The efferent connections of the ventromedial nucleus of the hypothalamus of the rat,

1976 ◽  
Vol 169 (4) ◽  
pp. 409-442 ◽  
Author(s):  
C. B. Saper ◽  
L. W. Swanson ◽  
W. M. Cowan
Keyword(s):  
Ventromedial Nucleus ◽  
Efferent Connections

1824-P: Leptin Receptor Neurons in the Hypothalamic Ventromedial Nucleus Play a Physiological Role in Regulating Glucose Tolerance

Diabetes ◽  
2019 ◽  
Vol 68 (Supplement 1) ◽  
pp. 1824-P
Author(s):  
CHELSEA L. FABER ◽  
TRISTA J. HARVEY ◽  
KEVIN R. VELASCO ◽  
VINCENT DAMIAN ◽  
MICHAEL W. SCHWARTZ ◽  
...  
Keyword(s):  
Glucose Tolerance ◽  
Leptin Receptor ◽  
Physiological Role ◽  
Ventromedial Nucleus ◽  
Receptor Neurons

Electrical activity of the ventromedial nucleus of the hypothalamus

1959 ◽  
Vol 197 (4) ◽  
pp. 829-834 ◽  
Author(s):  
Dana C. Brooks
Keyword(s):  
Electrical Activity ◽  
Slow Waves ◽  
Ventromedial Nucleus ◽  
Spontaneous Electrical Activity ◽  
Pentobarbital Anesthesia

The spontaneous electrical activity of the ventromedial nucleus was studied in the cat under pentobarbital anesthesia and in the unanesthetized, unrestrained state. Under light pentobarbital anesthesia the activity of the nucleus is characterized by a predominant 9–15 cps, 50–100 µv component which is uniform from second to second. With small additional doses of anesthesia there is a selective depression of this activity; with recovery from light anesthesia this activity is gradually replaced by irregular, large, slow waves characteristic of sleep. When the unanesthetized animal is aroused 20–35 cps activity having an amplitude of 40 µv or more appears in the nucleus. While the pattern of activity during sleep resembles that seen elsewhere in the hypothalamus, the activity seen during barbiturate anesthesia and during arousal is confined to the nucleus and not seen in other parts of the diencephalon.

Changes in the Expression of Steroidogenic Factor 1 (SF-1) in Neurons in the Ventromedial Nucleus of the Hypothalamus in Rats on Aging

2021 ◽  
Vol 51 (3) ◽  
pp. 346-349
Author(s):  
K. Yu. Moiseev ◽  
A. A. Spirichev ◽  
P. A. Vishnyakova ◽  
A. D. Nozdrachev ◽  
P. M. Masliukov
Keyword(s):  
Ventromedial Nucleus ◽  
Steroidogenic Factor 1

Efferent connections of the substantia nigra in the cat

1965 ◽  
Vol 11 (4) ◽  
pp. 474-482 ◽  
Author(s):  
Adel Afifi ◽  
William W. Kaelber
Keyword(s):  
Substantia Nigra ◽  
Efferent Connections

scholarly journals Changes in Prefrontal Cortex–Thalamic Circuitry after Acoustic Trauma

Biomedicines ◽  
2021 ◽  
Vol 9 (1) ◽  
pp. 77
Author(s):  
Kristin M. Barry ◽  
Donald Robertson ◽  
Wilhelmina H. A. M. Mulders
Keyword(s):  
Electrical Stimulation ◽  
Prefrontal Cortex ◽  
Auditory System ◽  
Acoustic Trauma ◽  
Neuron Activity ◽  
Compensatory Mechanism ◽  
Efferent Connections ◽  
Medial Geniculate ◽  
Central Auditory Pathways ◽  
Stimulation Of

In the adult auditory system, loss of input resulting from peripheral deafferentation is well known to lead to plasticity in the central nervous system, manifested as reorganization of cortical maps and altered activity throughout the central auditory pathways. The auditory system also has strong afferent and efferent connections with cortico-limbic circuitry including the prefrontal cortex and the question arises whether this circuitry is also affected by loss of peripheral input. Recent studies in our laboratory showed that PFC activation can modulate activity of the auditory thalamus or medial geniculate nucleus (MGN) in normal hearing rats. In addition, we have shown in rats that cochlear trauma resulted in altered spontaneous burst firing in MGN. However, whether the PFC influence on MGN is changed after cochlear trauma is unknown. We investigated the effects of electrical stimulation of PFC on single neuron activity in the MGN in anaesthetized Wistar rats 2 weeks after acoustic trauma or sham surgery. Electrical stimulation of PFC showed a variety of effects in MGN neurons both in sham and acoustic trauma groups but inhibitory responses were significantly larger in the acoustic trauma animals. These results suggest an alteration in functional connectivity between PFC and MGN after cochlear trauma. This change may be a compensatory mechanism increasing sensory gating after the development of altered spontaneous activity in MGN, to prevent altered activity reaching the cortex and conscious perception.

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