Transverse Myelitis due to Meningovascular Syphilis

1984 ◽  
Vol 41 (3) ◽  
pp. 337-338 ◽  
Author(s):  
E. P. Harrigan ◽  
T. J. McLaughlin ◽  
R. G. Feldman
Keyword(s):  
Transverse Myelitis ◽  
Meningovascular Syphilis

Longitudinal Extensive Transverse Myelitis following T-ALL

2016 ◽  
Vol 47 (S 01) ◽  
Author(s):  
V. Kraus ◽  
C. Makowski ◽  
H. Jünger ◽  
A. Wawer ◽  
P. Strotmann ◽  
...  
Keyword(s):  
Transverse Myelitis ◽  
Longitudinal Extensive Transverse Myelitis ◽  
Extensive Transverse Myelitis

Transverse Myelitis und Retrobulbärneuritis bei einer jugendlichen Patientin mit systemischem Lupus erythematodes

2016 ◽  
Vol 36 (03) ◽  
pp. 180-182
Author(s):  
F. Dressler ◽  
H. Hartmann ◽  
E. Bültmann ◽  
N. Schwerk ◽  
A. Thon ◽  
...  
Keyword(s):  
Transverse Myelitis ◽  
Lupus Erythematodes

Anti-Myelin Oligodendrocyte Glycoprotein Encephalomyelitis and Extensive Longitudinal Transverse Myelitis Associated with Compound Heterozygous NLRP3 Missense Mutations in a Young Child

2020 ◽  
Author(s):  
Deirdre O'Sullivan ◽  
Michael Moore ◽  
Susan Byrne ◽  
Andreas O. Reiff ◽  
Susanna Felsenstein
Keyword(s):  
Young Child ◽  
Genetic Basis ◽  
Transverse Myelitis ◽  
Acute Disseminated Encephalomyelitis ◽  
Myelin Oligodendrocyte Glycoprotein ◽  
Compound Heterozygous ◽  
Missense Mutations ◽  
Childhood Onset

AbstractAcute disseminated encephalomyelitis in association with extensive longitudinal transverse myelitis is reported in a young child with positive anti-myelin oligodendrocyte glycoprotein (MOG) antibody with heterozygous NLRP3 missense mutations; p.(Arg488Lys) and p.(Ser159Ile). This case may well present an exceptional coincidence, but may describe a yet unrecognized feature of the spectrum of childhood onset cryopyrinopathies that contribute to the understanding of the genetic basis for anti-MOG antibody positive encephalomyelitis. Based on this observation, a larger scale study investigating the role of NLRP3 and other inflammasomes in this entity would provide important pathophysiological insights and potentially novel avenues for treatment.

Akute Querschnittslähmung im Adoleszentenalter – Transverse Myelitis

2005 ◽  
Vol 209 (S 1) ◽  
Author(s):  
K Petersen ◽  
T Rettig ◽  
A Lingenauber ◽  
D Weise ◽  
A von der Wense
Keyword(s):  
Transverse Myelitis

Atypical presentation of transverse myelitis in a 15 months old child

2008 ◽  
Vol 39 (01) ◽  
Author(s):  
D Wüller ◽  
B Köster ◽  
T Rosenbaum ◽  
H Alfke
Keyword(s):  
Transverse Myelitis ◽  
Atypical Presentation

scholarly journals Meningitis as a recurrent manifestation of anti-AQP4/anti-MOG negative neuromyelitis optica spectrum disorder: a case report

BMC Neurology ◽  
2021 ◽  
Vol 21 (1) ◽  
Author(s):  
Chenyang Zhang ◽  
Kang Zhang ◽  
Bing Chen ◽  
Jiao Yin ◽  
Miaomiao Dong ◽  
...  
Keyword(s):  
Neuromyelitis Optica ◽  
Area Postrema ◽  
Neurological Diseases ◽  
Brain Magnetic Resonance Imaging ◽  
Transverse Myelitis ◽  
Corticosteroid Treatment ◽  
Neck Stiffness ◽  
Mixed Cell ◽  
Glucose Levels ◽  
The Right

Abstract Background Neuromyelitis optica spectrum disorders (NMOSD), a group of autoimmune neurological diseases, involve the optic nerve, spinal cord, and brain. Meningitis is rarely reported as the primary clinical manifestation of both anti-aquaporin-4 (AQP4)/ anti-myelin oligodendrocyte glycoprotein (MOG) antibody-negative NMOSD (NMOSDneg). Case presentation A 30-year-old man initially presented with fever, headache, and neck stiffness. Lumbar puncture revealed mixed cell reaction and decreased glucose levels. As a result, tuberculous meningitis was suspected. After 1 month, the patient developed longitudinally extensive transverse myelitis and area postrema syndrome. This was followed by the presentation of meningitis-like symptoms once again in the third attack, but his condition eventually improved after corticosteroid treatment without relapse for 2 years. However, he was readmitted to our hospital owing to symptoms of diplopia, hiccup, and numbness in the right hand. Brain magnetic resonance imaging (MRI) revealed that the area postrema still contained lesions. Spinal MRI revealed several segmental enhancements at the C4–C5, T1, and T5 levels. Anti-AQP4 and anti-MOG antibodies were persistently absent in the serum and cerebrospinal fluid (CSF). The patient was finally diagnosed with NMOSDneg. Conclusions Meningitis could be a recurrent manifestation of NMOSDneg and requires more careful evaluation.

Sign in / Sign up

Export Citation Format

Share Document