scholarly journals The ventilatory abnormalities and prognostic values of H 2 FPEF score in dyspnoeic patients with preserved left ventricle systolic function

2020 ◽  
Vol 7 (4) ◽  
pp. 1872-1879
Author(s):  
Wei‐Ming Huang ◽  
Hao‐Min Cheng ◽  
Wen‐Chung Yu ◽  
Chao‐Yu Guo ◽  
Chern‐En Chiang ◽  
...  
2012 ◽  
Vol 158 (1) ◽  
pp. 177-179 ◽  
Author(s):  
Fernando Cabrera-Bueno ◽  
Javier Alzueta ◽  
Isabel Ruiz-Zamora ◽  
Maria J. Molina-Mora ◽  
Alberto Barrera ◽  
...  

Author(s):  
A. B. Khadzegova ◽  
E. N. Yuschuk ◽  
R. G. Gabitova ◽  
I. A. Sinitsina ◽  
S. V. Ivanova ◽  
...  

2018 ◽  
Vol 2018 ◽  
pp. 1-8
Author(s):  
Xiaowei Gao ◽  
Maoen Zhu ◽  
Yanan Cao ◽  
Yue Yang ◽  
Zhi Ye ◽  
...  

Lipopolysaccharide induces rapid deterioration of cardiac function in rats with pulmonary arterial hypertension. It was desired to investigate if this cardiac dysfunction could be treated by C-type natriuretic peptide. Rat pulmonary arterial hypertension was induced by intraperitoneal injection of monocrotaline. Hemodynamics and cardiac function were measured by pressure-volume (P-V) catheter before and after the rats were treated with lipopolysaccharide and C-type natriuretic peptide. Cyclic guanosine 3′,5′-monophosphate (cGMP) level was determined by enzyme-linked immunosorbent assay analysis. After the rats were injected with low-dose lipopolysaccharide, they experienced left ventricle systolic function deterioration. Administration of C-type natriuretic peptide improved hemodynamics and left ventricle systolic function. cGMP level was elevated after C-type natriuretic peptide treatment. C-type natriuretic peptide could ameliorate lipopolysaccharide-induced cardiac dysfunction and restore hemodynamic deterioration in rats with pulmonary arterial hypertension.


2001 ◽  
Vol 8 (1) ◽  
pp. S30-S30
Author(s):  
O SOLOVYOV ◽  
D EFREMOV ◽  
E ONUCHINA ◽  
O MOCHALOVA ◽  
M CHERSTVOVA ◽  
...  

2011 ◽  
Vol 34 (6) ◽  
pp. 330 ◽  
Author(s):  
Huseyin U Yazici ◽  
Fatih Poyraz ◽  
Nihat Sen ◽  
Yusuf Tavil ◽  
Murat Turfan ◽  
...  

Purpose: Mean platelet volume (MPV) is an indicator of platelet activation, which is a central process in the pathophysiology of coronary heart disease. Metabolic syndrome (MS) may lead to worsened left ventricular systolic function by causing recurrent thrombotic events and by aggravating systemic inflammation in the course of acute myocardial infarction. The present study was designed to investigate the relationship between MPV and left ventricular systolic function in patients with metabolic syndrome who had first ST-elevation myocardial infarction. Methods: MPV was measured on admission in 33 patients who had preserved left ventricle systolic function (mean age, 56.9±10.2 years) and in 48 patients who had depressed left ventricle systolic function (mean age, 57.9±10.5 years) with metabolic syndrome and first ST elevation myocardial infarction. Depressed left ventricle systolic function was defined as ≤50% ejection fraction value. MPV levels were compared in the two groups. Results: MPV was significantly higher in patients with depressed left ventricle systolic function in comparison with patients showing preserved left ventricle systolic function (p=0.02). Logistic regression analysis showed an independent relationship between MPV and deteriorated left ventricular systolic function, even after adjustment for potential confounders (1.08 (1.04-1.20), CI: 95%, p=0.02). Conclusions: Increased MPV on admission can be associated with degree of left ventricle systolic depression in patients with metabolic syndrome with first ST-elevation myocardial infarction. MPV may prove to be useful as a prognostic marker in patients with metabolic syndrome and ST elevation MI.


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