Percutaneous closure of a postinfarction ventricular septal defect and an iatrogenic left ventricular free-wall perforation using two Amplatzer muscular VSD occluders

2009 ◽  
pp. NA-NA
Author(s):  
Parham Eshtehardi ◽  
Ali Garachemani ◽  
Bernhard Meier
Keyword(s):  
Ventricular Septal Defect ◽  
Septal Defect ◽  
Left Ventricular ◽  
Percutaneous Closure ◽  
Ventricular Free Wall ◽  
Free Wall ◽  
Left Ventricular Free Wall ◽  
Postinfarction Ventricular Septal Defect ◽  
Wall Perforation ◽  
Muscular Vsd

Percutaneous Closure of Left Ventricular Free Wall Rupture with Associated False Aneurysm to Prevent Cardioembolic Stroke

2008 ◽  
Vol 17 (3) ◽  
pp. 250-253 ◽  
Author(s):  
Wil Harrison ◽  
Peter N. Ruygrok ◽  
Sally Greaves ◽  
Namal Wijesinghe ◽  
Hamish Charleson ◽  
...  
Keyword(s):  
False Aneurysm ◽  
Left Ventricular ◽  
Percutaneous Closure ◽  
Cardioembolic Stroke ◽  
Ventricular Free Wall ◽  
Free Wall ◽  
Left Ventricular Free Wall ◽  
Free Wall Rupture

scholarly journals Percutaneous Closure of a Left Ventricular Free-Wall Rupture Site

Circulation ◽  
2006 ◽  
Vol 113 (18) ◽  
Author(s):  
Patrick A. Gladding ◽  
Peter N. Ruygrok ◽  
Sally C. Greaves ◽  
Ivor L. Gerber ◽  
Andrew W. Hamer
Keyword(s):  
Left Ventricular ◽  
Percutaneous Closure ◽  
Ventricular Free Wall ◽  
Free Wall ◽  
Left Ventricular Free Wall ◽  
Free Wall Rupture ◽  
Rupture Site

Percutaneous Ventricular Assist Device and Extracorporeal Membrane Oxygenation Support in a Patient with Postinfarction Ventricular Septal Defect and Free Wall Rupture

2013 ◽  
Vol 16 (3) ◽  
pp. 150 ◽  
Author(s):  
Igor D. Gregoric ◽  
Tomaz Mesar ◽  
Biswajit Kar ◽  
Sriram Nathan ◽  
Rajko Radovancevic ◽  
...  
Keyword(s):  
Ventricular Septal Defect ◽  
Extracorporeal Membrane Oxygenation ◽  
Ventricular Assist Device ◽  
Septal Defect ◽  
Ventricular Free Wall ◽  
Free Wall ◽  
Assist Device ◽  
Ventricular Assist ◽  
Free Wall Rupture ◽  
Postinfarction Ventricular Septal Defect

We describe the case of a 54-year-old woman with a postinfarction ventricular septal defect (VSD) and ventricular free wall rupture who was stabilized with a percutaneous ventricular assist device (pVAD) to allow for myocardial infarct stabilization. Following the rupture of the right ventricular free wall and cardiopulmonary arrest on hospital day 10, pVAD support was promptly converted to extracorporeal membrane oxygenation (ECMO) support for stabilization. After surgical repair was completed, pVAD support was continued for 4 days to allow recovery. The patient was discharged on postoperative day 11 and is alive and well 4 years later. Postinfarction VSD with free wall rupture may be salvaged with pVAD and ECMO support.

Intracellular EDEMA does not adversely affect the ability to cryopreserve intact hearts

1993 ◽  
Vol 51 ◽  
pp. 278-279
Author(s):  
CL Hastings ◽  
RD Carlton ◽  
FG Lightfoot ◽  
AF Tryka
Keyword(s):  
Cell Injury ◽  
Median Sternotomy ◽  
Left Ventricular ◽  
Ventricular Free Wall ◽  
Hypoxic Cell ◽  
Free Wall ◽  
Sprague Dawley ◽  
Left Ventricular Free Wall ◽  
Sprague Dawley Rats

The earliest ultrastructural manifestation of hypoxic cell injury is the presence of intracellular edema. Does this intracellular edema affect the ability to cryopreserve intact myocardium? To answer this guestion, a model for anoxia induced intracellular edema (IE) was designed based on clinical intraoperative myocardial preservation protocol. The aortas of 250 gm male Sprague-Dawley rats were cannulated and a retrograde flush of Plegisol at 8°C was infused over 90 sec. The hearts were excised and placed in a 28°C bath of Lactated Ringers for 1 h. The left ventricular free wall was then sliced and the myocardium was slam frozen. Control rats (C) were anesthetized, the hearts approached by median sternotomy, and the left ventricular free wall frozen in situ immediately after slicing. The slam frozen samples were obtained utilizing the DDK PS1000, which was precooled to -185°C in liguid nitrogen. The tissue was in contact with the metal mirror for a dwell time of 20 sec, and stored in liguid nitrogen until freeze dry processing (Lightfoot, 1990).

scholarly journals Echocardiographic Findings in Friedreich's Ataxia

1976 ◽  
Vol 3 (4) ◽  
pp. 329-332 ◽  
Author(s):  
H.F. Gattiker ◽  
A. Davignon ◽  
A. Bozio ◽  
J. Batlle-Diaz ◽  
G. Geoffroy ◽  
...  
Keyword(s):  
Friedreich’S Ataxia ◽  
Left Ventricular ◽  
Friedreich's Ataxia ◽  
Ventricular Free Wall ◽  
Slight Reduction ◽  
Free Wall ◽  
Left Ventricular Free Wall ◽  
Echocardiographic Examination ◽  
Septal Hypertrophy ◽  
Echocardiographic Findings

SUMMARY:Echocardiographic examination of 21 patients with Friedreich's ataxia (age 7 to 28 years) showed cardiac abnormalities in 90% of the cases. They were characterized by varying degrees of septal hypertrophy in 81%, left ventricular free wall hypertrophy in 61%, and a slight reduction of left ventricular internal dimension in 57% of the cases. Asymmetric septal hypertrophy (ASH) with a septal/left ventricular free wall ratio of over 1.3 was found in 29% of the cases, and systolic anterior motion (SAM) of the mitral valve in three patients. Two other patients showed evidence of a different type of cardiomyopathy with marked symmetric left ventricular hypertrophy and marked left ventricular enlargement.

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