediatric Dysphagia

The Role of Gastroesophageal Reflux in Pediatric Dysphagia

1991 ◽  
Vol 12 (2) ◽  
pp. 159-165 ◽  
Author(s):  
A. G. Catto-Smith ◽  
H. Machida ◽  
J. D. Butzner ◽  
D. G. Gall ◽  
R. B. Scott
Keyword(s):  
Gastroesophageal Reflux ◽  
Pediatric Dysphagia

MANAGEMENT OF PEDIATRIC DYSPHAGIA

1998 ◽  
Vol 31 (3) ◽  
pp. 453-476 ◽  
Author(s):  
Joan C. Arvedson
Keyword(s):  
Pediatric Dysphagia
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