scholarly journals Childhood maltreatment and social anxiety disorder: implications for symptom severity and response to pharmacotherapy

2011 ◽  
Vol 29 (2) ◽  
pp. 132-139 ◽  
Author(s):  
Laura C. Bruce ◽  
Richard G. Heimberg ◽  
Carlos Blanco ◽  
Franklin R. Schneier ◽  
Michael R. Liebowitz
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Childhood Maltreatment ◽  
Symptom Severity

scholarly journals Childhood maltreatment linked to greater symptom severity and poorer quality of life and function in social anxiety disorder

2009 ◽  
Vol 26 (11) ◽  
pp. 1027-1032 ◽  
Author(s):  
Naomi M. Simon ◽  
Nannette N. Herlands ◽  
Elizabeth H. Marks ◽  
Catherine Mancini ◽  
Andrea Letamendi ◽  
...  
Keyword(s):  
Quality Of Life ◽  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Childhood Maltreatment ◽  
Symptom Severity ◽  
And Function

scholarly journals The Role of Environmental Factors in the Aetiology of Social Anxiety Disorder: A Review of the Theoretical and Empirical Literature

2017 ◽  
Vol 34 (2) ◽  
pp. 76-97 ◽  
Author(s):  
Alice R. Norton ◽  
Maree J. Abbott
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Environmental Factors ◽  
Attachment Style ◽  
Social Anxiety Disorder ◽  
Childhood Maltreatment ◽  
Social Performance ◽  
Empirical Literature ◽  
Peer Experiences ◽  
Current Article

Social anxiety disorder (SAD) is characterised by a marked and persistent fear of social/performance situations, and a number of key environmental factors have been implicated in the aetiology of the disorder. Hence, the current article reviews theoretical and empirical evidence linking the development of SAD with parenting factors, traumatic life events, and aversive social experiences. Specifically, research suggests that the risk of developing SAD is increased by over-controlling, critical and cold parenting, an insecure attachment style, aversive social/peer experiences, emotional maltreatment, and to a lesser extent other forms of childhood maltreatment and adversity. Moreover, these factors may lead to posttraumatic reactions, distorted negative self-imagery, and internalised shame-based schemas that subsequently maintain SAD symptomatology. However, further research is necessary to clarify the nature, interactions, and relative contributions of these factors. It is likely that SAD develops via a complex interplay of biological and environmental factors, and that multiple aetiological pathways underlie the development of the disorder.

scholarly journals Emotional and interpersonal mechanisms in community SSRI treatment of social anxiety disorder

2020 ◽  
Vol 46 (1) ◽  
pp. E56-E64
Author(s):  
Lance M. Rappaport ◽  
Michael D. Hunter ◽  
Jennifer J. Russell ◽  
Gilbert Pinard ◽  
Pierre Bleau ◽  
...  
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Negative Affect ◽  
Positive Affect ◽  
Social Anxiety Disorder ◽  
Symptom Severity ◽  
Anxiety Symptom ◽  
Control Group ◽  
Subsequent Reduction ◽  
Interpersonal Behaviour

Background: Affective and interpersonal behavioural patterns characteristic of social anxiety disorder show improvement during treatment with serotonin agonists (e.g., selective serotonin reuptake inhibitors), commonly used in the treatment of social anxiety disorder. The present study sought to establish whether, during community psychopharmacological treatment of social anxiety disorder, changes in positive or negative affect and agreeable or quarrelsome behaviour mediate improvement in social anxiety symptom severity or follow from it. Methods: Adults diagnosed with social anxiety disorder (n = 48) recorded their interpersonal behaviour and affect naturalistically in an event-contingent recording procedure for 1-week periods before and during the first 4 months of treatment with paroxetine. Participants and treating psychiatrists assessed the severity of social anxiety symptoms monthly. A multivariate latent change score framework examined temporally lagged associations of change in affect and interpersonal behaviour with change in social anxiety symptom severity. Results: Elevated agreeable behaviour and positive affect predicted greater subsequent reduction in social anxiety symptom severity over the following month of treatment. Elevated negative affect, but not quarrelsome behaviour, predicted less subsequent reduction in symptom severity. Limitations: Limitations included limited assessment of extreme behaviour (e.g., violence) that may have precluded examining the efficacy of paroxetine because of the lack of a placebo control group. Conclusion: The present study suggests that interpersonal behaviour and affect may be putative mechanisms of action for serotonergic treatment of social anxiety disorder. Prosocial behaviour and positive affect increase during serotonergic treatment of social anxiety disorder. Specifically, modulating agreeable behaviour, positive affect and negative affect in individuals’ daily lives may partially explain and refine clinical intervention.

Modular Cognitive-Behavioral Therapy for a Young Adult With Autism Spectrum Disorder, Comorbid Obsessive Compulsive Disorder, and Social Anxiety Disorder

2018 ◽  
Vol 17 (3) ◽  
pp. 136-149 ◽  
Author(s):  
D. Luis Ordaz ◽  
Adam B. Lewin ◽  
Eric A. Storch
Keyword(s):  
Autism Spectrum Disorder ◽  
Case Report ◽  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Symptom Severity ◽  
Behavioral Therapy ◽  
Autism Spectrum ◽  
Obsessive Compulsive ◽  
Compulsive Disorder

This case report outlines the use of a modular cognitive-behavioral therapy (CBT) protocol used to treat obsessive compulsive disorder (OCD) and social anxiety disorder (SAD) in an 18-year-old female, “Jaina,” with autism spectrum disorder (ASD). Jaina completed 16 weekly CBT sessions that lasted approximately 60 min each. On completing the program, Jaina’s symptom severity for treatment targets of OCD and SAD had reduced based on the Anxiety and Related Disorders Interview Schedule for Diagnostic and Statistical Manual of Mental Disorders (5th ed.; DSM- 5; ADIS-5). In addition, overall anxiety symptom severity reduction was evident at posttreatment based on the Clinical Global Impression of Severity (CGI-S). This case report supports the use of a modular CBT program (developed for late adolescents and young adults with ASD and comorbid anxiety/OCD) for OCD and SAD.

Pharmacological treatments for social anxiety disorder in adults: a systematic review and network meta-analysis

2020 ◽  
Vol 32 (4) ◽  
pp. 169-176
Author(s):  
Taryn Williams ◽  
Michael McCaul ◽  
Guido Schwarzer ◽  
Andrea Cipriani ◽  
Dan J Stein ◽  
...  
Keyword(s):  
Systematic Review ◽  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Symptom Severity ◽  
Meta Analysis ◽  
Response To Treatment ◽  
Pharmacological Intervention ◽  
Quality Of Evidence

AbstractObjective:The aim of this paper was to provide a systematic review and update on the pharmacotherapy of social anxiety disorder (SAD), including the efficacy and tolerability of these agents, the ranking of interventions, and the grading of results by quality of evidence.Methods:The Common Mental Disorder Controlled Trial Register and two trial registries were searched for randomised controlled trials (RCTs) comparing any pharmacological intervention or placebo in the treatment of SAD. We performed a standard pairwise meta-analysis using a random effects model and carried out a network meta-analysis (NMA) using the statistical package, R. Quality of evidence was also assessed.Results:We included 67 RCTs in the review and 21 to 45 interventions in the NMA. Paroxetine was most effective in the reduction of symptom severity as compared to placebo. Superior response to treatment was also observed for paroxetine, brofaromine, bromazepam, clonazepam, escitalopram, fluvoxamine, phenelzine, and sertraline. Higher dropout rates were found for fluvoxamine. Brofaromine, escitalopram, fluvoxamine, paroxetine, pregabalin, sertraline, and venlafaxine performed worse in comparison to placebo for the outcome of dropouts due to adverse events. Olanzapine yielded a relatively high rank for treatment efficacy and buspirone the worse rank for dropouts due to any cause.Conclusion:The differences between drugs and placebo were small, apart from a significant reduction in symptom severity and response for paroxetine. We suggest paroxetine as a first-line treatment of SAD, with the consideration of future research on the drug olanzapine as well as brofaromine, bromazepam, clonazepam, escitalopram, fluvoxamine, phenelzine, and sertraline because we observed a response to treatment.

scholarly journals Reduced Heart Rate Variability in Social Anxiety Disorder: Associations with Gender and Symptom Severity

PLoS ONE ◽  
2013 ◽  
Vol 8 (7) ◽  
pp. e70468 ◽  
Author(s):  
Gail A. Alvares ◽  
Daniel S. Quintana ◽  
Andrew H. Kemp ◽  
Anita Van Zwieten ◽  
Bernard W. Balleine ◽  
...  
Keyword(s):  
Heart Rate ◽  
Heart Rate Variability ◽  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Symptom Severity

scholarly journals Brain activation to task-irrelevant disorder-related threat in social anxiety disorder: The impact of symptom severity

2017 ◽  
Vol 14 ◽  
pp. 323-333 ◽  
Author(s):  
Carina Yvonne Heitmann ◽  
Katharina Feldker ◽  
Paula Neumeister ◽  
Leonie Brinkmann ◽  
Elisabeth Schrammen ◽  
...  
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Symptom Severity ◽  
Brain Activation ◽  
Task Irrelevant ◽  
The Impact

Enlargement of visual processing regions in social anxiety disorder is related to symptom severity

2014 ◽  
Vol 583 ◽  
pp. 114-119 ◽  
Author(s):  
Andreas Frick ◽  
Jonas Engman ◽  
Iman Alaie ◽  
Johannes Björkstrand ◽  
Vanda Faria ◽  
...  
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Symptom Severity ◽  
Visual Processing
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