Retraction: “Over-expression of FoxM1 leads to epithelial-mesenchymal transition and cancer stem cell phenotype in pancreatic cancer cells” by Bao et al.

doi:10.1002/jcb.25588

Retraction: “Over-expression of FoxM1 leads to epithelial-mesenchymal transition and cancer stem cell phenotype in pancreatic cancer cells” by Bao et al.

Journal of Cellular Biochemistry ◽

10.1002/jcb.25588 ◽

2016 ◽

Vol 117 (8) ◽

pp. 1963-1963 ◽

Cited By ~ 1

Keyword(s):

Pancreatic Cancer ◽

Stem Cell ◽

Cancer Stem Cell ◽

Epithelial Mesenchymal Transition ◽

Cell Phenotype ◽

Mesenchymal Transition ◽

Over Expression ◽

Pancreatic Cancer Cells ◽

Cancer Stem Cell Phenotype ◽

Stem Cell Phenotype

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Over-expression of FoxM1 leads to epithelial-mesenchymal transition and cancer stem cell phenotype in pancreatic cancer cells

Journal of Cellular Biochemistry ◽

10.1002/jcb.23150 ◽

2011 ◽

Vol 112 (9) ◽

pp. 2296-2306 ◽

Cited By ~ 143

Author(s):

Bin Bao ◽

Zhiwei Wang ◽

Shadan Ali ◽

Dejuan Kong ◽

Sanjeev Banerjee ◽

...

Keyword(s):

Pancreatic Cancer ◽

Stem Cell ◽

Cancer Stem Cell ◽

Epithelial Mesenchymal Transition ◽

Cell Phenotype ◽

Mesenchymal Transition ◽

Over Expression ◽

Pancreatic Cancer Cells ◽

Cancer Stem Cell Phenotype ◽

Stem Cell Phenotype

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RETRACTED: Notch-1 induces epithelial–mesenchymal transition consistent with cancer stem cell phenotype in pancreatic cancer cells

Cancer Letters ◽

10.1016/j.canlet.2011.03.012 ◽

2011 ◽

Vol 307 (1) ◽

pp. 26-36 ◽

Cited By ~ 204

Author(s):

Bin Bao ◽

Zhiwei Wang ◽

Shadan Ali ◽

Dejuan Kong ◽

Yiwei Li ◽

...

Keyword(s):

Pancreatic Cancer ◽

Stem Cell ◽

Cancer Stem Cell ◽

Epithelial Mesenchymal Transition ◽

Cell Phenotype ◽

Notch 1 ◽

Mesenchymal Transition ◽

Pancreatic Cancer Cells ◽

Cancer Stem Cell Phenotype ◽

Stem Cell Phenotype

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Retraction notice to “Notch-1 induces Epithelial-mesenchymal transition consistent with cancer stem cell phenotype in pancreatic cancer cells”

Cancer Letters ◽

10.1016/j.canlet.2018.03.020 ◽

2018 ◽

Vol 423 ◽

pp. 153

Author(s):

Bin Bao ◽

Zhiwei Wang ◽

Shadan Ali ◽

Dejuan Kong ◽

Yiwei Li ◽

...

Keyword(s):

Pancreatic Cancer ◽

Stem Cell ◽

Epithelial Mesenchymal Transition ◽

Cell Phenotype ◽

Notch 1 ◽

Mesenchymal Transition ◽

Pancreatic Cancer Cells ◽

Cancer Stem Cell Phenotype ◽

Retraction Notice ◽

Stem Cell Phenotype

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MicroRNA-18a induces epithelial-mesenchymal transition like cancer stem cell phenotype via regulating RKIP pathway in pancreatic cancer

Annals of Translational Medicine ◽

10.21037/atm.2020.03.195 ◽

2020 ◽

Vol 8 (7) ◽

pp. 433-433

Author(s):

Honggang Kang ◽

Dan Ma ◽

Jing Zhang ◽

Jun Zhao ◽

Mengxiang Yang

Keyword(s):

Pancreatic Cancer ◽

Stem Cell ◽

Cancer Stem Cell ◽

Epithelial Mesenchymal Transition ◽

Cell Phenotype ◽

Mesenchymal Transition ◽

Cancer Stem Cell Phenotype ◽

Stem Cell Phenotype

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microRNA‐95 knockdown inhibits epithelial–mesenchymal transition and cancer stem cell phenotype in gastric cancer cells through MAPK pathway by upregulating DUSP5

Journal of Cellular Physiology ◽

10.1002/jcp.29010 ◽

2019 ◽

Vol 235 (2) ◽

pp. 944-956 ◽

Cited By ~ 7

Author(s):

Mei Du ◽

Yuan Zhuang ◽

Peng Tan ◽

Zongbu Yu ◽

Xiutian Zhang ◽

...

Keyword(s):

Gastric Cancer ◽

Stem Cell ◽

Cancer Stem Cell ◽

Epithelial Mesenchymal Transition ◽

Mapk Pathway ◽

Cell Phenotype ◽

Gastric Cancer Cells ◽

Mesenchymal Transition ◽

Cancer Stem Cell Phenotype ◽

Stem Cell Phenotype

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MicroRNA-9 is associated with epithelial-mesenchymal transition, breast cancer stem cell phenotype, and tumor progression in breast cancer

Breast Cancer Research and Treatment ◽

10.1007/s10549-014-3069-5 ◽

2014 ◽

Vol 147 (1) ◽

pp. 39-49 ◽

Cited By ~ 82

Author(s):

Jae Moon Gwak ◽

Hyun Jeong Kim ◽

Eun Joo Kim ◽

Yul Ri Chung ◽

Sumi Yun ◽

...

Keyword(s):

Breast Cancer ◽

Stem Cell ◽

Cancer Stem Cell ◽

Tumor Progression ◽

Epithelial Mesenchymal Transition ◽

Breast Cancer Stem Cell ◽

Cell Phenotype ◽

Mesenchymal Transition ◽

Cancer Stem Cell Phenotype ◽

Stem Cell Phenotype

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miR-200c at the nexus of epithelial-mesenchymal transition, resistance to apoptosis, and the breast cancer stem cell phenotype

Breast Cancer Research ◽

10.1186/bcr2885 ◽

2011 ◽

Vol 13 (3) ◽

Cited By ~ 35

Author(s):

Derek C Radisky

Keyword(s):

Breast Cancer ◽

Stem Cell ◽

Cancer Stem Cell ◽

Epithelial Mesenchymal Transition ◽

Breast Cancer Stem Cell ◽

Cell Phenotype ◽

Mesenchymal Transition ◽

Cancer Stem Cell Phenotype ◽

Transition Resistance ◽

Stem Cell Phenotype

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First Evidences of Epithelial-Mesenchymal Transition and Cancer Stem-Cell Phenotype Acquisition in Dermo-Epidermal Junction of BPV-Infected Neoplasms

Journal of Biotechnology and Biomedical Science ◽

10.14302/issn.2576-6694.jbbs-17-1869 ◽

2017 ◽

Vol 1 (2) ◽

pp. 10-30

Author(s):

Rodrigo Pinheiro Araldi ◽

Rafael Guilen de Oliveira ◽

Gabriela Mattoso ◽

Gabriela Casarin ◽

Diego Grando MÃ³dolo ◽

...

Keyword(s):

Stem Cell ◽

Cancer Stem Cell ◽

Epithelial Mesenchymal Transition ◽

Cell Phenotype ◽

Mesenchymal Transition ◽

Cancer Stem Cell Phenotype ◽

Stem Cell Phenotype

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Glycolysis promoting cancer stem cell phenotype and inducing epithelial-mesenchymal transition

Urological Science ◽

10.1016/j.urols.2015.06.272 ◽

2015 ◽

Vol 26 (2) ◽

pp. S20-S21

Author(s):

G.-S. Shieh ◽

W.-H. Yang ◽

C.-L. Wu ◽

A.-L. Shiau

Keyword(s):

Stem Cell ◽

Cancer Stem Cell ◽

Epithelial Mesenchymal Transition ◽

Cell Phenotype ◽

Mesenchymal Transition ◽

Cancer Stem Cell Phenotype ◽

Stem Cell Phenotype

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MicroRNA-34a regulates epithelial-mesenchymal transition and cancer stem cell phenotype of head and neck squamous cell carcinoma in vitro

International Journal of Oncology ◽

10.3892/ijo.2015.3142 ◽

2015 ◽

Vol 47 (4) ◽

pp. 1339-1350 ◽

Cited By ~ 20

Author(s):

ZHIFENG SUN ◽

WEIMING HU ◽

JINFENG XU ◽

ANDREAS M. KAUFMANN ◽

ANDREAS E. ALBERS

Keyword(s):

Squamous Cell Carcinoma ◽

Stem Cell ◽

Cancer Stem Cell ◽

Cell Carcinoma ◽

Epithelial Mesenchymal Transition ◽

Cell Phenotype ◽

Mesenchymal Transition ◽

Cancer Stem Cell Phenotype ◽

Stem Cell Phenotype

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