Nonpenetrating traumatic complete heart block and myocardial infarction with normal coronaries: A case report with review of the literature

1983 ◽  
Vol 9 (1) ◽  
pp. 63-74 ◽  
Author(s):  
Willard J. Kennedy ◽  
Marandapalli R. Sridharan ◽  
Nancy C. Flowers
Keyword(s):  
Myocardial Infarction ◽  
Case Report ◽  
Heart Block ◽  
Complete Heart Block ◽  
Review Of The Literature

scholarly journals Complete Heart Block in Thyrotoxicosis, Is It a Manifestation of Thyroid Storm? A Case Report and Review of the Literature

2012 ◽  
Vol 2012 ◽  
pp. 1-3 ◽  
Author(s):  
Rashed Al Bannay ◽  
Aysha Husain ◽  
Saeed Khalaf
Keyword(s):  
Case Report ◽  
Atrioventricular Block ◽  
Heart Block ◽  
Cardiac Involvement ◽  
Complete Heart Block ◽  
Rare Complication ◽  
Thyroid Storm ◽  
Common Disease ◽  
Review Of The Literature

Thyrotoxicosis complicated by advance degree atrioventricular block, a rare complication of a common disease. The term apathetic thyrotoxicosis, where palpitations and cardiac involvement are the sole manifestations of disease, is well known. Thyroxin's ability to sensitize the catecholamine receptors causing tachyarrhythmias is well addressed. However, as an aetiology for advanced heart block, thyrotoxicosis is ranked as one of the rarest.

Cardiac complications of wegener granulomatosis: A case report of complete heart block and review of the literature

1980 ◽  
Vol 10 (2) ◽  
pp. 148-154 ◽  
Author(s):  
Joseph Z. Forstot ◽  
Paul A. Overlie ◽  
Gwynne K. Neufeld ◽  
Catherine E. Harmon ◽  
S.Lance Forstot
Keyword(s):  
Case Report ◽  
Heart Block ◽  
Complete Heart Block ◽  
Cardiac Complications ◽  
Review Of The Literature ◽  
Wegener Granulomatosis

Drugs and pacemakers for complete heart block

1967 ◽  
Vol 5 (5) ◽  
pp. 19-20
Keyword(s):  
Myocardial Infarction ◽  
Heart Failure ◽  
Heart Rate ◽  
Heart Disease ◽  
Ischaemic Heart Disease ◽  
Heart Block ◽  
Complete Heart Block ◽  
Bundle Of His

Complete heart block can occur in ischaemic heart disease, and can acutely complicate myocardial infarction. Most other cases are associated with fibrosis of the bundle of His of unknown cause, or are congenital. In some patients with chronic heart block, especially the congenital type, adequate output is maintained. In other patients chronic or intermittent heart block may cause Stokes-Adams attacks, or heart failure may not respond to digitalis and diuretics until the heart rate is increased. These require treatment by drugs or, when this fails, by use of anartifical pacemaker.

scholarly journals Myocarditis presenting as typical acute myocardial infarction: A case report and review of the literature

2020 ◽  
Vol 8 (2) ◽  
pp. 415-424
Author(s):  
Ya-Min Hou ◽  
Peng-Xi Han ◽  
Xia Wu ◽  
Jing-Ru Lin ◽  
Fei Zheng ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Case Report ◽  
Review Of The Literature

scholarly journals ACUTE MYOCARDIAL INFARCTION;

2013 ◽  
Vol 20 (03) ◽  
pp. 332-340
Author(s):  
ATIF SITWAT HAYAT ◽  
MUHAMMAD ADNAN BAWANY ◽  
JAWAD AHMED QADRI ◽  
Kiran Khalil
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Heart Block ◽  
Complete Heart Block ◽  
Tertiary Care ◽  
Anterior Wall ◽  
P Value ◽  
Inferior Wall ◽  
Care Hospital ◽  
Significant Difference

Background: Ischemic heart disease is the most common cause for complete heart block (CHB) and sudden death. Heartblocks may occur as complications of acute myocardial infarction (AMI) and are associated with increased mortality. The aim of thisstudy is to determine the frequency of complete heart block (CHB) in acute myocardial infarction at a tertiary care hospital. Place andduration: This study was conducted in Cardiology Department of Liaquat University of Medical and Health Sciences from 1st August2009 to 31st January 2010. Study Design: Cross sectional and descriptive study. Materials and Methods: ST segment elevation equal toor more than 1mm (0.1mv) in two of these leads II, III and aVF. Rise in serum creatinine kinase level (CPK Level) more than twice thenormal value along with CK-MB fraction more than 6% of CPK value. Patients with history of chest pain, shortness of breath, nausea,vomiting and unconsciousness were enrolled in the study. The cardiac enzymes tropinin T was also performed at bed side by venousblood sample. Results: Total of 87 patients were included, prevalence of heart blocks was 27.58%. Anterior wall MI was in 50(57.5%)patients. Of these, 13(54.2%) had complete heart block. Inferior wall MI was in 37(42.5%) cases, of these, 11(45.8%) were found withcomplete heart block. There was no significant difference between anterior wall MI and inferior wall MI with complete heart block (P value> 0.05). Mortality was 2.3% with anterior wall MI. Conclusions: Development of complete heart blocks has important prognosticsignificance. Complete heart block was frequent complication of myocardial infarction.

Sign in / Sign up

Export Citation Format

Share Document