Aneurysm Formation After Stent Grafting in Vascular Behçet's Disease

2018 ◽  
Vol 70 (2) ◽  
pp. 322-322 ◽  
Author(s):  
Suguru Honda ◽  
Fumio Hirano ◽  
Mariko Mouri ◽  
Hisanori Hasegawa ◽  
Hitoshi Kohsaka
Keyword(s):  
Behçet’S Disease ◽  
Behcet’S Disease ◽  
Behçet's Disease ◽  
Behcet's Disease ◽  
Stent Grafting ◽  
Aneurysm Formation

scholarly journals Endovascular Stent-Grafting for Recurrent Aneurysm in Behcet's Disease

2005 ◽  
Vol 46 (4) ◽  
pp. 745-749 ◽  
Author(s):  
Hiroshi Watanabe ◽  
Hirotaka Oda ◽  
Tsuyoshi Yoshida ◽  
Masayuki Yamaura ◽  
Kazuyoshi Takahashi ◽  
...  
Keyword(s):  
Behçet’S Disease ◽  
Behcet’S Disease ◽  
Behçet's Disease ◽  
Endovascular Stent ◽  
Behcet's Disease ◽  
Stent Grafting ◽  
Endovascular Stent Grafting

scholarly journals Aorto-atrial fistula without aneurysm formation in Behcet's disease

Heart ◽  
1998 ◽  
Vol 80 (2) ◽  
pp. 200-201 ◽  
Author(s):  
A Melua ◽  
N Campbell ◽  
D McCluskey ◽  
S W MacGowan
Keyword(s):  
Behçet’S Disease ◽  
Behcet’S Disease ◽  
Behçet's Disease ◽  
Behcet's Disease ◽  
Aneurysm Formation

Coagulation and Fibrinolytic Activity in Behçet's Disease

1991 ◽  
Vol 66 (03) ◽  
pp. 292-294 ◽  
Author(s):  
K K Hampton ◽  
M A Chamberlain ◽  
D K Menon ◽  
J A Davies
Keyword(s):  
Plasminogen Activator ◽  
Tissue Plasminogen Activator ◽  
Behçet’S Disease ◽  
Fibrinolytic Activity ◽  
Behcet’S Disease ◽  
Behçet's Disease ◽  
Plasminogen Activator Inhibitor ◽  
Behcet's Disease ◽  
Tissue Plasminogen ◽  
Von Willebrand

SummaryCoagulation and fibrinolytic activities were studied in 18 subjects with Behçet's disease and compared with results from 14 matched control patients suffering from sero-negative arthritis. Significantly higher plasma concentrations (median and range) were found in Behçet's patients for the following variables: fibrinogen 3.7 (1.7-6.9) vs 3.0 (2.0-5.1) g/1, p <0.05; von Willebrand factor antigen, 115 (72-344) vs 74 (60-119)%, p <0.002; plasminogen activator activity (106/ECLT2) 219 (94-329) vs 137 (78-197) units, p <0.002; tissue plasminogen activator inhibitor (t-PA-I) activity, 9.1 (5.5-19.3) vs 5.1 (1.8-12.0) IU/ml, p <0.002; and PAI-1 antigen, 13.9 (4.5-20.9) vs 6.4 (2.4-11.1) ng/ml, p <0.002. Protein C antigen was significantly lower: 97 (70-183) vs 126 (96-220)%, p <0.02. No differences were observed in antithrombin III activity or antigen, factor VIII coagulant activity, fibrinopeptides A and Bβ15-42, plasminogen, α-2-antiplasmin, functional and immunological tissue-plasminogen activator, thrombin-antithrombin complexes and D-dimer. Levels of tissue plasminogen activator inhibitor (activity and antigen) correlated with disease activity while fibrinogen and von Willebrand factor concentrations did not. Seven of the 18 subjects with Behçet's disease had suffered thrombotic events but it was not possible to distinguish these from the 11 patients without thrombosis using the assays performed. The results suggest the abnormal fibrinolytic activity in Behçet's disease is due to increased inhibition of tissue plasminogen activator. No abnormality of coagulation or fibrinolytic activity specific to Behçet's disease was detected.

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