scholarly journals Involution of classic Kaposi sarcoma lesions under acitretin treatment Kaposi sarcoma treated with acitretin

2020 ◽  
Vol 8 (12) ◽  
pp. 3340-3343
Author(s):  
Najla Daadaa ◽  
Asmahane Souissi ◽  
Meryam Chaabani ◽  
Ines Chelly ◽  
Moez Ben Salem ◽  
...  
2009 ◽  
Vol 19 (8) ◽  
pp. 597-601 ◽  
Author(s):  
Colleen Pelser ◽  
Carmelo Dazzi ◽  
Barry I. Graubard ◽  
Carmela Lauria ◽  
Francesco Vitale ◽  
...  

2021 ◽  
Vol 31 (2) ◽  
pp. 269-270
Author(s):  
Thilo Gambichler ◽  
Victoria Brown ◽  
Alicia Siebold ◽  
Markus Stücker ◽  
Maria Bakirtzi

2013 ◽  
Vol 17 (5) ◽  
pp. 356-361 ◽  
Author(s):  
Mohammad Almohideb ◽  
A. Kevin Waiters ◽  
William Gerstein

Background: Kaposi sarcoma (KS) is a cutaneous endothelial vascular proliferation with four subtypes: iatrogenic, acquired immune deficiency syndrome (AIDS) related, African, and classic. Familial cases of KS are rare, with 72 cases reported to date, and all were described with the classic variant. The occurrence of classic KS in the Jewish population is well documented, and most of the familial classic KS cases were also reported in Jewish families. Objective: We briefly present the history, biopsies, laboratory data, diagnosis, and treatment of localized lower limb classic KS in two siblings of Jewish Eastern European ethnic descent with their response to different therapy modalities. One of our cases had the second longest reported period of follow-up for familial classic KS of 40 years.


2019 ◽  
Vol 6 (10) ◽  
Author(s):  
Sanni J Rinne ◽  
Lauri J Sipilä ◽  
Päivi Sulo ◽  
Emmanuelle Jouanguy ◽  
Vivien Béziat ◽  
...  

Abstract Familial clustering of classic Kaposi sarcoma (CKS) is rare with, approximately 100 families reported to date. We studied 2 consanguineous families, 1 Iranian and 1 Israeli, with multiple cases of adult CKS and without overt underlying immunodeficiency. We performed genome-wide linkage analysis and whole-genome sequencing to discover the putative genetic cause for predisposition. A 9-kb homozygous intronic deletion in RP11-259O2.1 in the Iranian family and 2 homozygous variants, 1 in SCUBE2 and the other in CDHR5, in the Israeli family were identified as possible candidates. The presented variants provide a robust starting point for validation in independent samples.


2014 ◽  
Vol 4 (2) ◽  
Author(s):  
Nursel Dilek ◽  
Aziz Ramazan Dilek ◽  
Yunus Saral ◽  
Recep Bedir

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