scholarly journals Artifact-reduced two-dimensional cine steady state free precession for myocardial blood- oxygen-level-dependent imaging

2010 ◽  
Vol 31 (4) ◽  
pp. 863-871 ◽  
Author(s):  
Xiangzhi Zhou ◽  
Sotirios A. Tsaftaris ◽  
Ying Liu ◽  
Richard Tang ◽  
Rachel Klein ◽  
...  
Keyword(s):  
Steady State ◽  
Steady State Free Precession ◽  
Blood Oxygen Level Dependent ◽  
Blood Oxygen ◽  
Two Dimensional ◽  
Oxygen Level ◽  
Free Precession ◽  
Blood Oxygen Level

T2-prepared Steady-State Free Precession Blood Oxygen Level–Dependent MR Imaging of Myocardial Perfusion in a Dog Stenosis Model

Radiology ◽  
2005 ◽  
Vol 236 (2) ◽  
pp. 503-509 ◽  
Author(s):  
Steven M. Shea ◽  
David S. Fieno ◽  
Brian E. Schirf ◽  
Xiaoming Bi ◽  
Jie Huang ◽  
...  
Keyword(s):  
Steady State ◽  
Myocardial Perfusion ◽  
Mr Imaging ◽  
Steady State Free Precession ◽  
Blood Oxygen Level Dependent ◽  
Blood Oxygen ◽  
Oxygen Level ◽  
Free Precession ◽  
Blood Oxygen Level

scholarly journals Quantitative relations between transient BOLD responses, cortical energetics, and impulse firing in different cortical regions

2019 ◽  
Vol 122 (3) ◽  
pp. 1226-1237 ◽  
Author(s):  
M. R. Bennett ◽  
L. Farnell ◽  
W. G. Gibson
Keyword(s):  
Blood Flow ◽  
Steady State ◽  
Neuronal Activity ◽  
Blood Oxygen Level Dependent ◽  
Blood Oxygen ◽  
Bold Signal ◽  
Oxygen Level ◽  
Blood Oxygen Level ◽  
Baseline Activity ◽  
Input Activity

The blood oxygen level-dependent (BOLD) functional magnetic resonance imaging signal arises as a consequence of changes in blood flow (cerebral blood flow) and oxygen usage (cerebral metabolic rate of oxygen) that in turn are modulated by changes in neuronal activity. Much attention has been given to both theoretical and experimental aspects of the energetics but not to the neuronal activity. Here we use our previous theory relating the steady-state BOLD signal to neuronal activity and amalgamate it with the standard dynamic causal model (DCM, Friston) theory to produce a quantitative model relating the time-dependent BOLD signal to the underlying neuronal activity. Unlike existing treatments, this new theory incorporates a nonzero baseline activity in a completely consistent way and is thus able to account for both positive and negative BOLD signals. It can reproduce a wide variety of experimental BOLD signals reported in the literature solely by adjusting the neuronal input activity. In this way it provides support for the claim that the main features of the signals, including poststimulus undershoot and overshoot, are principally a result of changes in neuronal activity. NEW & NOTEWORTHY A previous model relating the steady-state blood oxygen level-dependent (BOLD) signal to neuronal activity, both above and below baseline, is extended to account for transient BOLD signals. This allows for a detailed investigation of the role neuronal activity can play in such signals and also encompasses poststimulus undershoot and overshoot. A wide variety of experimental BOLD signals are reproduced solely by adjusting the neuronal input activity, including recent results regarding the BOLD signal in patients with schizophrenia.

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