Effect of a very low calorie diet on the diagnostic category of individuals with binge eating disorder

2001 ◽  
Vol 31 (1) ◽  
pp. 49-56 ◽  
Author(s):  
Nancy C. Raymond ◽  
Martina de Zwaan ◽  
James E. Mitchell ◽  
Diann Ackard ◽  
Paul Thuras
Keyword(s):  
Eating Disorder ◽  
Binge Eating ◽  
Binge Eating Disorder ◽  
Diagnostic Category ◽  
Very Low Calorie Diet ◽  
Low Calorie ◽  
Low Calorie Diet ◽  
Calorie Diet

Binge Eating Disorder Affects Outcome of Comprehensive Very-Low-Calorie Diet Treatment

1994 ◽  
Vol 2 (3) ◽  
pp. 205-212 ◽  
Author(s):  
Susan Zelitch Yanovski ◽  
James F. Gormally ◽  
Maureen S. Leser ◽  
Hurry E. Gwirtsman ◽  
Jack A. Yanovski
Keyword(s):  
Eating Disorder ◽  
Binge Eating ◽  
Binge Eating Disorder ◽  
Very Low Calorie Diet ◽  
Low Calorie ◽  
Low Calorie Diet ◽  
Diet Treatment ◽  
Calorie Diet

The effects of a very low calorie diet on binge eating

1993 ◽  
Vol 24 (2) ◽  
pp. 177-193 ◽  
Author(s):  
Christy F. Telch ◽  
W. Stewart Agras
Keyword(s):  
Binge Eating ◽  
Very Low Calorie Diet ◽  
Low Calorie ◽  
Low Calorie Diet ◽  
Calorie Diet

The early improvement of glycaemia following RYGB can be mimicked by a Very Low Calorie Diet in obese volunteers with diabetes

2017 ◽  
Author(s):  
Preeshila Behary ◽  
George Tharakan ◽  
Werd Al-Najim ◽  
Adrian Brown ◽  
Alexander Miras ◽  
...  
Keyword(s):  
Very Low Calorie Diet ◽  
Early Improvement ◽  
Low Calorie ◽  
Low Calorie Diet ◽  
Calorie Diet

Lipocalin Prostaglandin D2 Synthase (LPGDS) Levels following Sleeve Gastrectomy and a Very-Low-Calorie Diet (VLCD)—A Pilot Study

Diabetes ◽  
2018 ◽  
Vol 67 (Supplement 1) ◽  
pp. 299-LB
Author(s):  
RAYMOND G. LAU ◽  
SUNIL KUMAR ◽  
JENNY LEE ◽  
CHRIS HALL ◽  
THOMAS PALAIA ◽  
...  
Keyword(s):  
Sleeve Gastrectomy ◽  
Pilot Study ◽  
Prostaglandin D2 ◽  
Very Low Calorie Diet ◽  
Low Calorie ◽  
Low Calorie Diet ◽  
Calorie Diet ◽  
Prostaglandin D2 Synthase

scholarly journals Individuals Diagnosed with Binge-Eating Disorder Have DNA Hypomethylated Sites in Genes of the Metabolic System: A Pilot Study

Nutrients ◽  
2021 ◽  
Vol 13 (5) ◽  
pp. 1413
Author(s):  
Mariana Lizbeth Rodríguez-López ◽  
José Jaime Martínez-Magaña ◽  
David Ruiz-Ramos ◽  
Ana Rosa García ◽  
Laura Gonzalez ◽  
...  
Keyword(s):  
Dna Methylation ◽  
Eating Disorder ◽  
Protein Kinase ◽  
Binge Eating ◽  
Binge Eating Disorder ◽  
Environmental Changes ◽  
Diagnostic Category ◽  
The Body ◽  
Metabolic System ◽  
Genome Wide

Binge-eating disorder, recently accepted as a diagnostic category, is differentiated from bulimia nervosa in that the former shows the presence of binge-eating episodes and the absence of compensatory behavior. Epigenetics is a conjunct of mechanisms (like DNA methylation) that regulate gene expression, which are dependent on environmental changes. Analysis of DNA methylation in eating disorders shows that it is reduced. The present study aimed to analyze the genome-wide DNA methylation differences between individuals diagnosed with BED and BN. A total of 46 individuals were analyzed using the Infinium Methylation EPIC array. We found 11 differentially methylated sites between BED- and BN-diagnosed individuals, with genome-wide significance. Most of the associations were found in genes related to metabolic processes (ST3GAL4, PRKAG2, and FRK), which are hypomethylated genes in BED. Cg04781532, located in the body of the PRKAG2 gene (protein kinase AMP-activated non-catalytic subunit gamma 2), was hypomethylated in individuals with BED. Agonists of PRKAG2, which is the subunit of AMPK (AMP-activated protein kinase), are proposed to treat obesity, BED, and BN. The present study contributes important insights into the effect that BED could have on PRKAG2 activation.

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