scholarly journals Models of Inflammation: Carrageenan Air Pouch

2021 ◽  
Vol 1 (8) ◽  
Author(s):  
Jill C. Fehrenbacher ◽  
Kenneth E. McCarson
Keyword(s):  
Air Pouch

scholarly journals Macrophage ferritin and iron deposition in the rat air pouch model of inflammatory synovitis.

1987 ◽  
Vol 46 (4) ◽  
pp. 334-338 ◽  
Author(s):  
C J Morris ◽  
D R Blake ◽  
S D Hewitt ◽  
J Lunec
Keyword(s):  
Iron Deposition ◽  
Air Pouch ◽  
Air Pouch Model

Ampere-hour-scale zinc–air pouch cells

Nature Energy ◽  
2021 ◽  
Author(s):  
Sambhaji S. Shinde ◽  
Jin Young Jung ◽  
Nayantara K. Wagh ◽  
Chi Ho Lee ◽  
Dong-Hyung Kim ◽  
...  
Keyword(s):  
Air Pouch

Mechanism of calcium hydroxide–induced neutrophil migration into air-pouch cavity

2008 ◽  
Vol 105 (6) ◽  
pp. 814-821 ◽  
Author(s):  
Mariana Machado Teixeira de Moraes Costa ◽  
Sandra Helena Penha de Oliveira ◽  
João Eduardo Gomes-Filho
Keyword(s):  
Calcium Hydroxide ◽  
Neutrophil Migration ◽  
Air Pouch

An Interaction Between Genetic Factors and Gender Determines the Magnitude of the Inflammatory Response in the Mouse Air Pouch Model of Acute Inflammation

Inflammation ◽  
2005 ◽  
Vol 29 (1) ◽  
pp. 1-7 ◽  
Author(s):  
David L. Delano ◽  
M. Carmen Montesinos ◽  
Peter D'Eustachio ◽  
Tim Wiltshire ◽  
Bruce N. Cronstein
Keyword(s):  
Inflammatory Response ◽  
Acute Inflammation ◽  
Genetic Factors ◽  
Air Pouch ◽  
And Gender ◽  
Air Pouch Model

scholarly journals Lipopolysaccharides of Fusobacterium nucleatum and Porphyromonas gingivalis increase RANKL-expressing neutrophils in air pouches of mice

2021 ◽  
Vol 37 (1) ◽  
Author(s):  
Ae Ri Kim ◽  
Yun Kyong Lim ◽  
Joong-Ki Kook ◽  
Eun-Jung Bak ◽  
Yun-Jung Yoo
Keyword(s):  
Flow Cytometry ◽  
Porphyromonas Gingivalis ◽  
Osteoclast Differentiation ◽  
Fusobacterium Nucleatum ◽  
Pathological Changes ◽  
Air Pouch ◽  
E Coli ◽  
Differentiation Factor ◽  
Osteoclast Differentiation Factor

AbstractIncreases of neutrophils and osteoclasts are pathological changes of periodontitis. RANKL is an osteoclast differentiation factor. The effect of periodontopathogen LPS on RANKL-expressing neutrophils has not been clarified yet. We evaluated numerical changes of RANKL-expressing neutrophils in air pouches of mice injected with LPSs of Fusobacterium nucleatum and Porphyromonas gingivalis. Mice with air pouches were assigned into saline (C)-, E. coli LPS- (Ec LPS)-, F. nucleatum LPS (Fn LPS)-, P. gingivalis LPS (Pg LPS)-, and Fn LPS and Pg LPS (Fn + Pg LPS)-injected groups. CD11b+Ly6G+ neutrophils and CD11b+Ly6G+RANKL+ neutrophils in blood and air pouch exudates were determined by flow cytometry. In blood, compared to the C group, the Fn LPS group showed increases of CD11b+Ly6G+ neutrophils and CD11b+Ly6G+RANKL+ neutrophils whereas the Pg LPS group showed no significant differences. These increases in the Fn LPS group were not different to those in the Ec LPS group. In exudates, Fn LPS and Pg LPS groups showed increases of CD11b+Ly6G+ neutrophils and CD11b+Ly6G+RANKL+ neutrophils compared to the C group. Increased levels in the Fn LPS group were not different to those in the Ec LPS group, but Pg LPS group was lower than those in the Ec LPS group. In blood and exudates, the Fn + Pg LPS group showed no difference in levels of these neutrophils compared to the Ec LPS group. LPSs of F. nucleatum and P. gingivalis increased RANKL-expressing neutrophils although the degrees of increases were different. These suggest that periodontopathogen LPS can act as a stimulant to increase RANKL-expressing neutrophils.

Use of the rat air pouch model of inflammation to evaluate regional drug delivery

1992 ◽  
Vol 36 (3-4) ◽  
pp. C109-C111 ◽  
Author(s):  
D. E. Davies ◽  
A. J. Stevens ◽  
J. B. Houston
Keyword(s):  
Drug Delivery ◽  
Air Pouch ◽  
Air Pouch Model

scholarly journals Effect of blood on the activity and persistence of antigen induced inflammation in the rat air pouch.

1985 ◽  
Vol 44 (7) ◽  
pp. 485-490 ◽  
Author(s):  
S Yoshino ◽  
D R Blake ◽  
S Hewitt ◽  
C Morris ◽  
P A Bacon
Keyword(s):  
Air Pouch

scholarly journals DISTINCT CELLULAR MIGRATION INDUCED BY Leishmania infantum chagasi AND SALIVA FROM Lutzomyia longipalpis IN A HEMORRHAGIC POOL MODEL

2014 ◽  
Vol 56 (1) ◽  
pp. 21-27 ◽  
Author(s):  
Camila Oliveira Vasconcelos ◽  
Zirlane C. Branco Coelho ◽  
Cristina de Souza Chaves ◽  
Clarissa Romero Teixeira ◽  
Margarida M. Lima Pompeu ◽  
...  
Keyword(s):  
Cell Population ◽  
Cellular Migration ◽  
Sand Fly ◽  
Leishmania Infection ◽  
Lutzomyia Longipalpis ◽  
Specific Cell ◽  
Air Pouch ◽  
Leukocyte Accumulation ◽  
Pool Model ◽  
Sand Fly Vectors

Recruitment of a specific cell population after Leishmania infection can influence the outcome of the disease. Cellular migration in response to Leishmania or vector saliva has been reported in air pouch model, however, cellular migration induced by Leishmania associated with host's blood and vector saliva in this model has not been described. Herein we investigated cellular migration into air pouch of hamster after stimulation with combination of L. chagasi and host's blood and Lutzomyia longipalpis saliva. Migration induced by saliva was 3-fold more than those induced by L. chagasi alone. Additionally, L. chagasi associated with blood and saliva induced significantly even more leukocytes into air pouch than Leishmania alone. L. chagasi recruited a diverse cell population; however, most of these cells seem to have not migrated to the inflammatory exudate, remaining in the pouch lining tissue. These results indicate that L. chagasi can reduce leukocyte accumulation to the initial site of infection, and when associated with vector saliva in the presence of blood components, increase the influx of more neutrophils than macrophages, suggesting that the parasite has developed a strategy to minimize the initial inflammatory response, allowing an unlimited progression within the host. This work reinforces the importance of studies on the salivary components of sand fly vectors of leishmaniasis in the transmission process and the establishment of the infection.

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