Evidence for independent peripheral and central age‐related hearing impairment

2020 ◽  
Vol 98 (9) ◽  
pp. 1800-1814 ◽  
Author(s):  
Jianxin Bao ◽  
Yan Yu ◽  
Hui Li ◽  
John Hawks ◽  
Grace Szatkowski ◽  
...  
2020 ◽  
Vol 4 (Supplement_1) ◽  
pp. 797-797
Author(s):  
Nicholas Reed ◽  
Charlotte Yeh

Abstract Communication is fundamental to patient-centered care. However, sensory impairment limits communication among older adults. Specifically, hearing impairment strains communication via degraded auditory encoding while vision impairment distresses ability to read and interpret visual cues. The presence of dual sensory impairment, defined as concurrent hearing and vision impairment, may exacerbate these effects. The potential consequence s of age-related sensory loss on health care interactions and outcomes are beginning to surface in epidemiologic studies demonstrating poorer patient-provider communication, higher medical expenditures, increased risk of 30-day readmission, and longer length of stay when compared to individuals without sensory loss. Importantly, these associations may be amenable to intervention via sensory aids; however, uptake to sensory care is low. Notably, less than 20% of persons with hearing impairment have hearing aids and over 55% of Medicare Beneficiaries with reported vision problems have not had an eye examination in the prior year. Affordability and access may contribute to lack of sensory care uptake as Medicare explicitly excludes coverage of vision and hearing services. In this symposium, we will review current and new evidence for whether sensory loss affects health care outcomes, including satisfaction with care, incident delirium during hospitalization, navigation of Medicare, and present data on how persons with sensory loss are more likely to delay their care independent of cost and insurance factors suggesting fundamental changes in health care system interaction. We will place these results within the context of current national quality care and policy initiatives and review methods to address sensory loss.


2017 ◽  
Vol 29 (11) ◽  
pp. 1771-1784 ◽  
Author(s):  
Annie Pye ◽  
Anna Pavlina Charalambous ◽  
Iracema Leroi ◽  
Chrysoulla Thodi ◽  
Piers Dawes

ABSTRACTBackground:Cognitive screening tests frequently rely on items being correctly heard or seen. We aimed to identify, describe, and evaluate the adaptation, validity, and availability of cognitive screening and assessment tools for dementia which have been developed or adapted for adults with acquired hearing and/or vision impairment.Method:Electronic databases were searched using subject terms “hearing disorders” OR “vision disorders” AND “cognitive assessment,” supplemented by exploring reference lists of included papers and via consultation with health professionals to identify additional literature.Results:1,551 papers were identified, of which 13 met inclusion criteria. Four papers related to tests adapted for hearing impairment; 11 papers related to tests adapted for vision impairment. Frequently adapted tests were the Mini-Mental State Examination (MMSE) and the Montreal Cognitive Assessment (MOCA). Adaptations for hearing impairment involved deleting or creating written versions for hearing-dependent items. Adaptations for vision impairment involved deleting vision-dependent items or spoken/tactile versions of visual tasks. No study reported validity of the test in relation to detection of dementia in people with hearing/vision impairment. Item deletion had a negative impact on the psychometric properties of the test.Conclusions:While attempts have been made to adapt cognitive tests for people with acquired hearing and/or vision impairment, the primary limitation of these adaptations is that their validity in accurately detecting dementia among those with acquired hearing or vision impairment is yet to be established. It is likely that the sensitivity and specificity of the adapted versions are poorer than the original, especially if the adaptation involved item deletion. One solution would involve item substitution in an alternative sensory modality followed by re-validation of the adapted test.


2016 ◽  
pp. 1003 ◽  
Author(s):  
Masoumeh Falah ◽  
Mohammad Najafi ◽  
Massoud Houshmand ◽  
Mohammad Farhadi

1994 ◽  
Vol 75 (1-2) ◽  
pp. 27-37 ◽  
Author(s):  
Yuko Saitoh ◽  
Masanori Hosokawa ◽  
Atsuyoshi Shimada ◽  
Yoshiaki Watanabe ◽  
Norio Yasuda ◽  
...  

2015 ◽  
Vol 21 (4) ◽  
pp. 248
Author(s):  
ShubhangiKailas Pingle ◽  
LuckyRamesh Thakkar ◽  
DivyaVishambhar Kumbhakar ◽  
ArunaAnil Jawade ◽  
RajaniGanpatrao Tumane ◽  
...  

PLoS ONE ◽  
2015 ◽  
Vol 10 (9) ◽  
pp. e0137493 ◽  
Author(s):  
Tohru Tanigawa ◽  
Rei Shibata ◽  
Kazuhisa Kondo ◽  
Nobuyuki Katahira ◽  
Takahiro Kambara ◽  
...  

2017 ◽  
Vol 355 ◽  
pp. 97-101 ◽  
Author(s):  
Laura Kytövuori ◽  
Samuli Hannula ◽  
Elina Mäki-Torkko ◽  
Martti Sorri ◽  
Kari Majamaa

2014 ◽  
Vol 2014 ◽  
pp. 1-10 ◽  
Author(s):  
Yasue Uchida ◽  
Saiko Sugiura ◽  
Michihiko Sone ◽  
Hiromi Ueda ◽  
Tsutomu Nakashima

Age-related hearing impairment (ARHI) is a complex, multifactorial disorder that is attributable to confounding intrinsic and extrinsic factors. The degree of impairment shows substantial variation between individuals, as is also observed in the senescence of other functions. This individual variation would seem to refute the stereotypical view that hearing deterioration with age is inevitable and may indicate that there is ample scope for preventive intervention. Genetic predisposition could account for a sizable proportion of interindividual variation. Over the past decade or so, tremendous progress has been made through research into the genetics of various forms of hearing impairment, including ARHI and our knowledge of the complex mechanisms of auditory function has increased substantially. Here, we give an overview of recent investigations aimed at identifying the genetic risk factors involved in ARHI and of what we currently know about its pathophysiology. This review is divided into the following sections: (i) genes causing monogenic hearing impairment with phenotypic similarities to ARHI; (ii) genes involved in oxidative stress, biologic stress responses, and mitochondrial dysfunction; and (iii) candidate genes for senescence, other geriatric diseases, and neurodegeneration. Progress and prospects in genetic research are discussed.


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