Ceftriaxone pretreatment confers neuroprotection in rats with acute glaucoma and reduces the score of seizures induced by pentylenotetrazole in mice

2020 ◽  
Vol 34 (11) ◽  
Author(s):  
Helene A. Fachim ◽  
Renato Guizzo ◽  
Alexandra O. S. Cunha ◽  
Adriana C. Pereira ◽  
Lilian C. Anjos ◽  
...  
Keyword(s):  
Acute Glaucoma

Acute Glaucoma Following Cyanoacrylate Glue Patch for Corneal Perforation

2018 ◽  
Vol 27 (9) ◽  
pp. e148-e150 ◽  
Author(s):  
Alfonso Vasquez-Perez ◽  
Francesco Matarazzo ◽  
Niraj Mandal ◽  
Stephen Tuft
Keyword(s):  
Corneal Perforation ◽  
Cyanoacrylate Glue ◽  
Acute Glaucoma

scholarly journals Vascular and vegetative factors of glaucoma attack

2021 ◽  
Vol 14 (4) ◽  
pp. 22-30
Author(s):  
N. A. Bakunina ◽  
L. N. Kolesnikova ◽  
G. V. Poryadin ◽  
J. M. Salmasi ◽  
L. M. Balashova
Keyword(s):  
Heart Rate ◽  
Degradation Products ◽  
Lamina Cribrosa ◽  
Acute Attack ◽  
Ganglion Cell Complex ◽  
Ophthalmological Examination ◽  
Vascular Plexus ◽  
Acute Glaucoma ◽  
Sympathetic Regulation ◽  
Treatment Plans

Purpose. To study the involvement of vascular and vegetative factors in the pathogenesis of glaucoma attack. Material and methods. 12 patients (24 eyes) aged 49 to 82 — 5 men and 7 women, including 3 patients with acute glaucoma and 9 patients with subacute glaucoma were subjected to an ophthalmological examination that included visometry, tonometry, automated static perimetry, OCT and OCT angiography. They were also tested for heart rate variability (HRV) using a Polar heart rate monitor, and for plasminogen content and products of fibrin/fibrinogen degradation in the tear. For comparison, the contralateral eyes of these patients were examined. Results. In the eyes with an acute glaucoma attack, the vascular network was noticeably weakened, especially in the area of the deep peripapillary vascular plexus at the lamina cribrosa level, and focal capillary loss was observed. The peripapillary density of the deep vascular plexus in the eyes with an acute attack was 33.0 ± 5.6 % (М ± m), which was significantly (p < 0.01) lower as compared to 50.0 ± 4.7 % in the unaffected eyes. This indicator correlated with the thickness of the ganglion cell complex (GCC) (p < 0.01). In unaffected eyes, no correlations were found between these glaucoma-related parameters. A significant amount of fibrin/fibrinogen degradation products was found in the tear of glaucoma patients, which may point to a violation of blood circulation in the optic nerve vessels. It has been established that glaucoma attack occurs with increased activity of sympathetic regulation of blood flow. Conclusion. When monitoring this contingent of patients, it is essential to determine the sympathetic-parasympathetic status of the patient. Taking into account the vascular component of the condition, it is expedient to introduce the necessary additions into its treatment plans

scholarly journals Endoplasmic reticulum stress-regulated CXCR3 pathway mediates inflammation and neuronal injury in acute glaucoma

2015 ◽  
Vol 6 (10) ◽  
pp. e1900-e1900 ◽  
Author(s):  
Y Ha ◽  
H Liu ◽  
Z Xu ◽  
H Yokota ◽  
S P Narayanan ◽  
...  
Keyword(s):  
Endoplasmic Reticulum ◽  
Endoplasmic Reticulum Stress ◽  
Neuronal Injury ◽  
Acute Glaucoma

scholarly journals Topiramate-associated acute glaucoma in a migraine patient receiving concomitant citalopram therapy: a case-report

Cases Journal ◽  
2009 ◽  
Vol 2 (1) ◽  
Author(s):  
Luca Spaccapelo ◽  
Silvia Leschiutta ◽  
Claudio Aurea ◽  
Anna Ferrari
Keyword(s):  
Case Report ◽  
Migraine Patient ◽  
Acute Glaucoma

scholarly journals ACUTE GLAUCOMA: AN HISTORICAL NOTE

1932 ◽  
Vol 16 (9) ◽  
pp. 555-559 ◽  
Author(s):  
A. Sorsby
Keyword(s):  
Historical Note ◽  
Acute Glaucoma
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