scholarly journals Pacemaker syndrome due to atrial lead fracture

2019 ◽  
Vol 8 (1) ◽  
pp. 226-227
Author(s):  
Thomas Nguyen ◽  
Christopher Aldo Rinaldi
Keyword(s):  
Lead Fracture ◽  
Pacemaker Syndrome

MARKED FIRST-DEGREE ATRIOVENTRICULAR BLOCK ASSOCIATED WITH PSEUDO-PACEMAKER SYNDROME: A CASE REPORT

2019 ◽  
pp. 199-206
Author(s):  
О. З. Скакун ◽  
С. В. Федоров ◽  
О. С. Вербовська ◽  
І. З. Твердохліб
Keyword(s):  
Case Report ◽  
Atrioventricular Block ◽  
Heart Block ◽  
Pacemaker Implantation ◽  
Rare Condition ◽  
Permanent Pacemaker ◽  
Type I ◽  
Atrial Contraction ◽  
General Weakness ◽  
Pacemaker Syndrome

Distinctive atrioventricular type I heart block is diagnosed when the PQ interval is 0.30 s. or more. Prolongation of the PQ interval more than 0.50 s. is a very rare condition. Usually it is associated with a pseudo-pacemaker syndrome. The last one manifests itself with dizziness, syncope, general weakness, shortness of breath upon physical exertion, cough, seizures, cold sweat, a feeling of pulsation in the head, neck and abdomen, a headache, paroxysmal nocturnal dyspnea, swelling of the lower extremities, tachypnea and jugular venous pulsation. The P wave appears immediately after the previous QRS complex. Atrial contraction occurs at the moment when the ventricles don’t relax after the previous contraction; due to the fact that pressure in the ventricles at this moment is higher than in the atria, the tricuspid and mitral valves remains closed. During the atrial contraction, most of the blood is ejected not into the ventricles, but backward into the pulmonary veins from the left atrium and into the venae cavae from the right atrium. Also, an atrial kick is absent which results in a less ventricular filling. There is increased pressure in the atria leading to their distension and excessive secretion of the atrial natriuretic peptide. A case report of the distinctive atrioventricular type I heart block associated with the pseudo-pacemaker syndrome is described. The patient suffered from a pre-syncope, short-term dizziness during the previous two days, tinnitus, general weakness, feeling of pulsation in the abdomen, neck, head, which interfered with his sleep. He developed these complaints after an infectious disease, which manifested as a runny nose and sore throat. In this patient, an extremely prolonged PQ interval up to 0.70 s. was observed. Also, episodes of Mobitz I and Mobitz type II atrioventricular block were detected. During the monitoring of patient state, the interval PQ was gradually shortening, and in 1 month it reached the normаl duration. It can be assumed that in the case of distinctive atrioventricular type I heart block, a significant prolongation of the refractory period in the rapid pathways of the AV-node plays a key role in the pathogenesis of this condition. According to the recommendations of the ACC/AHA (1998), for patients with distinctive atrioventricular type I heart block accompanied by the pseudo-pacemaker syndrome and documented alleviation of symptoms with temporary AV pacing, the pacemaker implantation should be considered (IIaB). The implantation of dual chamber pacemaker may reduce symptoms and lead to an improvement in the functional state of patients, in whom shortening of the interval between atrial and ventricular contractions improves hemodynamics. For asymptomatic patients with the PQ interval of ≥ 0.30 s, pacemaker is not recommended. The distinctive atrioventricular type I heart block in patients with pseudo-pacemaker syndrome is a rare condition and often remains undiagnosed. But it may have a benign course with a gradual normalization of the PQ interval. Indications for permanent pacemaker implantation should be reviewed as this block may be completely reversible. A permanent pacemaker may be used in the case of absence of positive dynamics in a shortening of the PQ interval.    

Plutonium 238 pacemaker failure secondary to lead fracture

Heart Rhythm ◽  
2011 ◽  
Vol 8 (2) ◽  
pp. 322 ◽  
Author(s):  
Lynn N. Moran ◽  
Martin E. Matsumura ◽  
James J. Bradbury ◽  
Matthew W. Martinez
Keyword(s):  
Lead Fracture

Urgent Exchange of a HeartMate II Left Ventricular Assist Device After Percutaneous Lead Fracture

ASAIO Journal ◽  
2009 ◽  
Vol 55 (5) ◽  
pp. 523-524 ◽  
Author(s):  
Mehdi Jafar ◽  
Igor D. Gregoric ◽  
Rajko Radovancevic ◽  
William E. Cohn ◽  
Nichole McGuire ◽  
...  
Keyword(s):  
Left Ventricular Assist Device ◽  
Ventricular Assist Device ◽  
Left Ventricular ◽  
Assist Device ◽  
Heartmate Ii ◽  
Ventricular Assist ◽  
Lead Fracture ◽  
Left Ventricular Assist

Ventricular lead redundancy to prevent cardiovascular events and sudden death from lead fracture in pacemaker-dependent children

Heart Rhythm ◽  
2015 ◽  
Vol 12 (1) ◽  
pp. 111-116 ◽  
Author(s):  
Scott R. Ceresnak ◽  
Jennifer L. Perera ◽  
Kara S. Motonaga ◽  
Kishor Avasarala ◽  
Lindsey Malloy-Walton ◽  
...  
Keyword(s):  
Sudden Death ◽  
Cardiovascular Events ◽  
Dependent Children ◽  
Lead Fracture ◽  
Ventricular Lead

Dorsal root ganglion stimulation lead fractures: potential mechanisms and ways to avoid

2021 ◽  
Vol 14 (5) ◽  
pp. e241353
Author(s):  
Gaurav Chauhan ◽  
Brandon I Roth ◽  
Nagy Mekhail
Keyword(s):  
Spinal Cord ◽  
Chronic Pain ◽  
Case Report ◽  
Dorsal Root Ganglion ◽  
Dorsal Root ◽  
Unusual Case ◽  
Pain Syndrome ◽  
Structural Instability ◽  
Probable Mechanism ◽  
Lead Fracture

Dorsal root ganglion stimulation (DRGS) therapy is a rapidly emerging tool being used by pain physicians in the treatment of chronic pain. Complex regional pain syndrome (CRPS), a debilitating disease whose mechanism is still has yet to be fully elucidated, is a common pathology targeted by DRGS therapy, often better results than traditional spinal cord stimulation. DRGS therapy, however, is not bereft of complications. Lead migration and fracture are two examples in particular that are among the most common of these complications. The authors report an unusual case of lost efficacy due to lead fractures in patients with CRPS treated with DRGS. The case report narrates identification, management and probable mechanism of DRGS lead fracture. The structural instability of DRGS leads can yield distressing symptoms at any point during the therapy, and physicians should be cognisant of the complications of DRGS therapy.

Pacemaker syndrome

ESC CardioMed ◽  
2018 ◽  
pp. 2011-2016
Author(s):  
Giuseppe Boriani ◽  
Igor Diemberger
Keyword(s):  
Heart Rate ◽  
Additional Data ◽  
Cardiac Pacing ◽  
Patient Characteristics ◽  
Pharmacological Therapy ◽  
Ventricular Contraction ◽  
Intraventricular Dyssynchrony ◽  
Pacemaker Syndrome ◽  
Retrograde Conduction ◽  
Symptoms And Signs

Pacemaker syndrome (PMS) is a constellation of symptoms and signs provoked by haemodynamic and biohumoural modifications secondary to ‘any modification of the temporal sequence of atrial and ventricular contraction’ induced by artificial pacing. This clinical entity was early described after the development of artificial pacing but it still lacks a definite characterization. In particular, several mechanisms may induce PMS beyond ventriculoatrial retrograde conduction in a patient with single-chamber pacing, with the interplay of patient characteristics, device hardware/programming, and pharmacological therapy. The contemporary concept of PMS is strictly connected with the idea of ‘physiological pacing’ for which PMS represent the negative extreme. Moreover, any improvement in cardiac pacing provides additional data on PMS such as atrioventricular timing, intraventricular dyssynchrony, and heart rate adaptation.

Diagnosing and treating pacemaker syndrome

2001 ◽  
Vol 21 (1) ◽  
pp. 24-31 ◽  
Author(s):  
CJ Van Orden Wallace
Keyword(s):  
Pacemaker Syndrome
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