Afferent and efferent connections of the cerebellum of the chondrostean Acipenser baeri: A carbocyanine dye (DiI) tracing study

2003 ◽  
Vol 460 (3) ◽  
pp. 327-344 ◽  
Author(s):  
Gema Huesa ◽  
Ramón Anadón ◽  
Julián Yáñez
Keyword(s):  
Acipenser Baeri ◽  
Efferent Connections ◽  
Carbocyanine Dye ◽  
Dii Tracing

Afferent and Efferent Connections of the Pineal Organ in the European Sea Bass Dicentrarchus labrax: A Carbocyanine Dye Tract-Tracing Study

2011 ◽  
Vol 78 (4) ◽  
pp. 272-285 ◽  
Author(s):  
Arianna Servili ◽  
Patricia Herrera-Pérez ◽  
Julián Yáñez ◽  
José Antonio Muñoz-Cueto
Keyword(s):  
Pineal Organ ◽  
Dicentrarchus Labrax ◽  
Sea Bass ◽  
Tract Tracing ◽  
European Sea Bass ◽  
Efferent Connections ◽  
Carbocyanine Dye

Preplate neurons in embryonic mouse cortex: Ultrastructural observations using photoconversion of the fluorescent lipophilic dye dil

1992 ◽  
Vol 50 (1) ◽  
pp. 906-907
Author(s):  
Linda C. Hassinger ◽  
James E. Crandall
Keyword(s):  
Mouse Embryos ◽  
Embryonic Mouse ◽  
Cortical Afferents ◽  
Mouse Cortex ◽  
Carbocyanine Dye ◽  
Increased Sensitivity

We have begun to look directly at small numbers of afferent axons to early generated neurons that form the preplate in the developing mouse cortex. The carbocyanine dye Dil (1’1, dioctadecyl-3,3,3’3’-tetramethyl-indocarbocyanine) has proved especially useful for this goal. DiI labels axons and their terminals with greater sensitivity and without some of the disadvantages of axon filling with HRP. The increased sensitivity provided by labeling embryonic axons with DiI has given us new insights into the development of cortical afferents. For instance, we reported originally that afferents from the thalamus were present below the cortex as early as embryonic day 15 (E15) based on HRP injections into mouse embryos. By using DiI placements into the thalamus in aldehyde-fixed brains, we now know that thalamic fibers reach the cortex 24 hrs earlier.

Studies on the early embryonic development of artificially-bred Siberian sturgeon(Acipenser baeri)

2010 ◽  
Vol 34 (5) ◽  
pp. 777-785 ◽  
Author(s):  
Wei SONG ◽  
Jia-kun SONG ◽  
Chun-xin FAN ◽  
Tao ZHANG ◽  
Bin WANG
Keyword(s):  
Embryonic Development ◽  
Siberian Sturgeon ◽  
Early Embryonic Development ◽  
Acipenser Baeri

Efferent connections of the substantia nigra in the cat

1965 ◽  
Vol 11 (4) ◽  
pp. 474-482 ◽  
Author(s):  
Adel Afifi ◽  
William W. Kaelber
Keyword(s):  
Substantia Nigra ◽  
Efferent Connections

scholarly journals Changes in Prefrontal Cortex–Thalamic Circuitry after Acoustic Trauma

Biomedicines ◽  
2021 ◽  
Vol 9 (1) ◽  
pp. 77
Author(s):  
Kristin M. Barry ◽  
Donald Robertson ◽  
Wilhelmina H. A. M. Mulders
Keyword(s):  
Electrical Stimulation ◽  
Prefrontal Cortex ◽  
Auditory System ◽  
Acoustic Trauma ◽  
Neuron Activity ◽  
Compensatory Mechanism ◽  
Efferent Connections ◽  
Medial Geniculate ◽  
Central Auditory Pathways ◽  
Stimulation Of

In the adult auditory system, loss of input resulting from peripheral deafferentation is well known to lead to plasticity in the central nervous system, manifested as reorganization of cortical maps and altered activity throughout the central auditory pathways. The auditory system also has strong afferent and efferent connections with cortico-limbic circuitry including the prefrontal cortex and the question arises whether this circuitry is also affected by loss of peripheral input. Recent studies in our laboratory showed that PFC activation can modulate activity of the auditory thalamus or medial geniculate nucleus (MGN) in normal hearing rats. In addition, we have shown in rats that cochlear trauma resulted in altered spontaneous burst firing in MGN. However, whether the PFC influence on MGN is changed after cochlear trauma is unknown. We investigated the effects of electrical stimulation of PFC on single neuron activity in the MGN in anaesthetized Wistar rats 2 weeks after acoustic trauma or sham surgery. Electrical stimulation of PFC showed a variety of effects in MGN neurons both in sham and acoustic trauma groups but inhibitory responses were significantly larger in the acoustic trauma animals. These results suggest an alteration in functional connectivity between PFC and MGN after cochlear trauma. This change may be a compensatory mechanism increasing sensory gating after the development of altered spontaneous activity in MGN, to prevent altered activity reaching the cortex and conscious perception.

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