Resistance of oral cancer cells to 5‐ALA‐mediated photodynamic therapy

2018 ◽  
Vol 119 (4) ◽  
pp. 3554-3562 ◽  
Author(s):  
Flávia Cristina P. Rosin ◽  
Marina Gabriela Teixeira ◽  
Cibele Pelissari ◽  
Luciana Corrêa
Keyword(s):  
Photodynamic Therapy ◽  
Oral Cancer ◽  
Cancer Cells ◽  
Oral Cancer Cells

scholarly journals miR-145 mediates the anti-cancer stemness effect of photodynamic therapy with 5-aminolevulinic acid (ALA) in oral cancer cells

2018 ◽  
Vol 117 (8) ◽  
pp. 738-742 ◽  
Author(s):  
Chih-Yuan Fang ◽  
Pei-Yin Chen ◽  
Dennis Chun-Yu Ho ◽  
Lo-Lin Tsai ◽  
Pei-Ling Hsieh ◽  
...  
Keyword(s):  
Photodynamic Therapy ◽  
Oral Cancer ◽  
Cancer Cells ◽  
Aminolevulinic Acid ◽  
Cancer Stemness ◽  
Anti Cancer ◽  
Oral Cancer Cells

Photodynamic Therapy Mediated by Liposomal Chloroaluminum-Phthalocyanine Induces Necrosis in Oral Cancer Cells

2013 ◽  
Vol 3 (1) ◽  
pp. 148-156 ◽  
Author(s):  
João Paulo Figueiró Longo ◽  
Larissa Naves Duarte de Melo ◽  
Maitê Cevallos Mijan ◽  
Caroline Rodrigues Alves Valois ◽  
Graziella Anselmo Joanitti ◽  
...  
Keyword(s):  
Photodynamic Therapy ◽  
Oral Cancer ◽  
Cancer Cells ◽  
Chloroaluminum Phthalocyanine ◽  
Oral Cancer Cells

scholarly journals A Study of photodynamic therapy against human oral cancer cells using a new photosensitizer (pheophorbide-a).

1992 ◽  
Vol 38 (5) ◽  
pp. 729-736
Author(s):  
Shigeaki TORATANI ◽  
Tetsuji OKAMOTO ◽  
Tsunei SHINKI ◽  
Kazuaki TAKADA
Keyword(s):  
Photodynamic Therapy ◽  
Oral Cancer ◽  
Cancer Cells ◽  
Pheophorbide A ◽  
Oral Cancer Cells

Design, synthesis and antimetastatic evaluation of 1-benzothiazolylphenylbenzotriazoles for photodynamic therapy in oral cancer cells

MedChemComm ◽  
2016 ◽  
Vol 7 (6) ◽  
pp. 1151-1158 ◽  
Author(s):  
Gopal Chandru Senadi ◽  
Chieh-Ming Liao ◽  
Kung-Kai Kuo ◽  
Jian-Cheng Lin ◽  
Long-Sen Chang ◽  
...  
Keyword(s):  
Photodynamic Therapy ◽  
Oral Cancer ◽  
Cancer Cells ◽  
Cancer Cell ◽  
Design Synthesis ◽  
Oral Cancer Cell ◽  
Oral Cancer Cells

We have designed and synthesized a new series of 1-benzothiazolylphenylbenzotriazoles 9a–p and studied their antimetastatic mechanism involved in photosensitive effects induced by UVA in oral cancer cell Ca9-22.

In Vitro Anticancer Activity of Virgin Coconut Oil and its Fractions in Liver and Oral Cancer Cells

2020 ◽  
Vol 19 (18) ◽  
pp. 2223-2230 ◽  
Author(s):  
Poonam Verma ◽  
Sanjukta Naik ◽  
Pranati Nanda ◽  
Silvi Banerjee ◽  
Satyanarayan Naik ◽  
...  
Keyword(s):  
Fatty Acids ◽  
Fatty Acid ◽  
Oral Cancer ◽  
Anticancer Activity ◽  
Cancer Cells ◽  
Cell Line ◽  
Coconut Oil ◽  
Virgin Coconut Oil ◽  
Anticancer Efficacy ◽  
Oral Cancer Cells

Background: Coconut oil is an edible oil obtained from fresh, mature coconut kernels. Few studies have reported the anticancer role of coconut oil. The fatty acid component of coconut oil directly targets the liver by portal circulation and as chylomicron via lymph. However, the anti-cancer activity of coconut oil against liver cancer cells and oral cancer cells is yet to be tested. The active component of coconut oil, that is responsible for the anticancer activity is not well understood. In this study, three different coconut oils, Virgin Coconut Oil (VCO), Processed Coconut Oil (PCO) and Fractionated Coconut Oil (FCO), were used. Objective: Based on previous studies, it can be hypothesized that fatty acids in coconut oil may have anticancer potential and may trigger cell death in cancer cell lines. Methods: Each cell line was treated with different concentrations of Virgin Coconut Oil (VCO), Processed Coconut Oil (PCO) and Fractionated Coconut Oil (FCO). The treated cells were assayed by MTT after 72 hr of incubation. The fatty acid composition of different coconut oils was analyzed by gas chromatography. Result: Different concentrations of coconut oils were used to treat the cells. Interestingly, the anticancer efficacy of VCO, PCO and FCO was not uniform, rather the efficacy varied from cell line to cell line. Only 20% VCO showed significant anticancer activity in HepG2 cells in comparison to 80% PCO against the KB cell line. Remarkably, 20% of PCO and 5% of FCO showed potential growth inhibition in the KB cell line as compared to 80% PCO in HepG2 cells. Moreover, there was a difference in the efficacy of VCO, PCO and FCO, which might be due to their fatty acid composition. Comparing the anticancer efficacy of VCO, PCO and FCO in this study helped to predict which class of fatty acids and which fatty acid might be associated with the anticancer activity of VCO. Conclusion: This study shows that VCO, PCO and FCO have anticancer efficacy and may be used for the treatment of cancer, especially liver and oral cancer.

Econazole-induced Ca2+ fluxes and apoptosis in human oral cancer cells

2010 ◽  
Vol 71 (4) ◽  
pp. 240-248 ◽  
Author(s):  
Daih-Huang Kuo ◽  
Li-Min Liu ◽  
Hsin-Wei Chen ◽  
Fu-An Chen ◽  
Chung-Ren Jan
Keyword(s):  
Oral Cancer ◽  
Cancer Cells ◽  
Oral Cancer Cells
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