scholarly journals Repetitive differential finger motion increases shear strain between the flexor tendon and subsynovial connective tissue

2013 ◽  
Vol 31 (10) ◽  
pp. 1533-1539 ◽  
Author(s):  
Jimmy Tat ◽  
Aaron M. Kociolek ◽  
Peter J. Keir
2009 ◽  
Vol 35 (12) ◽  
pp. 1973-1981 ◽  
Author(s):  
Yuichi Yoshii ◽  
Hector R. Villarraga ◽  
Jacqueline Henderson ◽  
Chunfeng Zhao ◽  
Kai-Nan An ◽  
...  

2009 ◽  
Vol 34 (1) ◽  
pp. 65-73 ◽  
Author(s):  
Yuichi Yoshii ◽  
Chunfeng Zhao ◽  
Jacqueline Henderson ◽  
Kristin D. Zhao ◽  
Kai-Nan An ◽  
...  

2016 ◽  
Vol 41 (8) ◽  
pp. 822-828 ◽  
Author(s):  
K. Moriya ◽  
T. Yoshizu ◽  
N. Tsubokawa ◽  
H. Narisawa ◽  
K. Hara ◽  
...  

We report the results of complete release of the entire A2 pulley after zone 2C flexor tendon repair followed by early postoperative active mobilization in seven fingers and their comparisons with 33 fingers with partial A2 pulley release. In seven fingers, release of the entire A2 pulley was necessary to allow free gliding of the repairs in five fingers and complete release of both the A2 and C1 pulleys was necessary in two. No bowstringing was clinically evident in any finger. Two fingers required tenolysis. Using Tang’s criteria, the function of two digits was ranked as excellent, four good and one fair; there was no failure. The functional return in these seven fingers was similar with that in 33 fingers with partial A2 pulley release; in these patients only one finger required tenolysis. Our results support the suggestion that release of the entire A2 pulley together with the adjacent C1 pulley does not clinically affect finger motion or cause tendon bowstringing, provided that the other pulleys are left intact. Level of evidence: IV


Hand ◽  
2007 ◽  
Vol 3 (1) ◽  
pp. 34-40 ◽  
Author(s):  
Sangho Oh ◽  
Anke M. Ettema ◽  
Chunfeng Zhao ◽  
Mark E. Zobitz ◽  
Kai-Nan An ◽  
...  

2019 ◽  
Vol 7 (3) ◽  
pp. e000808
Author(s):  
Michael Joseph De Cozar ◽  
Elizabeth Barr

A 12-year-old Irish sports horse gelding presented with a recently developed oval firm mass at the plantaromedial aspect of right tarsus, located between the gastrocnemius and deep digital flexor tendon. At presentation, the horse was mildly right hind lame, with no exacerbation with flexion. Radiography and ultrasonography revealed an ovoid mineralised mass within soft tissues plantaromedially to tarsus and distal tibia and not associated with tarsal sheath or calcaneal bursa. Due to mild intermittent lameness and concerns regarding possibility for further increase in size, the horse underwent en bloc removal of the calcified mass under general anaesthesia. Histopathology showed the mass contained fibrous reactive connective tissue, undergoing bone and cartilage metaplasia and degeneration, with a central area of calcified bone and surrounding region of mononuclear inflammation. These characteristics although reminiscent of calcinosis circumscripta were diagnosed as focal dystrophic calcification. The horse made a full recovery and returned to a normal exercise level.


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