The Increase in Maternal Expression ofaxin1andaxin2Contribute to the Zebrafish MutantIchabodVentralized Phenotype

2015 ◽  
Vol 116 (3) ◽  
pp. 418-430 ◽  
Author(s):  
Fabio Valenti ◽  
Jessica Ibetti ◽  
Yuko Komiya ◽  
Melissa Baxter ◽  
Anna Maria Lucchese ◽  
...  
Keyword(s):  
Maternal Expression

scholarly journals Maternal expression of the histone demethylase Kdm4a is crucial for pre-implantation development

Development ◽  
2017 ◽  
Vol 144 (18) ◽  
pp. 3264-3277 ◽  
Author(s):  
Aditya Sankar ◽  
Susanne Marije Kooistra ◽  
Javier Martin Gonzalez ◽  
Claes Ohlsson ◽  
Matti Poutanen ◽  
...  
Keyword(s):  
Histone Demethylase ◽  
Maternal Expression

scholarly journals Monoallelic maternal expression of STAT5A affects embryonic survival in cattle

BMC Genetics ◽  
2009 ◽  
Vol 10 (1) ◽  
Author(s):  
Hasan Khatib ◽  
Christian Maltecca ◽  
Ricky L Monson ◽  
Valerie Schutzkus ◽  
Jack J Rutledge
Keyword(s):  
Embryonic Survival ◽  
Maternal Expression

Maternal expression increases the rate of bicoid evolution by relaxing selective constraint

Genetica ◽  
2006 ◽  
Vol 129 (1) ◽  
pp. 37-43 ◽  
Author(s):  
Jeffery P. Demuth ◽  
Michael J. Wade
Keyword(s):  
Selective Constraint ◽  
Maternal Expression

scholarly journals An alternatively spliced gene encoding a Y-box protein showing maternal expression and tissue-specific zygotic expression in the ascidian embryo

1998 ◽  
Vol 40 (6) ◽  
pp. 631-640
Author(s):  
Michiko R. Wada ◽  
Yoshiaki Ohtani ◽  
Yumiko Shibata ◽  
Kimio J. Tanaka ◽  
Naomi Tanimoto ◽  
...  
Keyword(s):  
Gene Encoding ◽  
Tissue Specific ◽  
Maternal Expression ◽  
Alternatively Spliced ◽  
Ascidian Embryo

scholarly journals Reduced maternal expression of adrenomedullin disrupts fertility, placentation, and fetal growth in mice

2006 ◽  
Vol 116 (10) ◽  
pp. 2653-2662 ◽  
Author(s):  
Manyu Li ◽  
Della Yee ◽  
Terry R. Magnuson ◽  
Oliver Smithies ◽  
Kathleen M. Caron
Keyword(s):  
Fetal Growth ◽  
Maternal Expression

Genomic imprinting, development and disease—is pre-eclampsia caused by a maternally imprinted gene?

1998 ◽  
Vol 10 (1) ◽  
pp. 23 ◽  
Author(s):  
Jennifer A. Marshall Graves
Keyword(s):  
Genomic Imprinting ◽  
Fetal Development ◽  
Mutant Gene ◽  
The Other ◽  
Paternal Allele ◽  
Imprinted Genes ◽  
Maternal Expression ◽  
Mutant Genes ◽  
Normal Placenta ◽  
Mode Of Inheritance

Several genes in conserved clusters are expressed from only the maternal or the paternal allele. The other allele has been genetically silenced (‘imprinted’) by its passage through one sex. Many known imprinted genes have effects on embryonic or trophoblast growth or fetal development, and mutation or loss of the single active copy causes diseases such as Prader–Willi, Angelmann and Beckwith–Wiederman syndromes. Imprinted genes show an unusual mode of inheritance, since mutant genes have an effect on the phenotype only if they come from the parent from which they are expressed. This may explain some conditions which appear to be heritable but show an inconsistant pattern in affected families. Of particular interest is pre-eclampsia/eclampsia, the most serious complication of pregnancy, which has some features suggesting that it results from fetal expression of the mutant gene, but others which imply it results from maternal expression. This could be resolved by proposing that the condition is due to mutation in a paternally imprinted, maternally active gene which must be expressed by the fetus in order to establish a normal placenta in the first pregnancy.

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