scholarly journals Potential effects of transdermal nitric oxide donor on fetal growth restriction and oligohydramnios: A case report

2020 ◽  
Vol 8 (12) ◽  
pp. 3288-3293
Author(s):  
Tsuyoshi Murata ◽  
Toma Fukuda ◽  
Tetsu Sato ◽  
Aya Kanno ◽  
Hyo Kyozuka ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Case Report ◽  
Fetal Growth ◽  
Fetal Growth Restriction ◽  
Growth Restriction ◽  
Nitric Oxide Donor

scholarly journals Effect of a nitric oxide donor on maternal hemodynamics in fetal growth restriction

2018 ◽  
Vol 51 (4) ◽  
pp. 514-518 ◽  
Author(s):  
G. M. Tiralongo ◽  
I. Pisani ◽  
B. Vasapollo ◽  
A. Khalil ◽  
B. Thilaganathan ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Fetal Growth ◽  
Fetal Growth Restriction ◽  
Growth Restriction ◽  
Nitric Oxide Donor

Advanced tubal pregnancy associated with severe fetal growth restriction: a case report

2003 ◽  
Vol 13 (6) ◽  
pp. 422-425 ◽  
Author(s):  
T. Radaelli ◽  
G. Bulfamante ◽  
I. Cetin ◽  
A. M. Marconi ◽  
G. Pardi
Keyword(s):  
Case Report ◽  
Fetal Growth ◽  
Fetal Growth Restriction ◽  
Tubal Pregnancy ◽  
Growth Restriction

scholarly journals Maternal sirolimus therapy and fetal growth restriction

2021 ◽  
Vol 8 (2) ◽  
pp. 19-24
Author(s):  
Sangay Tshering ◽  
Namkha Dorji ◽  
Youden Sonam
Keyword(s):  
Case Report ◽  
Signaling Pathway ◽  
Fetal Growth ◽  
Fetal Growth Restriction ◽  
Mtor Signaling ◽  
Growth Restriction ◽  
Mtor Signaling Pathway ◽  
Epigenetic Modulation ◽  
In Pregnancy

Fetal growth restriction associated with continued maternal sirolimus therapy in pregnancy has not been reported. We hereby present a case of maternal sirolimus therapy resulting in fetal growth restriction and propose a multi-hit model. This hypothetic model is based on inhibition of mTOR signaling pathway and epigenetic modulation. This case report adds to the paucity of literature on continued monotherapeutic maternal sirolimus in pregnancy and its adverse fetal effects.

scholarly journals Case report: Klippel-Trenaunay-Weber syndrome associated with fetal growth restriction

1996 ◽  
Vol 11 (11) ◽  
pp. 2544-2545 ◽  
Author(s):  
G. Fait ◽  
Y. Daniel ◽  
M.J. Kupfenninc ◽  
I. Gull ◽  
M.R. Peyser ◽  
...  
Keyword(s):  
Case Report ◽  
Fetal Growth ◽  
Fetal Growth Restriction ◽  
Growth Restriction ◽  
Weber Syndrome

scholarly journals Placental Cyst-a Rare Cause of Fetal Growth Restriction - A Case Report

2021 ◽  
Vol 7 (2) ◽  
pp. 109-111
Author(s):  
H P Sapna ◽  
◽  
T N Harsha ◽  
Gaana Sreenivas
Keyword(s):  
Case Report ◽  
Fetal Growth ◽  
Fetal Growth Restriction ◽  
Growth Restriction

scholarly journals Case Report: Candidate Genes Associated With Prenatal Ultrasound Anomalies in a Fetus With Prenatally Detected 1q23.3q31.2 Deletion

2021 ◽  
Vol 12 ◽  
Author(s):  
Jiahao Song ◽  
Qian Zhang ◽  
Bing Lu ◽  
Zhongshan Gou ◽  
Ting Wang ◽  
...  
Keyword(s):  
Case Report ◽  
Prenatal Diagnosis ◽  
Fetal Growth ◽  
Coronary Sinus ◽  
Fetal Growth Restriction ◽  
Growth Restriction ◽  
Chromosomal Microarray ◽  
Chromosomal Microarray Analysis ◽  
Phenotype Correlation ◽  
Renal Hypoplasia

Background: Patients with deletions involving the long arm of chromosome 1 are rare, and the main aim of this study was to refine the genotype-phenotype correlation.Case Report: In this report, a 28-year-old pregnant woman, gravida 2 para 1, at 25+4 weeks of gestation underwent ultrasound examination in our institute. The ultrasonographic findings of the fetus were as follows: (1) fetal growth restriction; (2) cleft lip and palate; (3) bilateral renal hypoplasia; (4) lateral ventriculomegaly; (5) single umbilical artery; (6) absent stomach; (7) coronary sinus dilatation with persistent left superior vena cava, ventricular septal defect and unroofed coronary sinus syndrome. Chromosomal microarray analysis of amniotic fluid from the fetus revealed a 28.025 Mb deletion in 1q23.3q31.2, spanning from position 164,559,675 to 192,584,768 (hg19).Conclusion: Genotype-phenotype correlation might improve prenatal diagnosis of fetuses with chromosome 1q deletion. PBX1 could be a candidate gene for fetal growth restriction, renal hypoplasia and congenital heart disease. Fetal growth restriction was accompanied by decreased renal volume in the fetus. Combined with ultrasonic examination, the application of chromosomal microarray analysis will provide accurate prenatal diagnosis.

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