Role of the adrenal cortex in maturation of the lymphoid system and immunological competence; the effects of aminoglutethimide in suckling mice

1973 ◽  
Vol 137 (3) ◽  
pp. 231-255 ◽  
Author(s):  
Sam L. Clark ◽  
Gary B. Schneider
Keyword(s):  
Adrenal Cortex ◽  
Suckling Mice ◽  
Lymphoid System

scholarly journals The Physiological Role of the Lymphoid System

1970 ◽  
Vol 245 (15) ◽  
pp. 3906-3910
Author(s):  
Asit K. Lahiri ◽  
William M. Mitchell ◽  
Victor A. Najjar
Keyword(s):  
Physiological Role ◽  
Lymphoid System

ROLE OF THE ADRENAL CORTEX IN DISEASE

1951 ◽  
Vol 2 (17) ◽  
pp. 549-553
Author(s):  
Keith Harrison
Keyword(s):  
Adrenal Cortex

Gut peptides and the autonomic nervous system in cysteamine-induced duodenal ulcer. Role of adrenal cortex and medulla

1990 ◽  
Vol 31 (3) ◽  
pp. 258
Author(s):  
Kimitoshi Koto ◽  
Kazuhiro Imatake ◽  
Hideki Mochizuki ◽  
Tomio Haramoto ◽  
Emiko Ito ◽  
...  
Keyword(s):  
Duodenal Ulcer ◽  
Nervous System ◽  
Autonomic Nervous System ◽  
Adrenal Cortex ◽  
Gut Peptides ◽  
Autonomic Nervous

Cache Valley virus: experimental infection in Culiseta inornata

1980 ◽  
Vol 26 (3) ◽  
pp. 287-290 ◽  
Author(s):  
Linda C. Corner ◽  
Audrei K. Robertson ◽  
Lance B. Hayles ◽  
John O. Iversen
Keyword(s):  
Blood Meal ◽  
Lethal Dose ◽  
Experimental Demonstration ◽  
Female Progeny ◽  
Cache Valley Virus ◽  
Suckling Mice ◽  
Cache Valley ◽  
Culiseta Inornata ◽  
Minimum Infection Rate

Experiments were conducted to examine the dynamics of Cache Valley virus in Culiseta inornata, the probable chief vector of the virus. Of about 1500 laboratory reared C. inornata exposed to viraemic suckling mice, 72 took a blood meal. A relatively high percentage (93%) of the latter mosquitoes became infected. The virus increased more than 100-fold in the experimentally infected mosquitoes. The increasing viral titres were noticed after 7 days and after 15 days. Peak titres averaged 105.0 (mean suckling mouse intracerebral lethal dose) SMICLD50/0.02 mL. The infected mosquitoes had peak titres until at least 35 days after the mosquitoes ingested blood from infected suckling mice. A single transmission of virus by bite occurred 30 days after the viraemic blood meal. Transovarial transmission was demonstrated. In two experiments, 3.3 and 2.9% of infected mosquitoes transovarially transmitted Cache Valley virus to both male and female progeny. The minimum infection rate for the progeny was 2.05/1000 mosquitoes. This is the first reported experimental demonstration of transovarian transmission in a species of mosquito which overwinters as an adult. The role of transovarian transmission in the natural maintenance of Cache Valley virus remains undetermined.

The significance of biogenic amines in neuroendocrine pathology in obstetrics and gynecology

1986 ◽  
Vol 67 (4) ◽  
pp. 241-245
Author(s):  
Z. Sh. Gilyazutdinova
Keyword(s):  
Parathyroid Hormone ◽  
Biogenic Amines ◽  
Adrenal Cortex ◽  
Obstetrics And Gynecology ◽  
Advanced Training ◽  
Great Honor

On the traditional day of the Kazan Institute for Advanced Training of Physicians, I had the great honor to give an act speech in front of its staff. It was based on the results of scientific works of the staff of the department in 1981-1986 mainly on studying the role of biogenic amines and some hormones (prolactin, hormones of adrenal cortex, parathyroid hormone and calcitonin) in the pathogenesis of some neuroendocrine diseases, which occur in obstetric-gynecological practice.

WILMS' TUMOR AND CONGENITAL HEMIHYPERTROPHY: REPORT OF FIVE NEW CASES AND REVIEW OF LITERATURE

PEDIATRICS ◽  
1967 ◽  
Vol 40 (5) ◽  
pp. 886-899
Author(s):  
Joseph F. Fraumeni ◽  
Clementina F. Geiser ◽  
Miriam D. Manning
Keyword(s):  
Cancer Research ◽  
Adrenal Cortex ◽  
Wilms Tumor ◽  
Cytogenetic Study ◽  
Congenital Defect ◽  
Familial Occurrence ◽  
Review Of Literature ◽  
Childhood Neoplasms ◽  
Congenital Hemihypertrophy

Among 225 patients with Wilms' tumor seen at the Children's Cancer Research Foundation in Boston, 7 had congenital hemihypertrophy (a frequency of 1:32), bringing to 26 the number of cases reported in the literature with this association. In one child the cytogenetic study of leukocyte cultures revealed elongation of the long arms of both No. 16 chromosomes; each parent and two of four siblings had a similar anomaly affecting one chromosome of pair 16. Dermatoglyphics on this patient and three others in the series were unremarkable, as were studies of urinary gonadotropin excretion. From a review of all cases reported with Wilms' tumor and hemihypertrophy, little was found to indicate a relationship to other disorders, such as Silver's syndrome or neurofibromatosis, in which hemihypertrophy has been described. A role of inheritance was suggested in our series by one patient who had a sibling with hemihypertrophy, the seventh reported familial occurrence of this congenital defect. From the sparse evidence available, it would appear that the origins of hemihypertrophy are heterogeneous and include genic, chromosomal, and other factors which are presently obscure. The association between hemihypertrophy and Wilms' tumor may reflect common etiologic factors or a pre-neoplastic anlage in "hemihypertrophic" kidneys. Since hemihypertrophy seems to be related also to childhood neoplasms originating in the adrenal cortex and liver, further research on this anomaly should enhance our understanding of oncogenic mechanisms.

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