Transhepatic portal vein access for balloon dilation of right upper pulmonary vein stenosis following infradiaphragmatic total anomalous pulmonary venous drainage repair

2011 ◽  
Vol 78 (5) ◽  
pp. 698-701 ◽  
Author(s):  
Aphrodite Tzifa ◽  
Irfan Ahmed ◽  
Eric Rosenthal
Keyword(s):  
Portal Vein ◽  
Pulmonary Vein ◽  
Balloon Dilation ◽  
Venous Drainage ◽  
Pulmonary Vein Stenosis ◽  
Anomalous Pulmonary Venous Drainage ◽  
Vein Access ◽  
Vein Stenosis

scholarly journals Repair Type Influences Mode of Pulmonary Vein Stenosis in Total Anomalous Pulmonary Venous Drainage

2015 ◽  
Vol 100 (2) ◽  
pp. 654-662 ◽  
Author(s):  
Mauro Lo Rito ◽  
Tamadhir Gazzaz ◽  
Travis Wilder ◽  
Arezou Saedi ◽  
Devin Chetan ◽  
...  
Keyword(s):  
Pulmonary Vein ◽  
Venous Drainage ◽  
Pulmonary Vein Stenosis ◽  
Anomalous Pulmonary Venous Drainage ◽  
Vein Stenosis

Relentless pulmonary vein stenosis after repair of total anomalous pulmonary venous drainage

1998 ◽  
Vol 66 (5) ◽  
pp. 1514-1519 ◽  
Author(s):  
Christopher A Caldarone ◽  
Hani K Najm ◽  
Margit Kadletz ◽  
Jeffrey F Smallhorn ◽  
Robert M Freedom ◽  
...  
Keyword(s):  
Pulmonary Vein ◽  
Venous Drainage ◽  
Pulmonary Vein Stenosis ◽  
Anomalous Pulmonary Venous Drainage ◽  
Vein Stenosis

scholarly journals The Role of Elevated Wall Shear Stress in Progression of Pulmonary Vein Stenosis: Evidence from Two Case Studies

Children ◽  
2021 ◽  
Vol 8 (9) ◽  
pp. 729
Author(s):  
Peter E. Hammer ◽  
Kerry McEnaney ◽  
Ryan Callahan ◽  
Christopher W. Baird ◽  
David M. Hoganson ◽  
...  
Keyword(s):  
Shear Stress ◽  
Wall Shear Stress ◽  
Disease Progression ◽  
Pulmonary Vein ◽  
Pulmonary Veins ◽  
Balloon Dilation ◽  
Pulmonary Vein Stenosis ◽  
Multiple Time ◽  
Wall Shear ◽  
Vein Stenosis

Pulmonary vein stenosis is a serious condition characterized by restriction or blockage due to fibrotic tissue ingrowth that develops in the pulmonary veins of infants or children. It is often progressive and can lead to severe pulmonary hypertension and death. Efforts to halt or reverse disease progression include surgery and catheter-based balloon dilation and stent implantation. Its cause and mechanism of progression are unknown. In this pilot study, we propose and explore the hypothesis that elevated wall shear stress at discrete pulmonary venous sites triggers stenosis. To assess this theory, we retrospectively analyzed cardiac catheterization, lung scan, and X-ray computed tomography data to estimate wall shear stress in the pulmonary veins at multiple time points during disease progression in two patients. Results are consistent with the existence of a level of elevated wall shear stress above which the disease is progressive and below which progression is halted. The analysis also suggests the possibility of predicting the target lumen size necessary in a given vein to reduce wall shear stress to normal levels and remove the trigger for stenosis progression.

scholarly journals The impact of right ventricular pressure and function on survival in patients with pulmonary vein stenosis

2018 ◽  
Vol 8 (2) ◽  
pp. 204589401877689 ◽  
Author(s):  
Michelle C. Sykes ◽  
Christina Ireland ◽  
Julia E. McSweeney ◽  
Emily Rosenholm ◽  
Kristofer G. Andren ◽  
...  
Keyword(s):  
Right Ventricular ◽  
Pulmonary Vein ◽  
Cardiac Catheterization ◽  
Systolic Function ◽  
Balloon Dilation ◽  
Pulmonary Vein Stenosis ◽  
Pulmonary Hemodynamics ◽  
Vein Stenosis ◽  
The Impact ◽  
Rv Function

Pulmonary vein stenosis (PVS) is associated with pulmonary hypertension (PH), but there is little information regarding the impact of PH on right ventricular (RV) systolic function and survival. We conducted a retrospective cohort study of our patients to explore this and other aspects of pulmonary hemodynamics with PVS. RV function was assessed using qualitative two-dimensional echocardiography. The ratio of systolic pulmonary artery (PA) and aortic pressures (PA:Ao) at cardiac catheterization reflected pulmonary hemodynamics. Reactivity testing employed inhaled nitric oxide + 100% fiO2, or 100% fiO2 only; “reactivity” was a ≥ 20% decrease in PA:Ao. There were 105 PVS patients, although not all had data at every time point. (1) The mean PA:Ao at first cardiac catheterization (n = 77) was 0.79 ± 0.36; at last catheterization (n = 54), PA:Ao = 0.69 ± 0.30; 90% had systolic PAP > one-half systemic. Survival was shorter with PA:Ao > 0.5. (2) Differences in survival relative to RV dysfunction on the first echocardiogram were not significant, although they were using the last echocardiogram. (3) The magnitude of RV dysfunction was positively correlated with PA:Ao. (4) Balloon dilation of PV acutely decreased PA:Ao (–0.13 ± 0.37, P = 0.03 [n = 40 patients]). 5. Of 20 patients tested, 13 were acutely reactive to vasodilators. PH is a major feature of PVS. Reduced RV function and PA:Ao appear to be predictors of survival. Given the importance of PH in this disease, clinical studies of PVS treatments should include measures of PAP and RV function as important variables of interest.

TIMELY DIAGNOSTICS OF CATHETER ABLATION COMPLICATIONS: CLINICAL CASE OF PULMONARY VEIN STENOSIS

2018 ◽  
Vol 8 (4) ◽  
pp. 248-255
Author(s):  
O.V. Sapelnikov ◽  
◽  
E.V. Merkulov ◽  
O.A. Nikolaeva ◽  
D.I. Cherkashin ◽  
...  
Keyword(s):  
Catheter Ablation ◽  
Pulmonary Vein ◽  
Clinical Case ◽  
Pulmonary Vein Stenosis ◽  
Vein Stenosis
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