Pulmonary Excretion of Absorbed Gases

2011 ◽  
Author(s):  
Robert A. Klocke
Keyword(s):  
Pulmonary Excretion

Pulmonary excretion of methanol in man

1983 ◽  
Vol 43 (5) ◽  
pp. 377-379 ◽  
Author(s):  
Dag Jacobsen ◽  
Steinar Øvrebø ◽  
Erik Arnesen ◽  
Povel Paus
Keyword(s):  
Pulmonary Excretion

Pulmonary excretion of carbon monoxide in the human infant as an index of bilirubin production

1981 ◽  
Vol 137 (3) ◽  
pp. 255-259 ◽  
Author(s):  
David K. Stevenson ◽  
Clinton R. Ostrander ◽  
Ronald S. Cohen ◽  
John D. Johnson ◽  
Herbert C. Schwartz
Keyword(s):  
Carbon Monoxide ◽  
Human Infant ◽  
Pulmonary Excretion

Pulmonary Excretion Rates of Carbon Monoxide Using a Modified Technique: Differences between Premature and Full-Term Infants

Neonatology ◽  
1982 ◽  
Vol 41 (5-6) ◽  
pp. 289-293 ◽  
Author(s):  
Ronald S. Cohen ◽  
Clinton R. Ostrander ◽  
Barrett E. Cowan ◽  
Gloria B. Stevens ◽  
Andrew O. Hopper ◽  
...  
Keyword(s):  
Carbon Monoxide ◽  
Full Term ◽  
Modified Technique ◽  
Term Infants ◽  
Excretion Rates ◽  
Pulmonary Excretion

scholarly journals 614 SIMULTANEOUS DETERMINATIONS OF BREATH CONCENTRATION AND PULMONARY EXCRETION RATE OF HYDROGEN IN NEWBORNS

1981 ◽  
Vol 15 ◽  
pp. 543-543
Author(s):  
Clinton R Ostrander ◽  
Barrett E Cowan ◽  
John A Kerner ◽  
David K Stevenson ◽  
John D Johnson
Keyword(s):  
Excretion Rate ◽  
Pulmonary Excretion

scholarly journals FACTORS INFLUENCING PULMONARY METHANE EXCRETION IN MAN

1971 ◽  
Vol 133 (3) ◽  
pp. 572-588 ◽  
Author(s):  
John H. Bond ◽  
Rolf R. Engel ◽  
Michael D. Levitt
Keyword(s):  
Genetic Factors ◽  
Excretion Rate ◽  
Adult Population ◽  
Familial Factors ◽  
Ch4 Production ◽  
Factors Influencing ◽  
Colonic Bacteria ◽  
Familial Factor ◽  
Accurate Indicator ◽  
Pulmonary Excretion

Measurements of pulmonary excretion of methane (CH4) were used to obtain information on the CH4-producing bacteria in man. Preliminary studies indicated that (a) all CH4 excreted by man is produced by colonic bacteria, (b) there is no appreciable utilization of CH4 by man, and (c) breath CH4 can serve as a relatively accurate indicator of CH4 production in the intestine. The rate of pulmonary CH4 excretion varied enormously, ranging from undetectable (<5 x 10–6 ml/min) to 0.66 ml/minute. In general, the CH4 excretion rate for subjects was consistently very low (nonproducers) or relatively large (producers). 33.6% of the adult population were producers of CH4. Whereas diet, age over 10 yr, and sex did not influence the rate of CH4 production, some familial factor appeared to play an important role. 84% of siblings of CH4 producers also were producers, while only 18% of the siblings of nonproducers were found to be CH4 producers. This familial tendency appeared to be determined by early environmental rather than genetic factors. These studies of CH4 excretion demonstrate that the exposure of individuals to intestinal bacterial metabolites may differ markedly and that these differences may be chronic and determined by familial factors.

Effect of elevated PICO2 on metabolic rate in humans and ponies

1982 ◽  
Vol 52 (6) ◽  
pp. 1623-1628 ◽  
Author(s):  
R. P. Kaminski ◽  
H. V. Forster ◽  
J. P. Klein ◽  
L. G. Pan ◽  
G. E. Bisgard ◽  
...  
Keyword(s):  
Respiratory Exchange Ratio ◽  
Human Subjects ◽  
Pulmonary Ventilation ◽  
Whole Body ◽  
Nonshivering Thermogenesis ◽  
Tissue Storage ◽  
Co2 Partial Pressure ◽  
Maximum Decrease ◽  
Proportional Increase ◽  
Pulmonary Excretion

The primary purpose of this study was to determine the effect of acute (20–30 min) elevations of inspired CO2 partial pressure (PICO2) on whole-body O2 consumption (VO2). In human subjects, VO2 increased approximately 15 ml.min-1.m-2 with each 7-Torr increment in PICO2 from 0.4 to 28 Torr (P less than 0.05), but VO2 did not change significantly when PICO2 was increased from 28 to 35 and 42 Torr (P greater than 0.05). In ponies, VO2 did not change when PICO2 was increased from 0.7 to 7 Torr (P greater than 0.05), but it increased about 6 ml.min-1.m-2 with each 7-Torr increment in PICO2 from 7 to 28 Torr, and it increased 18 ml.min-1.m-2 when PICO2 was increased from 28 to 42 Torr (P less than 0.05). At low PICO2 the delta VO2/ delta VE was 25 and 7 ml/l for humans and ponies, respectively, where VE is pulmonary ventilation. These values exceeded the expected O2 cost of breathing; hence, some factor, such as shivering or nonshivering thermogenesis, contributed to the elevated VO2. At high PICO2, VE increased without a proportional increase in VO2; thus the delta VO2/ delta VE decreased to about 2.5 ml/l in ponies and to near 0.0 in humans. Accordingly, at high PICO2 some VO2-suppressing factor partially counteracted those factors stimulating VO2. The maximum decrease from control pHa was 0.061 and 0.038 in humans and ponies, respectively. It is questionable whether this mild acidosis was sufficient to suppress VO2. In both species, pulmonary excretion of metabolic CO2 and the respiratory exchange ratio were below control during CO2 inhalation (P less than 0.01), which suggested an increased tissue storage of CO2.

Rate of pulmonary excretion of paraldehyde in man

1969 ◽  
Vol 15 (2) ◽  
pp. 269-274 ◽  
Author(s):  
Daniel W. Lang ◽  
Harold H. Borgstedt
Keyword(s):  
Pulmonary Excretion

Pulmonary Excretion of Carbon Monoxide in the Human Infant as an Index of Bilirubin Production IV. Effects of Breast-feeding and Caloric Intake in the First Postnatal Week

PEDIATRICS ◽  
1980 ◽  
Vol 65 (6) ◽  
pp. 1170-1172 ◽  
Author(s):  
David K. Stevenson ◽  
Albert L. Bartoletti ◽  
Clinton R. Ostrander ◽  
John D. Johnson
Keyword(s):  
Carbon Monoxide ◽  
Caloric Intake ◽  
Human Infant ◽  
Significant Difference ◽  
Group 3 ◽  
The Mean ◽  
Group 2 ◽  
Breast Fed ◽  
Pulmonary Excretion ◽  
Group 1

Measurements of the pulmonary excretion rate of carbon monoxide (VEco) as an index of bilirubin production in the first several days of life were taken from 64 breast-fed or bottle-fed infants. Twenty-one infants (≥37 weeks of gestation) were breast-fed; 43 infants (28 to 42 weeks of gestation) were bottle-fed a commercially prepared formula. Information pertaining to their caloric intake during the 24-hour period preceding VECO determination was taken from 38 of the 43 infants who were bottle-fed and they were placed into three groups based on their caloric intake: (1) ≤60 kcal/kg/day (19 infants); (2) 61 to 100 kcal/kg/day (7 infants); and (3) > 100 kcal/kg/day (12 infants). There was no significant difference in bilirubin production between bottle-fed and breast-fed infants. No effect of caloric deprivation on bilirubin production was demonstrated. The mean VECO values were 18.5 ± 0.9 (SE) for group 1, 17.7 ± 1.8 (SE) for group 2, and 16.2 ± 1.1 (SE) µl/kg/hr for group 3.

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