scholarly journals Successful non-operative management of traumatic extrahepatic portal venous injury without intraperitoneal hemorrhage: a case report

2016 ◽  
Vol 4 (2) ◽  
pp. 205-208 ◽  
Author(s):  
Kunio Hamanaka ◽  
Yuusuke Hirokawa ◽  
Tsuyoshi Itoh ◽  
Mitsuhiro Fujino ◽  
Kenichi Kano ◽  
...  
2021 ◽  
Vol 9 (7) ◽  
Author(s):  
Mohammed S. Foula ◽  
Mohammed Sharroufna ◽  
Zahra H. Alshammasi ◽  
Omar S. Alothman ◽  
Bayan A. Almusailh ◽  
...  

2020 ◽  
Vol 18 (2) ◽  
Author(s):  
Abdul Malek Mohamad ◽  
Azrin Waheedy Ahmad ◽  
Junaini Kasian

Introduction: We aim to report an uncommon case of post ERCP perforation that effectively managed conservatively in non-hepatobiliary surgery centre. Case report: A 46-year-old man diagnosed to have obstructive jaundice secondary to distal common bile duct (CBD) stone. He underwent ERCP at a private centre, sphincterotomy was performed, but, the operator had failed to insert the stent and complicated with post ERCP perforation evidenced by contrast extravasation beyond 1/3rd of the CBD and referred to our centre. Patient was subjected for re ERCP. There were difficulties in cannulating the CBD and stent was inserted. Cholangiogram revealed contrast leak around the pancreatic duct and bifurcation of hepatic duct. There was no evidence of CBD stone. Computed Tomography (CT) of the abdomen revealed extensive subcutaneous emphysema on the right side of the abdomen to right inguinal region, extensive retroperitoneal free air and pneumoperitoneum, but there were no free fluid or contrast extravasation. The patient subjected for non-operative management (NOM) for the complication and kept fasting with total parenteral nutrition and intravenous antibiotic. He recovered well with the opted management. Patient was programmed with Gastrograffin study after 10 days that showed no evidence of contrast leak to suggest free bowel injury. He was allowed orally after that and was discharged well after 15 days with stent in situ. During follow up, he was well, and the stent removed after 3 months. Patient planned for laparoscopic cholecystectomy and on table cholangiogram. Conclusion:  Post ERCP perforation is uncommon but lethal. Early recognition of the complication is crucial hence appropriate management can be arranged to avoid death. To date, surgery is not the only choice available to manage this complication.


2011 ◽  
Vol 6 (1) ◽  
pp. 1-3
Author(s):  
Mohsen Kalhor ◽  
S. Ameneh Sabzi ◽  
Mehdi Molaei

2020 ◽  
Vol 77 ◽  
pp. 862-865
Author(s):  
Norah Alsubaie ◽  
Bushr Mrad ◽  
Abdullah Albdah ◽  
Nadia Aljomah ◽  
Thamer Nouh

2015 ◽  
Vol 26 (4) ◽  
pp. 306
Author(s):  
Chye-Yang Lim ◽  
Chun-Hao Chen ◽  
Kun-Hung Shen

2021 ◽  
Vol 21 (1) ◽  
Author(s):  
Alberto Friziero ◽  
Cosimo Sperti ◽  
Gianfranco Da Dalt ◽  
Nicola Baldan ◽  
Gianpietro Zanchettin ◽  
...  

Abstract Background Small bowel obstruction is one of the leading reasons for accessing to the Emergency Department. Food poisoning from Clostridium botulinum has emerged as a very rare potential cause of small bowel obstruction. The relevance of this case report regards the subtle onset of pathognomonic neurological symptoms, which can delay diagnosis and subsequent life-saving treatment. Case presentation A 24-year-old man came to our Emergency Department complaining of abdominal pain, fever and sporadic self-limiting episodes of diplopia, starting 4 days earlier. Clinical presentation and radiological imaging suggested a case of small bowel obstruction. Non-operative management was adopted, which was followed by worsening of neurological signs. On specifically questioning the patient, we discovered that his parents had experienced similar, but milder symptoms. The patient also recalled eating home-made preserves some days earlier. A clinical diagnosis of foodborne botulism was established and antitoxin was promptly administered with rapid clinical resolution. Conclusions Though very rare, botulism can mimic small bowel obstruction, and could be associated with a rapid clinical deterioration if misdiagnosed. An accurate family history, frequent clinical reassessments and involvement of different specialists can guide to identify this unexpected diagnosis.


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