scholarly journals Monocytes Engineered with iSNAP Inhibit Human B‐Lymphoma Progression

2021 ◽  
Author(s):  
Haohsiang Wu ◽  
Siamak Amirfakhri ◽  
Hsin‐Hung Lin ◽  
Hannah Hollandsworth ◽  
Filemoni Filemoni ◽  
...  
Keyword(s):  
B Lymphoma

The role of bcl-xL in CD40-mediated rescue from anti-μ-induced apoptosis in WEHI-231 B lymphoma cells

1995 ◽  
Vol 25 (5) ◽  
pp. 1352-1357 ◽  
Author(s):  
Michael S. K. Choi ◽  
Lawrence H. Boise ◽  
Alexander R. Gottschalk ◽  
José Quintans ◽  
Craig B. Thompson ◽  
...  
Keyword(s):  
Lymphoma Cells ◽  
B Lymphoma ◽  
B Lymphoma Cells ◽  
Wehi 231 ◽  
Induced Apoptosis

An intermediate-conductance Ca2+-activated K+ channel mediates B lymphoma cell cycle progression induced by serum

2007 ◽  
Vol 454 (6) ◽  
pp. 945-956 ◽  
Author(s):  
Jing Wang ◽  
Yu-Qing Xu ◽  
You-You Liang ◽  
Rafael Gongora ◽  
David G. Warnock ◽  
...  
Keyword(s):  
Cell Cycle ◽  
Cell Cycle Progression ◽  
Lymphoma Cell ◽  
K Channel ◽  
Cycle Progression ◽  
B Lymphoma

Therapy of Advanced B-Lymphoma Xenografts with a Combination of 90Y-anti-CD22 IgG (Epratuzumab) and Unlabeled Anti-CD20 IgG (Veltuzumab)

2008 ◽  
Vol 14 (19) ◽  
pp. 6154-6160 ◽  
Author(s):  
M. Jules Mattes ◽  
Robert M. Sharkey ◽  
Habibe Karacay ◽  
Myron S. Czuczman ◽  
David M. Goldenberg
Keyword(s):  
B Lymphoma ◽  
Anti Cd20

Selective loss of H-2Ds antigen on a murine B lymphoma due to a post-transcriptional block in expression

1995 ◽  
Vol 32 (14-15) ◽  
pp. 1011-1020 ◽  
Author(s):  
Hua Luo ◽  
Lynne Sopchak ◽  
Stephen P. Lerman ◽  
Steven R. King
Keyword(s):  
B Lymphoma ◽  
Selective Loss

Efficacy of anti-CD20 chimeric Fab’ fragment on proliferation of B lymphoma cells

2001 ◽  
Vol 46 (14) ◽  
pp. 1182-1186 ◽  
Author(s):  
Zengzu Lai ◽  
Dongsheng Xiong ◽  
Dongmei Fan ◽  
Yuanfu Xu ◽  
Hanzhi Liu ◽  
...  
Keyword(s):  
Fab Fragment ◽  
Lymphoma Cells ◽  
B Lymphoma ◽  
B Lymphoma Cells ◽  
Anti Cd20

CD4+ T cells kill Id+ B-lymphoma cells: FasLigand-Fas interaction is dominant in vitro but is redundant in vivo

2004 ◽  
Vol 53 (12) ◽  
pp. 1135-1145 ◽  
Author(s):  
Katrin U. Lundin ◽  
Valentina Screpanti ◽  
Hilde Omholt ◽  
Peter O. Hofgaard ◽  
Hideo Yagita ◽  
...  
Keyword(s):  
T Cells ◽  
Cd4 T Cells ◽  
Lymphoma Cells ◽  
B Lymphoma ◽  
B Lymphoma Cells

An intronic 10-base-pair deletion in a class II A beta gene affects RNA processing

1989 ◽  
Vol 9 (10) ◽  
pp. 4402-4408
Author(s):  
Z Ghogawala ◽  
E Choi ◽  
K R Daly ◽  
L R Blanco ◽  
I J Griffith ◽  
...  
Keyword(s):  
Rna Processing ◽  
Rna Splicing ◽  
Rna Stability ◽  
Class Ii ◽  
Base Pairs ◽  
B Lymphoma ◽  
Histocompatibility Complex ◽  
Base Pair Deletion ◽  
Mutant Cells ◽  
Beta Gene

Several biologically important examples of posttranscriptionally regulated genes have recently been described (T. Gerster, D. Picard, and W. Schaffner, Cell 45:45-52, 1986; R. Reeves, T.S. Elton, M.S. Nissen, D. Lehn, and K.R. Johnson, Proc. Natl. Acad. Sci. USA 84:6531-6535, 1987; H.A. Young, L. Varesio, and P. Hwu, Mol. Cell. Biol. 6:2253-2256, 1986). Little is known, however, regarding sequences that mediate posttranscriptional RNA stability. Characterization in our laboratory of a mutant murine B lymphoma, M12.C3, revealed a posttranscriptional defect affecting the synthesis of a major histocompatibility complex class II gene (A beta d) whose product normally controls both the specificity and magnitude of the immune response. Molecular studies revealed that the mutation responsible for diminished A beta d gene expression was an intronic deletion of 10 base pairs (bp) located 99 bp 5' of the third exon. This deletion lies in a region not known to be critical for accurate and efficient splicing. Furthermore, sequence analysis of amplified A beta-specific cDNA demonstrated that the small number of A beta d transcripts produced in the mutant cells was correctly spliced. It appears that the mechanism by which this intronic 10-bp deletion acts to decrease RNA stability is unlikely to be at the level of RNA splicing.

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