Spinal ganglia and peripheral nerves innervating the regenerating tail and muscles of lizards

2021 ◽  
Author(s):  
Lorenzo Alibardi
Keyword(s):  
Peripheral Nerves ◽  
Spinal Ganglia

Spinal ganglia and peripheral nerves from a patient with Tay-Sachs disease

1985 ◽  
Vol 66 (3) ◽  
pp. 239-244 ◽  
Author(s):  
T. Abe ◽  
K. Ogawa ◽  
H. Fuziwara ◽  
K. Urayama ◽  
K. Nagashima
Keyword(s):  
Peripheral Nerves ◽  
Spinal Ganglia ◽  
Tay Sachs Disease

Syndrome of Painful Legs and Moving Toes

2001 ◽  
Vol 91 (7) ◽  
pp. 361-364 ◽  
Author(s):  
Joonhyun Yoon ◽  
Christopher Crabtree ◽  
Georgeanne Botek
Keyword(s):  
Peripheral Nerves ◽  
Lower Extremities ◽  
Spinal Ganglia ◽  
Nerve Roots ◽  
Involuntary Movements

Painful legs and moving toes syndrome is characterized by spontaneous causalgic pain in the lower extremities associated with peculiar involuntary movements of the lower extremities, especially the toes and feet. The pain is diffuse, intractable, aching, and deep. The movements consist of persistent writhing movements in the digits that cannot be limited voluntarily. The syndrome has been observed after a variety of abnormalities affecting the posterior nerve roots, the spinal ganglia, and the peripheral nerves. This article reviews commonly reported findings and current concepts in the etiology and management of this condition. (J Am Podiatr Med Assoc 91(7): 361-364, 2001)

scholarly journals ALLERGIC NEURITIS: AN EXPERIMENTAL DISEASE OF RABBITS INDUCED BY THE INJECTION OF PERIPHERAL NERVOUS TISSUE AND ADJUVANTS

1955 ◽  
Vol 102 (2) ◽  
pp. 213-236 ◽  
Author(s):  
Byron H. Waksman ◽  
Raymond D. Adams
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Sciatic Nerve ◽  
Cell Count ◽  
Peripheral Nerves ◽  
Skin Tests ◽  
Spinal Ganglia ◽  
The Central Nervous System ◽  
Experimental Allergic ◽  
Allergic Neuritis

In experimental allergic encephalomyelitis (EAE), produced by injecting rabbits with whole rabbit spinal cord together with tubercle bacilli and mineral oil, lesions comparable to those seen in the central nervous system are found in the nerve roots, spinal ganglia, and peripheral nerves. When special fractions of bovine white matter are used as antigen in rabbits, the same distribution of lesions is seen but peripheral nerve involvement is relatively less frequent. When rabbit sciatic nerve or spinal ganglia are used as antigen in rabbits, lesions occur only in the roots, ganglia, and peripheral nerves. Lesions are not produced in the central nervous system, nor is there a meningitis. This disease picture has been called experimental allergic neuritis (EAN). The antigenicity of rabbit nerve is not impaired by autoclaving. Sciatic nerve of other mammalian species produces the same disease in rabbits as does rabbit nerve. Optic nerve, used as antigen, produces the typical picture of EAE, not EAN. The optic nerves are not affected in EAN, whereas they commonly contain lesions in EAE. There are differences of symptomatology, referable to the difference in distribution of lesions, between EAE and EAN. The spinal fluid of EAE shows an increase both in the number of cells and in the total protein content. In EAN, the same changes in protein are observed, but usually the cell count remains normal. The cell count appears to be related to the involvement of cerebral and spinal meninges, which is an almost invariable accompaniment of EAE. The skin tests and serologic studies made with homologous and heterologous antigens were essentially non-contributory, apparently as a consequence of the diversity of antigens present in the inoculated materials. The similarity between EAN and certain of the human polyneuritides is indicated and discussed.

A Familial Peripheral Neuropathy and Glomerulopathy in Gelbvieh Calves

2003 ◽  
Vol 40 (1) ◽  
pp. 63-70 ◽  
Author(s):  
R. J. Panciera ◽  
K. E. Washburn ◽  
R. N. Streeter ◽  
J. G. Kirkpatrick
Keyword(s):  
Peripheral Neuropathy ◽  
Peripheral Nerves ◽  
Pedigree Analysis ◽  
Spinal Nerve ◽  
Ventral Horn ◽  
Spinal Ganglia ◽  
Limb Ataxia ◽  
Familial Relationship ◽  
Spinal Cord Cell ◽  
Glomerular Lesions

Nine Gelbvieh calves originating in four herds and clinically presenting with rear limb ataxia/ paresis had histopathologically confirmed peripheral neuropathy and a proliferative glomerulopathy. Degenerative lesions were severe in peripheral nerves, dorsal and ventral spinal nerve roots, and less marked in dorsal fasciculi of the spinal cord. Cell bodies of spinal ganglia were minimally diseased; ventral horn neurons occasionally had central chromatolysis and nuclear displacement. Glomerular lesions ranged from mild mesangial hypercellularity to glomerulosclerosis. Pedigree analysis of affected animals from one herd indicated a strong familial relationship and probable hereditary basis for the syndrome.

Effects of tri-ortho-cresyl-phosphate on spinal ganglia and peripheral nerves of chicken

1971 ◽  
Vol 17 (2) ◽  
pp. 103-113 ◽  
Author(s):  
S. Le Vay ◽  
C. Meier ◽  
P. Glees
Keyword(s):  
Peripheral Nerves ◽  
Spinal Ganglia

Evidence for a Blood Ganglion Barrier in the Superior Cervical Ganglion of the Rat

1982 ◽  
Vol 40 ◽  
pp. 204-204
Author(s):  
D. M. DePace
Keyword(s):  
Blood Vessels ◽  
Superior Cervical Ganglion ◽  
Peripheral Nerves ◽  
Time Schedule ◽  
Transmission Electron ◽  
Cervical Ganglion ◽  
The Central Nervous System ◽  
Cervical Ganglia ◽  
Capillary Circulation ◽  
And Control

The majority of blood vessels in the superior cervical ganglion possess a continuous endothelium with tight junctions. These same features have been associated with the blood brain barrier of the central nervous system and peripheral nerves. These vessels may perform a barrier function between the capillary circulation and the superior cervical ganglion. The permeability of the blood vessels in the superior cervical ganglion of the rat was tested by intravenous injection of horseradish peroxidase (HRP). Three experimental groups of four animals each were given intravenous HRP (Sigma Type II) in a dosage of.08 to.15 mg/gm body weight in.5 ml of.85% saline. The animals were sacrificed at five, ten or 15 minutes following administration of the tracer. Superior cervical ganglia were quickly removed and fixed by immersion in 2.5% glutaraldehyde in Sorenson's.1M phosphate buffer, pH 7.4. Three control animals received,5ml of saline without HRP. These were sacrificed on the same time schedule. Tissues from experimental and control animals were reacted for peroxidase activity and then processed for routine transmission electron microscopy.

Intramuscular Nerve and Neuromuscular Junction Alteration In Extraocular Muscles of Diabetic Mice

1985 ◽  
Vol 43 ◽  
pp. 682-683
Author(s):  
Bruce R. Pachter
Keyword(s):  
Diabetes Mellitus ◽  
Peripheral Nerves ◽  
Sudden Onset ◽  
Extraocular Muscles ◽  
Diabetic Patients ◽  
Oculomotor Palsy ◽  
Third Nerve Palsy ◽  
Patients With Diabetes ◽  
Intramuscular Nerve ◽  
Oculomotor Nerves

Diabetes mellitus is one of the commonest causes of neuropathy. Diabetic neuropathy is a heterogeneous group of neuropathic disorders to which patients with diabetes mellitus are susceptible; more than one kind of neuropathy can frequently occur in the same individual. Abnormalities are also known to occur in nearly every anatomic subdivision of the eye in diabetic patients. Oculomotor palsy appears to be common in diabetes mellitus for their occurrence in isolation to suggest diabetes. Nerves to the external ocular muscles are most commonly affected, particularly the oculomotor or third cranial nerve. The third nerve palsy of diabetes is characteristic, being of sudden onset, accompanied by orbital and retro-orbital pain, often associated with complete involvement of the external ocular muscles innervated by the nerve. While the human and experimental animal literature is replete with studies on the peripheral nerves in diabetes mellitus, there is but a paucity of reported studies dealing with the oculomotor nerves and their associated extraocular muscles (EOMs).

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