scholarly journals Deterministic Switching of Hierarchy during Wrinkling in Quasi‐Planar Bilayers

2016 ◽  
Vol 18 (6) ◽  
pp. 938-943 ◽  
Author(s):  
Sourabh K. Saha ◽  
Martin L. Culpepper
Keyword(s):  
Planar Bilayers ◽  
Deterministic Switching

scholarly journals Reducing Dzyaloshinskii-Moriya interaction and field-free spin-orbit torque switching in synthetic antiferromagnets

2021 ◽  
Vol 12 (1) ◽  
Author(s):  
Ruyi Chen ◽  
Qirui Cui ◽  
Liyang Liao ◽  
Yingmei Zhu ◽  
Ruiqi Zhang ◽  
...  
Keyword(s):  
Domain Wall ◽  
Tunnel Junctions ◽  
Low Power Consumption ◽  
Stray Field ◽  
Magnetic Memory ◽  
Spin Orbit ◽  
Theoretic Analysis ◽  
Spintronic Devices ◽  
Deterministic Switching ◽  
Moriya Interaction

AbstractPerpendicularly magnetized synthetic antiferromagnets (SAF), possessing low net magnetization and high thermal stability as well as easy reading and writing characteristics, have been intensively explored to replace the ferromagnetic free layers of magnetic tunnel junctions as the kernel of spintronic devices. So far, utilizing spin-orbit torque (SOT) to realize deterministic switching of perpendicular SAF have been reported while a large external magnetic field is typically needed to break the symmetry, making it impractical for applications. Here, combining theoretic analysis and experimental results, we report that the effective modulation of Dzyaloshinskii-Moriya interaction by the interfacial crystallinity between ferromagnets and adjacent heavy metals plays an important role in domain wall configurations. By adjusting the domain wall configuration between Bloch type and Néel type, we successfully demonstrate the field-free SOT-induced magnetization switching in [Co/Pd]/Ru/[Co/Pd] SAF devices constructed with a simple wedged structure. Our work provides a practical route for utilization of perpendicularly SAF in SOT devices and paves the way for magnetic memory devices with high density, low stray field, and low power consumption.

scholarly journals Amphiphilic COSAN and I2-COSAN crossing synthetic lipid membranes: planar bilayers and liposomes

2014 ◽  
Vol 50 (51) ◽  
pp. 6700 ◽  
Author(s):  
Carmina Verdiá-Báguena ◽  
Antonio Alcaraz ◽  
Vicente M. Aguilella ◽  
Ana M. Cioran ◽  
Shoji Tachikawa ◽  
...  
Keyword(s):  
Lipid Membranes ◽  
Planar Bilayers

scholarly journals On the Formation of Lipid Nano-Scale Structures at Interfaces Beyond Planar Bilayers

2016 ◽  
Vol 110 (3) ◽  
pp. 189a
Author(s):  
Aleksandra Dabkowska ◽  
Cassandra Niman ◽  
Gaelle Offranc Piret ◽  
Henrik Persson ◽  
Hanna Wacklin ◽  
...  
Keyword(s):  
Planar Bilayers ◽  
Nano Scale ◽  
Scale Structures

scholarly journals Toward Deterministic Switching in Ferroelectric Systems: Insight Gained from In Situ TEM

2015 ◽  
Vol 21 (S3) ◽  
pp. 1347-1348
Author(s):  
James Hart ◽  
Michael Jablonski ◽  
Andrew Lang ◽  
Anoop Damadoran ◽  
Shi Liu ◽  
...  
Keyword(s):  
In Situ Tem ◽  
Deterministic Switching

scholarly journals Actin and agonist MHC–peptide complex–dependent T cell receptor microclusters as scaffolds for signaling

2005 ◽  
Vol 202 (8) ◽  
pp. 1031-1036 ◽  
Author(s):  
Gabriele Campi ◽  
Rajat Varma ◽  
Michael L. Dustin
Keyword(s):  
T Cell ◽  
T Cell Receptor ◽  
Actin Polymerization ◽  
Kinase Inhibitor ◽  
Cell Receptor ◽  
Intercellular Adhesion Molecule ◽  
Cell Contact ◽  
Intercellular Adhesion Molecule 1 ◽  
Planar Bilayers ◽  
Latrunculin A

T cell receptor (TCR) microclusters form within seconds of T cell contact with supported planar bilayers containing intercellular adhesion molecule-1 and agonist major histocompatibility complex (MHC)–peptide complexes, and elevation of cytoplasmic Ca2+ is observed within seconds of the first detectable microclusters. At 0–30 s after contact, TCR microclusters are colocalized with activated forms of Lck, ZAP-70, and the linker for activation of T cells. By 2 min, activated kinases are reduced in the older central microclusters, but are abundant in younger peripheral microclusters. By 5 min, TCR in the central supramolecular activation cluster have reduced activated kinases, whereas faint peripheral TCR microclusters efficiently generated activated Lck and ZAP-70. TCR microcluster formation is resistant to inhibition by Src family kinase inhibitor PP2, but is abrogated by actin polymerization inhibitor latrunculin A. We propose that Src kinase–independent formation of TCR microclusters in response to agonist MHC–peptide provides an actin-dependent scaffold for signal amplification.

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