Induction of apoptosis in BCL‐2‐expressing rat prostate cancer cells using the Semliki Forest virus vector

2001 ◽  
Vol 94 (4) ◽  
pp. 572-578 ◽  
Author(s):  
Ann‐Marie Murphy ◽  
Brian J. Sheahan ◽  
Gregory J. Atkins
Keyword(s):  
Prostate Cancer ◽  
Cancer Cells ◽  
Semliki Forest Virus ◽  
Virus Vector ◽  
Prostate Cancer Cells ◽  
Induction Of Apoptosis ◽  
Rat Prostate

Chronic hypoxia promotes an aggressive phenotype in rat prostate cancer cells

2007 ◽  
Vol 41 (7) ◽  
pp. 788-797 ◽  
Author(s):  
Omar Alqawi ◽  
Hong P. Wang ◽  
Myrna Espiritu ◽  
Gurmit Singh
Keyword(s):  
Prostate Cancer ◽  
Cancer Cells ◽  
Chronic Hypoxia ◽  
Prostate Cancer Cells ◽  
Aggressive Phenotype ◽  
Rat Prostate

A Study of Membrane Activity in Rat Prostate Cancer Cells: An Evaluation of the FM1–43 Dye Technique

2000 ◽  
Vol 20 (1) ◽  
pp. 31-40 ◽  
Author(s):  
Maria E. Mycielska ◽  
Monika Schäfer ◽  
Scott P. Fraser ◽  
Mustafa B. A. Djamgoz ◽  
C. Lindsay Bashford
Keyword(s):  
Prostate Cancer ◽  
Cell Division ◽  
Cancer Cells ◽  
Cell Lines ◽  
Prostate Cancer Cells ◽  
Excitable Cells ◽  
Membrane Activity ◽  
Variable Pattern ◽  
Cells In Culture ◽  
Rat Prostate

A study was initiated to test whether the FM1–43 dye technique could beapplied to the study of endocytic membrane activity in two rodent prostatecancer (MAT-LyLu and AT-2) cell lines of markedly different metastaticability. The lipophilic dye FM1–43, which has frequently been used tomonitor endo/exocytic activity in excitable cells was employed. We found,as in excitable tissues, that both strongly metastatic (MAT-LyLu) andweakly metastatic (AT-2) cells in culture take up FM1–43 to give vesicularstaining of a variable pattern, which appeared to differ between the twocell lines. However, unlike excitable tissues, neither cell linesubsequently released the dye. Indeed, both cell lines retained the dyethrough several rounds of cell division suggesting that dye incorporatedby cells does not enter the endo/exocytotic cycle. Uptake of dye wasindependent of temperature, Na+/K+ gradients, pH or metabolism. Wesuggest that passive accumulation of FM1–43 can occur in cancer cells andshould not, automatically, be interpreted as evidence of endocytosis.

Induction of apoptosis and cell cycle arrest by ethyl acetate fraction of Phoenix dactylifera L. (Ajwa dates) in prostate cancer cells

2018 ◽  
Vol 218 ◽  
pp. 35-44 ◽  
Author(s):  
Muqtadir Baig Mirza ◽  
Ayman I. Elkady ◽  
Atef M. Al-Attar ◽  
Fareeduddin Quadri Syed ◽  
Furkhan Ahmed Mohammed ◽  
...  
Keyword(s):  
Prostate Cancer ◽  
Cell Cycle ◽  
Ethyl Acetate ◽  
Cancer Cells ◽  
Ethyl Acetate Fraction ◽  
Phoenix Dactylifera ◽  
Prostate Cancer Cells ◽  
Cycle Arrest ◽  
Phoenix Dactylifera L ◽  
Induction Of Apoptosis

Transferrin receptor-dependent cytotoxicity of artemisinin–transferrin conjugates on prostate cancer cells and induction of apoptosis

2009 ◽  
Vol 274 (2) ◽  
pp. 290-298 ◽  
Author(s):  
Ikuhiko Nakase ◽  
Byron Gallis ◽  
Tomoka Takatani-Nakase ◽  
Steve Oh ◽  
Eric Lacoste ◽  
...  
Keyword(s):  
Prostate Cancer ◽  
Cancer Cells ◽  
Transferrin Receptor ◽  
Prostate Cancer Cells ◽  
Induction Of Apoptosis
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